Magali Espinosa

642 citations
25 papers · 515 indexed · h-index 13
Topics
Cell death mechanisms and regulation (9 papers)Protease and Inhibitor Mechanisms (5 papers)Cancer-related Molecular Pathways (5 papers)
Partner nations
MexicoChileIreland

In The Last Decade

Magali Espinosa

24 papers receiving 509 citations

Peers

Magali Espinosa
Comparison fields: 5 of 74
  • Molecular Biology 358
  • Cancer Research 187
  • Oncology 161
  • Immunology 82
  • Surgery 49
Replace Madhuri Kalathur with:
Madhuri Kalathur United States
Tiziana Tonini United States
Raoying Xie China
Shuo You China
Yeo Song Lee South Korea
Ryosuke Moriai Japan
Hossein Tabatabaeian Iran
Shengchun Wang China
Jing Xun China
Magali Espinosa relative to Madhuri Kalathur United States Madhuri Kalathur's profile →
Citations per field
00.5×1.6×
Madhuri Kalathur · 1×
Citations per year

Countries citing papers authored by Magali Espinosa

Since Specialization
Citations

This map shows the geographic impact of Magali Espinosa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Magali Espinosa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Magali Espinosa more than expected).

Fields of papers citing papers by Magali Espinosa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Magali Espinosa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Magali Espinosa. The network helps show where Magali Espinosa may publish in the future.

Co-authorship network of co-authors of Magali Espinosa

This figure shows the co-authorship network connecting the top 25 collaborators of Magali Espinosa. A scholar is included among the top collaborators of Magali Espinosa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Magali Espinosa. Magali Espinosa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 2
2 2
3 93
4 11
5 27
6 7
7
Tabaco, alcohol y cáncer de laringe
0
8 9
9 13
10 5
11 10
12 11
13 22
14 85
15 37
16 34
17 6
18 17
19 13
20 37

About Magali Espinosa

Magali Espinosa is a scholar working on Cancer Research, Immunology and Allergy and Oncology, having authored 25 papers that have together received 515 indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (9 papers), Protease and Inhibitor Mechanisms (5 papers) and Cancer-related Molecular Pathways (5 papers). The work is most often cited by research in Cancer Research (187 citations), Oncology (161 citations) and Molecular Biology (358 citations). Magali Espinosa has collaborated with scholars based in Mexico, Chile and Ireland. Frequent co-authors include Jorge Meléndez-Zajgla, Vilma Maldonado, Gisela Ceballos‐Cancino, Vilma Maldonado, Floria Lizárraga, Iván Bahena, David Cantú de León, Angela Schwarz‐Cruz y Celis, Luz María Torres‐Espíndola and Guadalupe Ayala. Their work appears in journals such as PLoS ONE, Oncogene and Biochemical Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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