Mara Stellato

500 total citations
9 papers, 301 citations indexed

About

Mara Stellato is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Mara Stellato has authored 9 papers receiving a total of 301 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cardiology and Cardiovascular Medicine, 4 papers in Molecular Biology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Mara Stellato's work include Cardiac Fibrosis and Remodeling (6 papers), Systemic Sclerosis and Related Diseases (3 papers) and Tissue Engineering and Regenerative Medicine (2 papers). Mara Stellato is often cited by papers focused on Cardiac Fibrosis and Remodeling (6 papers), Systemic Sclerosis and Related Diseases (3 papers) and Tissue Engineering and Regenerative Medicine (2 papers). Mara Stellato collaborates with scholars based in Switzerland, Poland and Germany. Mara Stellato's co-authors include Przemysław Błyszczuk, Gabriela Kania, Oliver Distler, Konrad Basler, Urs Eriksson, Silvia Behnke, Tomáš Valenta, Björn Müller‐Edenborn, Thomas F. Lüscher and Elena Osto and has published in prestigious journals such as International Journal of Molecular Sciences, European Heart Journal and Annals of the Rheumatic Diseases.

In The Last Decade

Mara Stellato

9 papers receiving 301 citations

Peers

Mara Stellato

CCM

IMMUN

ONCOL

SURGE

Taylor A. Johnson United States

Lulu Lai United States

B Liccardo Italy

Olga Lettau Germany

Mikoto Yoshida Japan

Omonigho Aisagbonhi United States

Björn‐Anders Jonsson Sweden

Jonathan Yap United States

Joel G. Rurik United States

Daria Domańska-Senderowska Poland

Taylor A. Johnson United States

Mara Stellato

170 ×1.2

103 ×0.8

CCM

35 ×0.5

IMMUN

36 ×0.7

ONCOL

43 ×1.0

SURGE

Citations per year, relative to Mara Stellato Mara Stellato (= 1×) peers Taylor A. Johnson

Countries citing papers authored by Mara Stellato

Since Specialization

Citations

This map shows the geographic impact of Mara Stellato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mara Stellato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mara Stellato more than expected).

Fields of papers citing papers by Mara Stellato

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mara Stellato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mara Stellato. The network helps show where Mara Stellato may publish in the future.

Co-authorship network of co-authors of Mara Stellato

This figure shows the co-authorship network connecting the top 25 collaborators of Mara Stellato. A scholar is included among the top collaborators of Mara Stellato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mara Stellato. Mara Stellato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

Stellato, Mara, Matthias Dewenter, Elena Pachera, et al.. (2023). The AP-1 transcription factor Fosl-2 drives cardiac fibrosis and arrhythmias under immunofibrotic conditions. Communications Biology. 6(1). 161–161. 8 indexed citations

Stellato, Mara, Burkhard Ludewig, Karin Klingel, et al.. (2021). The AP-1 Transcription Factor Fosl-2 Regulates Autophagy in Cardiac Fibroblasts during Myocardial Fibrogenesis. International Journal of Molecular Sciences. 22(4). 1861–1861. 25 indexed citations

Jordan, Suzana, et al.. (2021). Regulation of Monocyte Adhesion and Type I Interferon Signaling by CD52 in Patients With Systemic Sclerosis. Arthritis & Rheumatology. 73(9). 1720–1730. 19 indexed citations

Stellato, Mara, Claudia Haftmann, Alexander Vogetseder, et al.. (2020). The AP1 Transcription Factor Fosl2 Promotes Systemic Autoimmunity and Inflammation by Repressing Treg Development. Cell Reports. 31(13). 107826–107826. 62 indexed citations

Błyszczuk, Przemysław, Christian Zuppinger, Daria Nurzyńska, et al.. (2020). Activated Cardiac Fibroblasts Control Contraction of Human Fibrotic Cardiac Microtissues by a β-Adrenoreceptor-Dependent Mechanism. Cells. 9(5). 1270–1270. 9 indexed citations

Stellato, Mara, Marcin Czepiel, Oliver Distler, Przemysław Błyszczuk, & Gabriela Kania. (2019). Identification and Isolation of Cardiac Fibroblasts From the Adult Mouse Heart Using Two-Color Flow Cytometry. Frontiers in Cardiovascular Medicine. 6. 105–105. 23 indexed citations

Stellato, Mara, Alexander Vogetseder, Arun Subramaniam, et al.. (2019). SAT0001 FOSL-2 IS A REPRESSOR OF FOXP3 EXPRESSION DURING TREG DEVELOPMENT AND CONTROLS AUTOIMMUNITY. Annals of the Rheumatic Diseases. 78. 1065–1065. 1 indexed citations

Stellato, Mara, Elena Pachera, Karl Sotlar, et al.. (2017). OP0084 Rescue from the failing heart in systemic sclerosis, a novel insight: targeting TGF-β/FRA2-dependent autophagy. Annals of the Rheumatic Diseases. 76. 86–87. 1 indexed citations

Błyszczuk, Przemysław, Björn Müller‐Edenborn, Tomáš Valenta, et al.. (2016). Transforming growth factor-β-dependent Wnt secretion controls myofibroblast formation and myocardial fibrosis progression in experimental autoimmune myocarditis. European Heart Journal. 38(18). ehw116–ehw116. 153 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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