Mako Toyoda

4.4k total citations
19 papers, 169 citations indexed

About

Mako Toyoda is a scholar working on Virology, Infectious Diseases and Molecular Biology. According to data from OpenAlex, Mako Toyoda has authored 19 papers receiving a total of 169 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Virology, 11 papers in Infectious Diseases and 5 papers in Molecular Biology. Recurrent topics in Mako Toyoda's work include HIV Research and Treatment (12 papers), SARS-CoV-2 and COVID-19 Research (5 papers) and Immune Cell Function and Interaction (4 papers). Mako Toyoda is often cited by papers focused on HIV Research and Treatment (12 papers), SARS-CoV-2 and COVID-19 Research (5 papers) and Immune Cell Function and Interaction (4 papers). Mako Toyoda collaborates with scholars based in Japan, United States and Canada. Mako Toyoda's co-authors include Takamasa Ueno, Zabrina L. Brumme, Mark A. Brockman, Xiaomei T. Kuang, Toong Seng Tan, Bruce D. Walker, Hidenori Kaminishi, Hiroji Chibana, Masayuki Sudoh and Francis Mwimanzi and has published in prestigious journals such as Nature Communications, Journal of Virology and Scientific Reports.

In The Last Decade

Mako Toyoda

19 papers receiving 168 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mako Toyoda Japan 8 95 87 67 48 34 19 169
Slim Fourati United States 6 81 0.9× 120 1.4× 105 1.6× 66 1.4× 73 2.1× 13 281
Carly E. Starke United States 7 68 0.7× 81 0.9× 44 0.7× 36 0.8× 39 1.1× 15 160
Sheetal Sawant United States 8 100 1.1× 39 0.4× 98 1.5× 63 1.3× 42 1.2× 17 213
Yudai Kaneko Japan 8 61 0.6× 118 1.4× 84 1.3× 44 0.9× 41 1.2× 21 257
Nadeene E. Riddick United States 8 220 2.3× 106 1.2× 107 1.6× 66 1.4× 77 2.3× 10 270
Catherine K. Koofhethile United States 7 80 0.8× 40 0.5× 118 1.8× 61 1.3× 28 0.8× 16 190
Pholo Maenetje South Africa 10 73 0.8× 79 0.9× 94 1.4× 34 0.7× 74 2.2× 19 225
David Easterhoff United States 8 147 1.5× 64 0.7× 95 1.4× 96 2.0× 26 0.8× 13 237
Elena Heusinger Germany 6 89 0.9× 56 0.6× 72 1.1× 56 1.2× 51 1.5× 6 168
Marcos V. P. Gondim Brazil 6 149 1.6× 82 0.9× 115 1.7× 54 1.1× 49 1.4× 8 216

Countries citing papers authored by Mako Toyoda

Since Specialization
Citations

This map shows the geographic impact of Mako Toyoda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mako Toyoda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mako Toyoda more than expected).

Fields of papers citing papers by Mako Toyoda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mako Toyoda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mako Toyoda. The network helps show where Mako Toyoda may publish in the future.

Co-authorship network of co-authors of Mako Toyoda

This figure shows the co-authorship network connecting the top 25 collaborators of Mako Toyoda. A scholar is included among the top collaborators of Mako Toyoda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mako Toyoda. Mako Toyoda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Maeda, Yosuke, Mako Toyoda, Takeo Kuwata, et al.. (2024). Differential Ability of Spike Protein of SARS-CoV-2 Variants to Downregulate ACE2. International Journal of Molecular Sciences. 25(2). 1353–1353. 1 indexed citations
2.
Ngare, Isaac, Toong Seng Tan, Mako Toyoda, et al.. (2024). Factors Associated with Neutralizing Antibody Responses following 2-Dose and 3rd Booster Monovalent COVID-19 Vaccination in Japanese People Living with HIV. Viruses. 16(4). 555–555. 2 indexed citations
3.
Tan, Toong Seng, et al.. (2023). Relative resistance of patient-derived envelope sequences to SERINC5-mediated restriction of HIV-1 infectivity. Journal of Virology. 97(10). e0082323–e0082323. 2 indexed citations
4.
Toyoda, Mako, Toong Seng Tan, Chihiro Motozono, et al.. (2023). Evaluation of Neutralizing Activity against Omicron Subvariants in BA.5 Breakthrough Infection and Three-Dose Vaccination Using a Novel Chemiluminescence-Based, Virus-Mediated Cytopathic Assay. Microbiology Spectrum. 11(4). e0066023–e0066023. 2 indexed citations
5.
Motozono, Chihiro, Mako Toyoda, Toong Seng Tan, et al.. (2022). The SARS-CoV-2 Omicron BA.1 spike G446S mutation potentiates antiviral T-cell recognition. Nature Communications. 13(1). 5440–5440. 14 indexed citations
6.
Tan, Toong Seng, Mako Toyoda, Hirotaka Ode, et al.. (2022). Dissecting Naturally Arising Amino Acid Substitutions at Position L452 of SARS-CoV-2 Spike. Journal of Virology. 96(20). e0116222–e0116222. 7 indexed citations
7.
8.
Tan, Toong Seng, Mako Toyoda, Kenzo Tokunaga, & Takamasa Ueno. (2021). Aromatic Side Chain at Position 412 of SERINC5 Exerts Restriction Activity toward HIV-1 and Other Retroviruses. Journal of Virology. 95(18). e0063421–e0063421. 7 indexed citations
9.
Mwimanzi, Francis, Isaac Ngare, Mako Toyoda, et al.. (2020). An HIV-1 Nef genotype that diminishes immune control mediated by protective human leucocyte antigen alleles. AIDS. 34(9). 1325–1330. 1 indexed citations
10.
Toyoda, Mako, Toong Seng Tan, Jun Ohashi, et al.. (2020). Impaired ability of Nef to counteract SERINC5 is associated with reduced plasma viremia in HIV-infected individuals. Scientific Reports. 10(1). 19416–19416. 8 indexed citations
11.
Alsahafi, Nirmin, Xiaomei T. Kuang, Mako Toyoda, et al.. (2019). Natural HIV-1 Nef Polymorphisms Impair SERINC5 Downregulation Activity. Cell Reports. 29(6). 1449–1457.e5. 21 indexed citations
12.
Barton, John P., Jaclyn K. Mann, Dariusz K. Murakowski, et al.. (2019). Modelling and in vitro testing of the HIV-1 Nef fitness landscape. Virus Evolution. 5(2). vez029–vez029. 7 indexed citations
13.
14.
Toyoda, Mako, Xiaofei Jia, Xiaomei T. Kuang, et al.. (2016). Relative Resistance of HLA-B to Downregulation by Naturally Occurring HIV-1 Nef Sequences. mBio. 7(1). e01516–15. 13 indexed citations
15.
Toyoda, Mako, Yoko Ogata, Yosuke Maeda, et al.. (2015). Differential Ability of Primary HIV-1 Nef Isolates To Downregulate HIV-1 Entry Receptors. Journal of Virology. 89(18). 9639–9652. 21 indexed citations
16.
Mwimanzi, Francis, Mako Toyoda, Masahiko Mori, et al.. (2015). Association between a naturally arising polymorphism within a functional region of HIV-1 Nef and disease progression in chronic HIV-1 infection. Archives of Virology. 160(8). 2033–2041. 5 indexed citations
17.
Kuang, Xiaomei T., Mako Toyoda, Eric S. Martin, et al.. (2015). A robust and scalable TCR-based reporter cell assay to measure HIV-1 Nef-mediated T cell immune evasion. Journal of Immunological Methods. 426. 104–113. 19 indexed citations
18.
Kurihara, Daisuke, et al.. (2009). Lipoteichoic acid improves the capability of mast cells in the host defense system against bacteria. Inflammation Research. 58(11). 797–807. 4 indexed citations
19.
Toyoda, Mako, et al.. (2004). Transcriptional profiling of the early stages of germination in by real-time RT-PCR. FEMS Yeast Research. 5(3). 287–296. 21 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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