Makoto Shibutani

6.7k total citations
333 papers, 5.3k citations indexed

About

Makoto Shibutani is a scholar working on Molecular Biology, Developmental Neuroscience and Cancer Research. According to data from OpenAlex, Makoto Shibutani has authored 333 papers receiving a total of 5.3k indexed citations (citations by other indexed papers that have themselves been cited), including 134 papers in Molecular Biology, 60 papers in Developmental Neuroscience and 56 papers in Cancer Research. Recurrent topics in Makoto Shibutani's work include Neurogenesis and neuroplasticity mechanisms (56 papers), Genomics, phytochemicals, and oxidative stress (36 papers) and Carcinogens and Genotoxicity Assessment (35 papers). Makoto Shibutani is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (56 papers), Genomics, phytochemicals, and oxidative stress (36 papers) and Carcinogens and Genotoxicity Assessment (35 papers). Makoto Shibutani collaborates with scholars based in Japan, China and South Korea. Makoto Shibutani's co-authors include Masao Hirose, Kunitoshi Mitsumori, Chikako Uneyama, Toshinori Yoshida, Hironori Takagi, Kaoru Inoue, Riki Okeda, Akiyoshi Nishikawa, Masashi Takahashi and Gye‐Hyeong Woo and has published in prestigious journals such as Journal of Biological Chemistry, Biomaterials and Analytical Biochemistry.

In The Last Decade

Makoto Shibutani

321 papers receiving 5.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Makoto Shibutani Japan 37 1.8k 915 656 565 556 333 5.3k
Kenneth R. Reuhl United States 45 2.4k 1.4× 1.1k 1.2× 512 0.8× 681 1.2× 358 0.6× 141 7.6k
Kunitoshi Mitsumori Japan 35 2.0k 1.1× 883 1.0× 1.1k 1.7× 759 1.3× 237 0.4× 356 5.0k
Ellen Fritsche Germany 38 1.5k 0.8× 859 0.9× 476 0.7× 190 0.3× 575 1.0× 126 4.2k
Rodrigo Franco United States 45 4.2k 2.4× 662 0.7× 634 1.0× 496 0.9× 114 0.2× 112 8.3k
Eun-Sook Lee United States 40 1.2k 0.7× 707 0.8× 329 0.5× 402 0.7× 307 0.6× 128 4.3k
Eisuke F. Sato Japan 39 2.0k 1.1× 376 0.4× 272 0.4× 310 0.5× 173 0.3× 255 5.9k
Bryan Mackenzie United States 32 2.0k 1.1× 1.5k 1.6× 231 0.4× 644 1.1× 83 0.1× 60 7.9k
Carsten Berndt Germany 36 4.9k 2.7× 306 0.3× 496 0.8× 305 0.5× 189 0.3× 68 7.8k
Kaoru Inoue Japan 34 1.5k 0.8× 369 0.4× 496 0.8× 215 0.4× 235 0.4× 245 4.7k
Kyung‐Chul Choi South Korea 48 3.0k 1.7× 1.7k 1.8× 1.0k 1.5× 1.2k 2.1× 103 0.2× 271 8.4k

Countries citing papers authored by Makoto Shibutani

Since Specialization
Citations

This map shows the geographic impact of Makoto Shibutani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Makoto Shibutani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Makoto Shibutani more than expected).

Fields of papers citing papers by Makoto Shibutani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Makoto Shibutani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Makoto Shibutani. The network helps show where Makoto Shibutani may publish in the future.

Co-authorship network of co-authors of Makoto Shibutani

This figure shows the co-authorship network connecting the top 25 collaborators of Makoto Shibutani. A scholar is included among the top collaborators of Makoto Shibutani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Makoto Shibutani. Makoto Shibutani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kobayashi, Mio, et al.. (2025). Emerging mycotoxin moniliformin induces renal tubular necrosis after oral exposure in mice. Food and Chemical Toxicology. 199. 115336–115336.
3.
Tang, Qian, et al.. (2024). DNA methylation-altered genes in the rat hippocampal neurogenic niche after continuous exposure to amorphous curcumin. Journal of Chemical Neuroanatomy. 137. 102414–102414. 1 indexed citations
4.
Ishii, Yuji, et al.. (2024). Formation of hepatocyte cytoplasmic inclusions and their contribution to methylcarbamate-induced hepatocarcinogenesis in F344 rats. Toxicological Sciences. 198(1). 40–49. 1 indexed citations
5.
Kobayashi, Mio, Tatsuya Usui, Mohamed Elbadawy, et al.. (2024). Anorectal Remodeling in the Transitional Zone with Increased Expression of LGR5, SOX9, SOX2, and Keratin 13 and 5 in a Dextran Sodium Sulfate-Induced Mouse Model of Ulcerative Colitis. International Journal of Molecular Sciences. 25(23). 12706–12706. 2 indexed citations
9.
Tang, Qian, Wen Zeng, Hiromu Okano, et al.. (2023). Amelioration of lipopolysaccharides-induced impairment of fear memory acquisition by alpha-glycosyl isoquercitrin through suppression of neuroinflammation in rats. The Journal of Toxicological Sciences. 48(3). 121–137. 5 indexed citations
10.
Takahashi, Yasunori, Saori Shimizu, Qian Tang, et al.. (2023). Suppression of neurogranin expression by disruption of epigenetic DNA methylation in hippocampal mature granule cells after developmental exposure to neurotoxicants in rats. Toxicology Letters. 390. 33–45. 2 indexed citations
11.
Bradley, Alys, Brad Bolon, Mark T. Butt, et al.. (2020). Proliferative and Nonproliferative Lesions of the Rat and Mouse Central and Peripheral Nervous Systems: New and Revised INHAND Terms. Toxicologic Pathology. 48(7). 827–844. 20 indexed citations
12.
Nishijima, Hitoshi, Yoshiki Matsuoka, Yasuhiro Mouri, et al.. (2017). Paradoxical development of polymyositis-like autoimmunity through augmented expression of autoimmune regulator (AIRE). Journal of Autoimmunity. 86. 75–92. 14 indexed citations
13.
Suzuki, Kazuhiko, et al.. (2011). Low-grade Myofibroblastic Sarcoma of the Maxillary Region in a Dog. Journal of Comparative Pathology. 147(1). 42–45. 4 indexed citations
14.
Ohishi, Takumi, Liyun Wang, Masashi Takahashi, et al.. (2010). Disruptive neuronal development by acrylamide in the hippocampal dentate hilus after developmental exposure in rats. Archives of Toxicology. 85(8). 987–994. 28 indexed citations
15.
Nishimura, Jihei, Yasuaki Dewa, Toshiya Okamura, et al.. (2008). Role of Nrf2 and Oxidative stress on Fenofibrate-Induced Hepatocarcinogenesis in Rats. Toxicological Sciences. 106(2). 339–349. 12 indexed citations
16.
Inoue, Kaoru, Makoto Shibutani, Naoya Masutomi, et al.. (2007). A 13-week subchronic toxicity study of madder color in F344 rats. Food and Chemical Toxicology. 46(1). 241–252. 18 indexed citations
17.
Ueda, Makoto, Naoko Niho, Toshio Imai, et al.. (2003). Lack of Significant Effects of Genistein on the Progression of 7,12-dimethylbenz(a)anthracene-Induced Mammary Tumors in Ovariectomized Sprague-Dawley Rats. Nutrition and Cancer. 47(2). 141–147. 20 indexed citations
19.
Maekawa, Akihiko, Hiroshi Onodera, Y Matsushima, et al.. (1990). High Yields of Granulosa Cell Tumors/Luteomas in F344 Rat Ovaries after Transplacental Administration of N‐Nitrosobis(2‐oxopropyl)amine. Japanese Journal of Cancer Research. 81(11). 1077–1080. 5 indexed citations
20.
Matsushima, Y, Akira Maekawa, H. Onodera, et al.. (1990). [Toxicity and carcinogenicity studies of musk xylol in B6C3F1 mouse].. PubMed. 89–94. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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