Manuela Brückner

1.2k total citations · 1 hit paper
5 papers, 893 citations indexed

About

Manuela Brückner is a scholar working on Molecular Biology, Immunology and Infectious Diseases. According to data from OpenAlex, Manuela Brückner has authored 5 papers receiving a total of 893 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Molecular Biology, 2 papers in Immunology and 1 paper in Infectious Diseases. Recurrent topics in Manuela Brückner's work include interferon and immune responses (2 papers), Cell death mechanisms and regulation (2 papers) and Cancer-related Molecular Pathways (1 paper). Manuela Brückner is often cited by papers focused on interferon and immune responses (2 papers), Cell death mechanisms and regulation (2 papers) and Cancer-related Molecular Pathways (1 paper). Manuela Brückner collaborates with scholars based in Austria, France and Italy. Manuela Brückner's co-authors include Giulio Superti‐Furga, Keiryn L. Bennett, Stefania Scorzoni, Manuele Rebsamen, Johannes W. Bigenzahn, Astrid Fauster, K. Huber, Richard K. Kandasamy, Berend Snijder and Leonhard X. Heinz and has published in prestigious journals such as Nature, The Journal of Experimental Medicine and Cell Death and Differentiation.

In The Last Decade

Manuela Brückner

5 papers receiving 885 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

SLC38A9 is a component of the lysosomal amino acid sensing machinery that controls mTORC1

2015 506 citations Manuele Rebsamen, Lorena Pochini et al. Nature profile →

Peers

Countries citing papers authored by Manuela Brückner

Since Specialization

Citations

This map shows the geographic impact of Manuela Brückner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Manuela Brückner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Manuela Brückner more than expected).

Fields of papers citing papers by Manuela Brückner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Manuela Brückner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Manuela Brückner. The network helps show where Manuela Brückner may publish in the future.

Co-authorship network of co-authors of Manuela Brückner

This figure shows the co-authorship network connecting the top 25 collaborators of Manuela Brückner. A scholar is included among the top collaborators of Manuela Brückner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Manuela Brückner. Manuela Brückner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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5 of 5 papers shown

#	Work	Indexed citations
1	Systematic genetic mapping of necroptosis identifies SLC39A7 as modulator of death receptor trafficking 2018 ·Cell Death and Differentiation ·Astrid Fauster, Manuele Rebsamen, Katharina Willmann, Adrián César‐Razquin, Enrico Girardi, Johannes W. Bigenzahn, Fiorella Schischlik, Stefania Scorzoni, Manuela Brückner, (unknown), (unknown), Leonhard X. Heinz, Kaan Boztuǧ, Giulio Superti‐Furga	26
2	A time-resolved molecular map of the macrophage response to VSV infection 2016 ·npj Systems Biology and Applications ·Richard K. Kandasamy, Gregory I. Vladimer, Berend Snijder, André C. Müller, Manuele Rebsamen, Johannes W. Bigenzahn, (unknown), (unknown), Adrijana Stefanovic, Stefania Scorzoni, Manuela Brückner, Thomas Penz, Ciara Cleary, Róbert Královics, Jacques Colinge, Keiryn L. Bennett, (unknown)	24
3	A cellular screen identifies ponatinib and pazopanib as inhibitors of necroptosis 2015 ·Cell Death and Disease ·Astrid Fauster, Manuele Rebsamen, K. Huber, Johannes W. Bigenzahn, Alexey Stukalov, (unknown), Stefania Scorzoni, Manuela Brückner, Manuela Gridling, Katja Parapatics, Jacques Colinge, Karen L. Bennett, Stefan Kubicek, Stefan Krautwald, Andreas Linkermann, Giulio Superti‐Furga	159
4	SLC38A9 is a component of the lysosomal amino acid sensing machinery that controls mTORC1 breakdown → 2015 ·Nature ·Manuele Rebsamen, Lorena Pochini, Taras Stasyk, Mariana E. G. de Araújo, Michele Galluccio, Richard K. Kandasamy, Berend Snijder, Astrid Fauster, Elena L. Rudashevskaya, Manuela Brückner, Stefania Scorzoni, Przemyslaw A. Filipek, K. Huber, Johannes W. Bigenzahn, Leonhard X. Heinz, Claudine Kraft, Keiryn L. Bennett, Cesare Indiveri, Lukas A. Huber, Giulio Superti‐Furga	506
5	CD14 is a coreceptor of Toll-like receptors 7 and 9 2010 ·The Journal of Experimental Medicine ·Christoph Baumann, Irene M. Aspalter, Omar Sharif, Andreas Pichlmair, Stephan Blüml, Florian Grebien, Manuela Brückner, Paweł Pasierbek, Karin Aumayr, Melanie Planyavsky, Keiryn L. Bennett, Jacques Colinge, Sylvia Knapp, Giulio Superti‐Furga	178

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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