Maha Μ. Gebara

587 total citations
8 papers, 511 citations indexed

About

Maha Μ. Gebara is a scholar working on Molecular Biology, Genetics and Ecology. According to data from OpenAlex, Maha Μ. Gebara has authored 8 papers receiving a total of 511 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 3 papers in Genetics and 2 papers in Ecology. Recurrent topics in Maha Μ. Gebara's work include RNA Research and Splicing (3 papers), RNA modifications and cancer (2 papers) and Bacteriophages and microbial interactions (2 papers). Maha Μ. Gebara is often cited by papers focused on RNA Research and Splicing (3 papers), RNA modifications and cancer (2 papers) and Bacteriophages and microbial interactions (2 papers). Maha Μ. Gebara collaborates with scholars based in United States, United Kingdom and China. Maha Μ. Gebara's co-authors include Anton Yuryev, Meera Patturajan, Cosimo Gentile, J L Corden, Ying Litingtung, Radhika Joshi, John R. McCarrey, Michael H. Sayre, Susan A. Harcourt and C.F. Arlett and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Molecular and Cellular Biology and Journal of Cellular Biochemistry.

In The Last Decade

Maha Μ. Gebara

8 papers receiving 503 citations

Peers

Countries citing papers authored by Maha Μ. Gebara

Since Specialization

Citations

This map shows the geographic impact of Maha Μ. Gebara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Maha Μ. Gebara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Maha Μ. Gebara more than expected).

Fields of papers citing papers by Maha Μ. Gebara

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Maha Μ. Gebara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Maha Μ. Gebara. The network helps show where Maha Μ. Gebara may publish in the future.

Co-authorship network of co-authors of Maha Μ. Gebara

This figure shows the co-authorship network connecting the top 25 collaborators of Maha Μ. Gebara. A scholar is included among the top collaborators of Maha Μ. Gebara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Maha Μ. Gebara. Maha Μ. Gebara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Phosphorylation of the carboxy‐terminal repeat domain in RNA polymerase II by cyclin‐dependent kinases is sufficient to inhibit transcription 1997 ·Journal of Cellular Biochemistry ·Maha Μ. Gebara, Michael H. Sayre, (unknown)	2
2	Phosphorylation of the carboxy-terminal repeat domain in RNA polymerase II by cyclin-dependent kinases is sufficient to inhibit transcription 1997 ·Journal of Cellular Biochemistry ·Maha Μ. Gebara, Michael H. Sayre, (unknown)	77
3	The C-terminal domain of the largest subunit of RNA polymerase II interacts with a novel set of serine/arginine-rich proteins. 1996 ·Proceedings of the National Academy of Sciences ·Anton Yuryev, Meera Patturajan, Ying Litingtung, Radhika Joshi, Cosimo Gentile, Maha Μ. Gebara, J L Corden	306
4	Protein-DNA Interactions Associated with the Onset of TestisSpecific Expression of the Mammalian Pgk-2 Gene 1992 ·Molecular and Cellular Biology ·Maha Μ. Gebara, John R. McCarrey	4
5	Protein-DNA interactions associated with the onset of testis-specific expression of the mammalian Pgk-2 gene. 1992 ·Molecular and Cellular Biology ·Maha Μ. Gebara, John R. McCarrey	59
6	Mutagenic treatments result in inactivation of expression of a transfected bacterial gene integrated into a human cell line 1989 ·Mutation Research/Reviews in Genetic Toxicology ·Alan R. Lehmann, C.F. Arlett, Susan A. Harcourt, Herdis Steingrimsdottir, Maha Μ. Gebara	10
7	Inactivation of a transfected gene in human fibroblasts can occur by deletion, amplification, phenotypic switching, or methylation. 1987 ·Molecular and Cellular Biology ·Maha Μ. Gebara, C. Drevon, Susan A. Harcourt, Herdis Steingrimsdottir, Michael R. James, J. F. Burke, C.F. Arlett, Alan R. Lehmann	42
8	Inactivation of a Transfected Gene in Human Fibroblasts Can Occur by Deletion, Amplification, Phenotypic Switching, or Methylation 1987 ·Molecular and Cellular Biology ·Maha Μ. Gebara, C. Drevon, Susan A. Harcourt, Herdis Steingrimsdottir, Michael R. James, Julian F. Burke, C.F. Arlett, Alan R. Lehmann	11

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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