Manuel Méndez

779 total citations
46 papers, 473 citations indexed

About

Manuel Méndez is a scholar working on Molecular Biology, Rheumatology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Manuel Méndez has authored 46 papers receiving a total of 473 indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 15 papers in Rheumatology and 14 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Manuel Méndez's work include Porphyrin Metabolism and Disorders (30 papers), Heme Oxygenase-1 and Carbon Monoxide (23 papers) and Folate and B Vitamins Research (15 papers). Manuel Méndez is often cited by papers focused on Porphyrin Metabolism and Disorders (30 papers), Heme Oxygenase-1 and Carbon Monoxide (23 papers) and Folate and B Vitamins Research (15 papers). Manuel Méndez collaborates with scholars based in Spain, Argentina and United States. Manuel Méndez's co-authors include María‐Josefa Morán‐Jiménez, Alcira Batlle, Rafael Enrı́quez de Salamanca, Antonio Fontanellas, Cécile Ged, Hubert de Verneuil, María García‐Bravo, François Moreau‐Gaudry, Frédéric Mazurier and Adriana De Siervi and has published in prestigious journals such as SHILAP Revista de lepidopterología, The American Journal of Human Genetics and Gene.

In The Last Decade

Manuel Méndez

46 papers receiving 457 citations

Peers

Manuel Méndez

RHEUM

HEMAT

GENET

PPCH

G. Biolcati Italy

Miguel Angel Alcántara‐Ortigoza Mexico

Pietro Cavalli Italy

Yvonne Mark United States

Christian M. Abratte United States

Anna Stanisławska‐Sachadyn Poland

Junya Shimizu Japan

Peter Friend United Kingdom

MH Steinberg United States

M. van Eygen Belgium

G. Biolcati Italy

Manuel Méndez

330 ×1.0

96 ×0.8

RHEUM

25 ×0.2

HEMAT

20 ×0.2

GENET

57 ×0.6

PPCH

Citations per year, relative to Manuel Méndez Manuel Méndez (= 1×) peers G. Biolcati

Countries citing papers authored by Manuel Méndez

Since Specialization

Citations

This map shows the geographic impact of Manuel Méndez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Manuel Méndez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Manuel Méndez more than expected).

Fields of papers citing papers by Manuel Méndez

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Manuel Méndez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Manuel Méndez. The network helps show where Manuel Méndez may publish in the future.

Co-authorship network of co-authors of Manuel Méndez

This figure shows the co-authorship network connecting the top 25 collaborators of Manuel Méndez. A scholar is included among the top collaborators of Manuel Méndez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Manuel Méndez. Manuel Méndez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Salamanca, Rafael Enrı́quez de, et al.. (2018). Molecular analysis of 19 Spanish patients with mixed porphyrias. European Journal of Medical Genetics. 62(12). 103589–103589. 2 indexed citations

Morán‐Jiménez, María‐Josefa, et al.. (2013). Familial porphyria cutanea tarda in Spain: Characterization of eight novel mutations in the UROD gene and haplotype analysis of the common p.G281E mutation. Gene. 522(1). 89–95. 2 indexed citations

Méndez, Manuel, et al.. (2012). Molecular analysis of the UROD gene in 17 Argentinean patients with familial porphyria cutanea tarda: Characterization of four novel mutations. Molecular Genetics and Metabolism. 105(4). 629–633. 4 indexed citations

Méndez, Manuel, et al.. (2012). Functional associations of genetic variants involved in the clinical manifestation of erythropoietic protoporphyria in the Argentinean population. Journal of the European Academy of Dermatology and Venereology. 27(6). 754–762. 3 indexed citations

Rueda, A. del Castillo, et al.. (2012). Mutations in the HFE, TFR2, and SLC40A1 genes in patients with hemochromatosis. Gene. 508(1). 15–20. 24 indexed citations

Rueda, A. del Castillo, et al.. (2011). Hiperferritinemia, ferropenia y síndrome metabólico en un paciente con una nueva mutación en el gen TFR2 y otra en el gen FTL. Estudio familiar. Medicina Clínica. 137(2). 68–72. 3 indexed citations

Rueda, A. del Castillo, et al.. (2010). Two novel mutations in the SLC40A1 and HFE genes implicated in iron overload in a Spanish man. European Journal Of Haematology. 86(3). 260–264. 10 indexed citations

Méndez, Manuel, et al.. (2009). HFE gene mutations in patients with altered iron metabolism in Argentina.. PubMed. 55(2). 31–5. 3 indexed citations

García‐Bravo, María, María‐Josefa Morán‐Jiménez, Óscar Quintana-Bustamante, et al.. (2009). Bone Marrow-Derived Cells Promote Liver Regeneration in Mice With Erythropoietic Protoporphyria. Transplantation. 88(12). 1332–1340. 7 indexed citations

10.

Herrero, Carmen, et al.. (2007). Clinical, Biochemical, and Genetic Study of 11 Patients With Erythropoietic Protoporphyria Including One With Homozygous Disease. Archives of Dermatology. 143(9). 1125–9. 16 indexed citations

11.

Morán‐Jiménez, María‐Josefa, María García‐Bravo, Manuel Méndez, et al.. (2007). Bone marrow transplantation into hemochromatotic mice decreases hepatic and duodenal iron overload. The International Journal of Biochemistry & Cell Biology. 40(1). 135–146. 4 indexed citations

12.

Ged, Cécile, Manuel Méndez, Magalie Lalanne, et al.. (2005). A knock-in mouse model of congenital erythropoietic porphyria. Genomics. 87(1). 84–92. 21 indexed citations

13.

García‐Bravo, María, Susana Navarro, María‐Josefa Morán‐Jiménez, et al.. (2003). Volatile anaesthetics induce biochemical alterations in the heme pathway in a B-lymphocyte cell line established from hepatoerythropoietic porphyria patients (LBHEP) and in mice inoculated with LBHEP cells. The International Journal of Biochemistry & Cell Biology. 36(2). 216–222. 5 indexed citations

14.

Cruz‐Rojo, Jaime, Antonio Fontanellas, María‐Josefa Morán‐Jiménez, et al.. (2002). Precipitating/aggravating factors of porphyria cutanea tarda in Spanish patients.. PubMed. 48(8). 845–52. 23 indexed citations

15.

Siervi, Adriana De, et al.. (2002). Estudios genéticos de las porfirias agudas en Argentina. Acta bioquímica clínica latinoamericana. 36(4). 505–513. 1 indexed citations

16.

Oubiña, José R., Jorge Quarleri, Verónica L. Mathet, et al.. (2001). Hepatitis C Virus and GBV-C/Hepatitis G Virus in Argentine Patients with Porphyria Cutanea Tarda. Intervirology. 44(4). 215–218. 4 indexed citations

17.

Fontanellas, Antonio, Manuel Méndez, Frédéric Mazurier, et al.. (2001). Successful therapeutic effect in a mouse model of erythropoietic protoporphyria by partial genetic correction and fluorescence-based selection of hematopoietic cells. Gene Therapy. 8(8). 618–626. 22 indexed citations

18.

Richard, Emmanuel, Manuel Méndez, Frédéric Mazurier, et al.. (2001). Gene Therapy of a Mouse Model of Protoporphyria with a Self-Inactivating Erythroid-Specific Lentiviral Vector without Preselection. Molecular Therapy. 4(4). 331–338. 49 indexed citations

19.

Siervi, Adriana De, et al.. (1999). Acute intermittent porphyria: Characterization of two novel mutations in the porphobilinogen deaminase gene, one amino acid deletion (453-455delAGC) and one splicing aceptor site mutation (IVS8-1G>T). Human Mutation. 14(4). 355–355. 7 indexed citations

20.

Méndez, Manuel, et al.. (1998). Familial Porphyria Cutanea Tarda: Characterization of Seven Novel Uroporphyrinogen Decarboxylase Mutations and Frequency of Common Hemochromatosis Alleles. The American Journal of Human Genetics. 63(5). 1363–1375. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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