M.A. Pest

593 total citations
18 papers, 444 citations indexed

About

M.A. Pest is a scholar working on Rheumatology, Molecular Biology and Pharmacology. According to data from OpenAlex, M.A. Pest has authored 18 papers receiving a total of 444 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Rheumatology, 9 papers in Molecular Biology and 6 papers in Pharmacology. Recurrent topics in M.A. Pest's work include Osteoarthritis Treatment and Mechanisms (14 papers), Bone Metabolism and Diseases (7 papers) and Inflammatory mediators and NSAID effects (6 papers). M.A. Pest is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (14 papers), Bone Metabolism and Diseases (7 papers) and Inflammatory mediators and NSAID effects (6 papers). M.A. Pest collaborates with scholars based in Canada, United States and South Korea. M.A. Pest's co-authors include Frank Beier, S.E. Usmani, Vasek Pitelka, John S. Mort, A. Ratneswaran, Ling Qin, C. Thomas Appleton, Gunwoo Kim, Yuwen Zhang and Jae‐Wook Jeong and has published in prestigious journals such as SHILAP Revista de lepidopterología, The EMBO Journal and PLoS ONE.

In The Last Decade

M.A. Pest

18 papers receiving 440 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
M.A. Pest Canada	11	223	174	150	96	74	18	444
M.-F. Hsueh United States	10	352 1.6×	152 0.9×	114 0.8×	75 0.8×	87 1.2×	24	540
F. Cailotto France	15	220 1.0×	247 1.4×	48 0.3×	69 0.7×	57 0.8×	31	522
Y.H. Sniekers Netherlands	6	341 1.5×	87 0.5×	128 0.9×	56 0.6×	130 1.8×	9	454
Jean F. Gardin United States	7	286 1.3×	94 0.5×	74 0.5×	48 0.5×	117 1.6×	9	435
A.‐M. Zuurmond Netherlands	11	441 2.0×	114 0.7×	158 1.1×	56 0.6×	150 2.0×	21	536
Stefan N. Andersen Netherlands	8	279 1.3×	85 0.5×	76 0.5×	51 0.5×	68 0.9×	10	421
Guanqiao Liu China	10	140 0.6×	189 1.1×	41 0.3×	63 0.7×	97 1.3×	25	459
Vera Prestipino Italy	12	152 0.7×	144 0.8×	66 0.4×	31 0.3×	48 0.6×	12	488
Martina S. Elias United Kingdom	9	194 0.9×	95 0.5×	56 0.4×	66 0.7×	163 2.2×	11	580
B. Stott United Kingdom	8	167 0.7×	76 0.4×	76 0.5×	104 1.1×	45 0.6×	13	461

Countries citing papers authored by M.A. Pest

Since Specialization

Citations

This map shows the geographic impact of M.A. Pest's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M.A. Pest with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M.A. Pest more than expected).

Fields of papers citing papers by M.A. Pest

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M.A. Pest. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M.A. Pest. The network helps show where M.A. Pest may publish in the future.

Co-authorship network of co-authors of M.A. Pest

This figure shows the co-authorship network connecting the top 25 collaborators of M.A. Pest. A scholar is included among the top collaborators of M.A. Pest based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M.A. Pest. M.A. Pest is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

18 of 18 papers shown

Pest, M.A., et al.. (2023). Anabolic phenotype in cartilage-specific mitogen-inducible gene-6 knockout mice is independent of transforming growth factor-α. SHILAP Revista de lepidopterología. 5(3). 100387–100387. 1 indexed citations

Pest, M.A., et al.. (2020). Overexpression of MIG-6 in the cartilage induces an osteoarthritis-like phenotype in mice. Arthritis Research & Therapy. 22(1). 119–119. 10 indexed citations

Dupuis, Holly, et al.. (2019). Exposure to the RXR Agonist SR11237 in Early Life Causes Disturbed Skeletal Morphogenesis in a Rat Model. International Journal of Molecular Sciences. 20(20). 5198–5198. 9 indexed citations

Castillo‐Azofeifa, David, Elena N. Fazio, Roy Nattiv, et al.. (2019). Atoh1 ⁺ secretory progenitors possess renewal capacity independent of Lgr5 ⁺ cells during colonic regeneration. The EMBO Journal. 38(4). 55 indexed citations

Pest, M.A., et al.. (2017). EGFR/ MIG-6 signalling in osteoarthritis. Osteoarthritis and Cartilage. 25. S143–S144. 1 indexed citations

McCann, Matthew R., Cynthia Yeung, M.A. Pest, et al.. (2016). Whole-body vibration of mice induces articular cartilage degeneration with minimal changes in subchondral bone. Osteoarthritis and Cartilage. 25(5). 770–778. 17 indexed citations

McCann, Matthew R., A. Ratneswaran, M.A. Pest, et al.. (2016). C57BL/6 mice are resistant to joint degeneration induced by whole-body vibration. Osteoarthritis and Cartilage. 25(3). 421–425. 15 indexed citations

Usmani, S.E., Veronica Ulici, M.A. Pest, et al.. (2016). Context-specific protection of TGFα null mice from osteoarthritis. Scientific Reports. 6(1). 30434–30434. 19 indexed citations

Pest, M.A., Clayon B. Hamilton, A. Ratneswaran, et al.. (2016). The rock inhibitor fasudil does not protect against cartilage degeneration in a rat model of post-traumatic OA. Osteoarthritis and Cartilage. 24. S391–S391. 1 indexed citations

10.

Beier, Frank, et al.. (2016). Recent developments in emerging therapeutic targets of osteoarthritis. Current Opinion in Rheumatology. 29(1). 96–102. 67 indexed citations

11.

Pest, M.A., et al.. (2015). Deletion of Dual Specificity Phosphatase 1 Does Not Predispose Mice to Increased Spontaneous Osteoarthritis. PLoS ONE. 10(11). e0142822–e0142822. 7 indexed citations

12.

Appleton, C. Thomas, S.E. Usmani, M.A. Pest, et al.. (2015). Reduction in Disease Progression by Inhibition of Transforming Growth Factor α–CCL2 Signaling in Experimental Posttraumatic Osteoarthritis. Arthritis & Rheumatology. 67(10). 2691–2701. 58 indexed citations

13.

Hamilton, Clayon B., M.A. Pest, Vasek Pitelka, et al.. (2015). Weight-bearing asymmetry and vertical activity differences in a rat model of post-traumatic knee osteoarthritis. Osteoarthritis and Cartilage. 23(7). 1178–1185. 31 indexed citations

14.

McCann, Matthew R., M.A. Pest, A. Ratneswaran, et al.. (2015). Repeated Exposure to High‐Frequency Low‐Amplitude Vibration Induces Degeneration of Murine Intervertebral Discs and Knee Joints. Arthritis & Rheumatology. 67(8). 2164–2175. 55 indexed citations

15.

Pest, M.A., et al.. (2014). Loss of mitogen-inducible gene 6 results in disturbed cartilage and joint homeostasis. Osteoarthritis and Cartilage. 22. S41–S41. 1 indexed citations

16.

Pest, M.A. & Frank Beier. (2014). Is there such a thing as a cartilage-specific knockout mouse?. Nature Reviews Rheumatology. 10(12). 702–704. 5 indexed citations

17.

Pest, M.A., et al.. (2014). Disturbed Cartilage and Joint Homeostasis Resulting From a Loss of Mitogen‐Inducible Gene 6 in a Mouse Model of Joint Dysfunction. Arthritis & Rheumatology. 66(10). 2816–2827. 40 indexed citations

18.

Usmani, S.E., et al.. (2012). Transforming growth factor alpha controls the transition from hypertrophic cartilage to bone during endochondral bone growth. Bone. 51(1). 131–141. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact