Mana Iizuka

839 total citations
28 papers, 656 citations indexed

About

Mana Iizuka is a scholar working on Physiology, Immunology and Molecular Biology. According to data from OpenAlex, Mana Iizuka has authored 28 papers receiving a total of 656 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 11 papers in Immunology and 8 papers in Molecular Biology. Recurrent topics in Mana Iizuka's work include Salivary Gland Disorders and Functions (15 papers), T-cell and B-cell Immunology (9 papers) and Monoclonal and Polyclonal Antibodies Research (8 papers). Mana Iizuka is often cited by papers focused on Salivary Gland Disorders and Functions (15 papers), T-cell and B-cell Immunology (9 papers) and Monoclonal and Polyclonal Antibodies Research (8 papers). Mana Iizuka collaborates with scholars based in Japan, United States and Belgium. Mana Iizuka's co-authors include Hiroto Tsuboi, Takayuki Sumida, Isao Matsumoto, Hiromitsu Asashima, Yuya Kondo, Akihiko Tanaka, Seiji Nakamura, Masafumi Moriyama, Tomoya Hirota and Naomi Matsuo and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and Arthritis Research & Therapy.

In The Last Decade

Mana Iizuka

27 papers receiving 651 citations

Peers

Mana Iizuka
Nancy Solowski United States
King‐Hung Ko Hong Kong
Lynn M. Hassman United States
Kristi A. Koelsch United States
Silvia Rihs Switzerland
Nancy Solowski United States
Mana Iizuka
Citations per year, relative to Mana Iizuka Mana Iizuka (= 1×) peers Nancy Solowski

Countries citing papers authored by Mana Iizuka

Since Specialization
Citations

This map shows the geographic impact of Mana Iizuka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mana Iizuka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mana Iizuka more than expected).

Fields of papers citing papers by Mana Iizuka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mana Iizuka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mana Iizuka. The network helps show where Mana Iizuka may publish in the future.

Co-authorship network of co-authors of Mana Iizuka

This figure shows the co-authorship network connecting the top 25 collaborators of Mana Iizuka. A scholar is included among the top collaborators of Mana Iizuka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mana Iizuka. Mana Iizuka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Brems, Hilde, Mana Iizuka, Setsuko Mise‐Omata, et al.. (2024). Legius syndrome mutations in the Ras-regulator SPRED1 abolish its membrane localization and potentially cause neurodegeneration. Journal of Biological Chemistry. 300(12). 107969–107969.
2.
Suzuki, Katsuya, Masaru Takeshita, Mana Iizuka, et al.. (2023). Anti-human-TIGIT agonistic antibody ameliorates autoimmune diseases by inhibiting Tfh and Tph cells and enhancing Treg cells. Communications Biology. 6(1). 500–500. 18 indexed citations
3.
Kondo, Yuya, Masahiro Tahara, Mana Iizuka, et al.. (2015). Involvement of RORγt-overexpressing T cells in the development of autoimmune arthritis in mice. Arthritis Research & Therapy. 17(1). 105–105. 15 indexed citations
4.
Iizuka, Mana, Yuhya Wakasa, Hiroto Tsuboi, et al.. (2014). Suppression of collagen‐induced arthritis by oral administration of transgenic rice seeds expressing altered peptide ligands of type II collagen. Plant Biotechnology Journal. 12(8). 1143–1152. 24 indexed citations
5.
Furukawa, Sachiko, Masafumi Moriyama, Akihiko Tanaka, et al.. (2014). Preferential M2 macrophages contribute to fibrosis in IgG4-related dacryoadenitis and sialoadenitis, so-called Mikulicz's disease. Clinical Immunology. 156(1). 9–18. 101 indexed citations
6.
Sumida, Takayuki, Hiroto Tsuboi, Mana Iizuka, et al.. (2014). The role of M3 muscarinic acetylcholine receptor reactive T cells in Sjögren's syndrome: A critical review. Journal of Autoimmunity. 51. 44–50. 44 indexed citations
7.
Iizuka, Mana, Yuhya Wakasa, Hiroto Tsuboi, et al.. (2014). Prophylactic effect of the oral administration of transgenic rice seeds containing altered peptide ligands of type II collagen on rheumatoid arthritis. Bioscience Biotechnology and Biochemistry. 78(10). 1662–1668. 7 indexed citations
8.
Iizuka, Mana, Hiroto Tsuboi, Hiromitsu Asashima, et al.. (2014). M3 muscarinic acetylcholine receptor reactive IL-17 producing T cells promotes development of Sjögren's syndrome like sialadenitis. Modern Rheumatology. 25(1). 158–160. 10 indexed citations
9.
Tsuboi, Hiroto, Hiromasa Ohira, Hiromitsu Asashima, et al.. (2014). Anti‐M3 muscarinic acetylcholine receptor antibodies in patients with primary biliary cirrhosis. Hepatology Research. 44(14). E471–9. 6 indexed citations
10.
Sumida, Takayuki, Hiroto Tsuboi, Mana Iizuka, Hiromitsu Asashima, & Isao Matsumoto. (2013). Anti-M3 muscarinic acetylcholine receptor antibodies in patients with Sjögren’s syndrome. Modern Rheumatology. 23(5). 841–845. 24 indexed citations
11.
Sumida, Takayuki, Mana Iizuka, Hiromitsu Asashima, Hiroto Tsuboi, & Isao Matsumoto. (2012). Pathogenic role of anti-M3 muscarinic acetylcholine receptor immune response in Sjögren's syndrome. La Presse Médicale. 41(9). e461–e466. 15 indexed citations
12.
Sumida, Takayuki, Hiroto Tsuboi, Mana Iizuka, Hiromitsu Asashima, & Isao Matsumoto. (2012). Anti-M3 muscarinic acetylcholine receptor antibodies in patients with Sjögren’s syndrome. Modern Rheumatology. 23(5). 841–845. 27 indexed citations
13.
Tsuboi, Hiroto, Naomi Matsuo, Mana Iizuka, et al.. (2012). Analysis of IgG4 class switch-related molecules in IgG4-related disease. Arthritis Research & Therapy. 14(4). R171–R171. 85 indexed citations
14.
Iizuka, Mana, et al.. (2011). AN RNA APTAMER CONTAINING TWO BINDING SITES AGAINST THE HCV MINUS-IRES DOMAIN I. Nucleosides Nucleotides & Nucleic Acids. 30(3). 185–202. 8 indexed citations
15.
Kondo, Yuya, Mana Iizuka, Ei Wakamatsu, et al.. (2011). Overexpression of T‐bet gene regulates murine autoimmune arthritis. Arthritis & Rheumatism. 64(1). 162–172. 27 indexed citations
16.
17.
Iizuka, Mana, Ei Wakamatsu, Hiroto Tsuboi, et al.. (2010). Pathogenic role of immune response to M3 muscarinic acetylcholine receptor in Sjögren’s syndrome-like sialoadenitis. Journal of Autoimmunity. 35(4). 383–389. 61 indexed citations
18.
Tsuboi, Hiroto, Ei Wakamatsu, Mana Iizuka, et al.. (2010). Importance of serine727 phosphorylated STAT1 in IFNγ‐induced signaling and apoptosis of human salivary gland cells. International Journal of Rheumatic Diseases. 14(1). 86–91. 11 indexed citations
19.
Sumida, Takayuki, et al.. (2010). Functional role of M3 muscarinic acetylcholine receptor (M3R) reactive T cells and anti-M3R autoantibodies in patients with Sjögren's syndrome. Autoimmunity Reviews. 9(9). 615–617. 25 indexed citations
20.
Tsuboi, Hiroto, et al.. (2010). Analysis of epitopes and function of anti-M3 muscarinic acetylcholine receptor antibodies in patients with Sjoegren's syndrome. Japanese Journal of Clinical Immunology. 33(4). 222–228. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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