Mara Dottore

1.5k total citations
23 papers, 1.2k citations indexed

About

Mara Dottore is a scholar working on Immunology, Oncology and Hematology. According to data from OpenAlex, Mara Dottore has authored 23 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Immunology, 5 papers in Oncology and 5 papers in Hematology. Recurrent topics in Mara Dottore's work include Immune Cell Function and Interaction (11 papers), Immune Response and Inflammation (9 papers) and Immunotherapy and Immune Responses (6 papers). Mara Dottore is often cited by papers focused on Immune Cell Function and Interaction (11 papers), Immune Response and Inflammation (9 papers) and Immunotherapy and Immune Responses (6 papers). Mara Dottore collaborates with scholars based in Australia, United States and India. Mara Dottore's co-authors include Angel F. López, Joanna M. Woodcock, Timothy R. Hercus, Mathew A. Vadas, Bronwyn Cambareri, M Shannon, Frank C. Stomski, Barbara J. McClure, Mark A. Guthridge and Michael W. Parker and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Mara Dottore

23 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mara Dottore Australia 18 730 380 320 149 139 23 1.2k
Sazuku Nisitani Japan 17 730 1.0× 242 0.6× 485 1.5× 102 0.7× 156 1.1× 20 1.4k
Christi L. Wood United States 8 580 0.8× 443 1.2× 235 0.7× 97 0.7× 64 0.5× 10 1.1k
AL Mui Canada 11 592 0.8× 445 1.2× 430 1.3× 280 1.9× 78 0.6× 13 1.2k
Shirly Becker-Herman Israel 21 1.2k 1.6× 277 0.7× 373 1.2× 138 0.9× 63 0.5× 33 1.7k
Osamu Hosono Japan 23 352 0.5× 623 1.6× 491 1.5× 73 0.5× 110 0.8× 70 1.4k
Alexandra Schebesta Austria 12 759 1.0× 236 0.6× 586 1.8× 200 1.3× 41 0.3× 13 1.4k
Tomoaki Kuwaki Japan 11 417 0.6× 246 0.6× 359 1.1× 309 2.1× 84 0.6× 18 977
Yutaka Kawakami Japan 17 672 0.9× 452 1.2× 331 1.0× 52 0.3× 191 1.4× 31 1.2k
Kouji Matsushima Japan 9 930 1.3× 990 2.6× 543 1.7× 137 0.9× 54 0.4× 9 1.8k
Deborah Webb United States 11 502 0.7× 290 0.8× 253 0.8× 72 0.5× 90 0.6× 15 894

Countries citing papers authored by Mara Dottore

Since Specialization
Citations

This map shows the geographic impact of Mara Dottore's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mara Dottore with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mara Dottore more than expected).

Fields of papers citing papers by Mara Dottore

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mara Dottore. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mara Dottore. The network helps show where Mara Dottore may publish in the future.

Co-authorship network of co-authors of Mara Dottore

This figure shows the co-authorship network connecting the top 25 collaborators of Mara Dottore. A scholar is included among the top collaborators of Mara Dottore based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mara Dottore. Mara Dottore is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tvorogov, Denis, Mara Dottore, Frank C. Stomski, et al.. (2022). Targeting human CALR‐mutated MPN progenitors with a neoepitope‐directed monoclonal antibody. EMBO Reports. 23(4). e52904–e52904. 18 indexed citations
2.
Tvorogov, Denis, Danièl Thomas, Nicholas P. D. Liau, et al.. (2018). Accumulation of JAK activation loop phosphorylation is linked to type I JAK inhibitor withdrawal syndrome in myelofibrosis. Science Advances. 4(11). eaat3834–eaat3834. 41 indexed citations
3.
Broughton, Sophie E., Timothy R. Hercus, Tracy L. Nero, et al.. (2018). A dual role for the N-terminal domain of the IL-3 receptor in cell signalling. Nature Communications. 9(1). 386–386. 31 indexed citations
4.
Brumatti, Gabriela, Ceilidh Marchant, Jarrod J. Sandow, et al.. (2017). High CD123 levels enhance proliferation in response to IL-3, but reduce chemotaxis by downregulating CXCR4 expression. Blood Advances. 1(15). 1067–1079. 27 indexed citations
5.
Broughton, Sophie E., Timothy R. Hercus, Tracy L. Nero, et al.. (2016). Conformational Changes in the GM-CSF Receptor Suggest a Molecular Mechanism for Affinity Conversion and Receptor Signaling. Structure. 24(8). 1271–1281. 40 indexed citations
6.
Broughton, Sophie E., Timothy R. Hercus, Matthew P. Hardy, et al.. (2014). Dual Mechanism of Interleukin-3 Receptor Blockade by an Anti-Cancer Antibody. Cell Reports. 8(2). 410–419. 39 indexed citations
7.
Hercus, Timothy R., Emma F. Barry, Mara Dottore, et al.. (2013). High Yield Production of a Soluble Human Interleukin-3 Variant from E. coli with Wild-Type Bioactivity and Improved Radiolabeling Properties. PLoS ONE. 8(8). e74376–e74376. 12 indexed citations
8.
Hansen, Guido, Timothy R. Hercus, Barbara J. McClure, et al.. (2008). The Structure of the GM-CSF Receptor Complex Reveals a Distinct Mode of Cytokine Receptor Activation. Cell. 134(3). 496–507. 229 indexed citations
9.
Guthridge, Mark A., Jason A. Powell, Emma F. Barry, et al.. (2006). Growth factor pleiotropy is controlled by a receptor Tyr/Ser motif that acts as a binary switch. The EMBO Journal. 25(3). 479–489. 65 indexed citations
10.
Holland, Jane D., Marina Kochetkova, Chareeporn Akekawatchai, et al.. (2006). Differential Functional Activation of Chemokine Receptor CXCR4 Is Mediated by G Proteins in Breast Cancer Cells. Cancer Research. 66(8). 4117–4124. 84 indexed citations
11.
Guthridge, Mark A., Frank C. Stomski, Emma F. Barry, et al.. (2000). Site-Specific Serine Phosphorylation of the IL-3 Receptor Is Required for Hemopoietic Cell Survival. Molecular Cell. 6(1). 99–108. 62 indexed citations
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Hercus, Timothy R., C.J. Bagley, Bronwyn Cambareri, et al.. (1994). Specific human granulocyte-macrophage colony-stimulating factor antagonists.. Proceedings of the National Academy of Sciences. 91(13). 5838–5842. 96 indexed citations
17.
Barry, Simon C., C.J. Bagley, Julie A. Phillips, et al.. (1994). Two contiguous residues in human interleukin-3, Asp21 and Glu22, selectively interact with the alpha- and beta-chains of its receptor and participate in function.. Journal of Biological Chemistry. 269(11). 8488–8492. 74 indexed citations
18.
Korpelainen, Eija, Jennifer R. Gamble, William Smith, et al.. (1993). The receptor for interleukin 3 is selectively induced in human endothelial cells by tumor necrosis factor alpha and potentiates interleukin 8 secretion and neutrophil transmigration.. Proceedings of the National Academy of Sciences. 90(23). 11137–11141. 64 indexed citations
19.
Elliott, Michael J., et al.. (1992). Differential Binding of IL-3 and GM-CSF to Human Monocytes. Growth Factors. 6(1). 15–29. 15 indexed citations
20.
López, Angel F., M Shannon, Simon C. Barry, et al.. (1992). A human interleukin 3 analog with increased biological and binding activities.. Proceedings of the National Academy of Sciences. 89(24). 11842–11846. 44 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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