F. Cailotto

943 total citations
31 papers, 522 citations indexed

About

F. Cailotto is a scholar working on Rheumatology, Molecular Biology and Oncology. According to data from OpenAlex, F. Cailotto has authored 31 papers receiving a total of 522 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Rheumatology, 14 papers in Molecular Biology and 7 papers in Oncology. Recurrent topics in F. Cailotto's work include Osteoarthritis Treatment and Mechanisms (14 papers), Inflammatory mediators and NSAID effects (6 papers) and Connective tissue disorders research (5 papers). F. Cailotto is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (14 papers), Inflammatory mediators and NSAID effects (6 papers) and Connective tissue disorders research (5 papers). F. Cailotto collaborates with scholars based in France, Belgium and Germany. F. Cailotto's co-authors include Rik Lories, Jean‐Yves Jouzeau, Silvia Monteagudo, Frank P. Luyten, Arnaud Bianchi, F.M. Cornelis, Peter Carmeliet, Sylvie Sébillaud, Patrick Netter and David Moulin and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and PLoS ONE.

In The Last Decade

F. Cailotto

31 papers receiving 519 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
F. Cailotto France 15 247 220 72 69 63 31 522
Zoltán Griger Hungary 12 223 0.9× 190 0.9× 87 1.2× 33 0.5× 46 0.7× 56 722
Angel Soto‐Hermida Spain 13 302 1.2× 292 1.3× 120 1.7× 37 0.5× 74 1.2× 17 539
Corinne Schiltz France 11 285 1.2× 133 0.6× 51 0.7× 83 1.2× 47 0.7× 11 534
Jianwei Lv China 12 209 0.8× 147 0.7× 78 1.1× 84 1.2× 16 0.3× 30 520
Heba Ismail United Kingdom 12 261 1.1× 212 1.0× 83 1.2× 52 0.8× 32 0.5× 24 519
Teresa Decklever United States 6 163 0.7× 85 0.4× 56 0.8× 51 0.7× 31 0.5× 7 461
Johanna Huhtakangas Finland 10 134 0.5× 104 0.5× 21 0.3× 46 0.7× 95 1.5× 24 520
Yu‐Tian Xiao China 12 245 1.0× 145 0.7× 141 2.0× 90 1.3× 13 0.2× 25 724

Countries citing papers authored by F. Cailotto

Since Specialization
Citations

This map shows the geographic impact of F. Cailotto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F. Cailotto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F. Cailotto more than expected).

Fields of papers citing papers by F. Cailotto

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F. Cailotto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F. Cailotto. The network helps show where F. Cailotto may publish in the future.

Co-authorship network of co-authors of F. Cailotto

This figure shows the co-authorship network connecting the top 25 collaborators of F. Cailotto. A scholar is included among the top collaborators of F. Cailotto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with F. Cailotto. F. Cailotto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cornelis, F.M., et al.. (2024). IGF1 drives Wnt-induced joint damage and is a potential therapeutic target for osteoarthritis. Nature Communications. 15(1). 9170–9170. 9 indexed citations
2.
Bourgaud, Frédéric, F. Cailotto, Pascal Reboul, et al.. (2022). Anti-Inflammatory Activity of Bryophytes Extracts in LPS-Stimulated RAW264.7 Murine Macrophages. Molecules. 27(6). 1940–1940. 14 indexed citations
3.
Cornelis, F.M., C. Cherifi, An Sermon, et al.. (2021). Hypoxia induces DOT1L in articular cartilage to protect against osteoarthritis. JCI Insight. 6(24). 19 indexed citations
4.
Jouzeau, Jean‐Yves, et al.. (2019). Secreted Frizzled‐related proteins (sFRPs) in osteo‐articular diseases: much more than simple antagonists of Wnt signaling?. FEBS Journal. 286(24). 4832–4851. 32 indexed citations
5.
Monteagudo, Silvia, et al.. (2018). SMOC2 inhibits calcification of osteoprogenitor and endothelial cells. PLoS ONE. 13(6). e0198104–e0198104. 15 indexed citations
6.
Monteagudo, Silvia, et al.. (2018). A cholecystokinin receptor antagonist inhibits the osteoarthritis features induced by GPR22 in vitro and in vivo. Osteoarthritis and Cartilage. 26. S32–S33. 2 indexed citations
7.
Lories, Rik, et al.. (2017). SMOC2, A secreted calcium-binding protein affects chondrogenic differentiation of precursor cells and homeostasis of adult cartilage. Osteoarthritis and Cartilage. 25. S154–S154. 1 indexed citations
8.
Monteagudo, Silvia, et al.. (2017). DOT1L safeguards cartilage homeostasis and protects against osteoarthritis. Nature Communications. 8(1). 108 indexed citations
9.
Cailotto, F., et al.. (2016). SMOC2, a secreted calcium-binding protein, is an inhibitor of osteogenesis and chondrogenesis. Osteoarthritis and Cartilage. 24. S141–S142. 2 indexed citations
10.
Bianchi, Arnaud, F. Cailotto, Adeline Gasser, et al.. (2016). Fibroblast Growth Factor 23 drives MMP13 expression in human osteoarthritic chondrocytes in a Klotho-independent manner. Osteoarthritis and Cartilage. 24(11). 1961–1969. 33 indexed citations
11.
Calebiro, Davide, et al.. (2016). A cholecystokinin receptor antagonist inhibits chondrocyte hypertrophy and protein kinase a activity decrease induced by activation of GPR22. Osteoarthritis and Cartilage. 24. S178–S179. 1 indexed citations
12.
Cailotto, F., et al.. (2016). Osteogenesis induced by frizzled-related protein (FRZB) is linked to the netrin-like domain. Laboratory Investigation. 96(5). 570–580. 17 indexed citations
13.
Cailotto, F., et al.. (2014). A5.12 SFRPS in cartilage biology: more than just WNT antagonists. Annals of the Rheumatic Diseases. 73. A67–A68. 1 indexed citations
15.
Cailotto, F., Frank P. Luyten, & Rik Lories. (2012). COG5 inhibition induces glycosylation defects affecting chondrogenesis and interfering with Wnt, but not BMP signaling. Osteoarthritis and Cartilage. 20. S142–S143. 2 indexed citations
16.
Cailotto, F., et al.. (2012). Tight regulation of wingless-type signaling in the articular cartilage - subchondral bone biomechanical unit: transcriptomics in Frzb-knockout mice. Arthritis Research & Therapy. 14(1). R16–R16. 36 indexed citations
17.
Cailotto, F., Sylvie Sébillaud, Patrick Netter, Jean‐Yves Jouzeau, & Arnaud Bianchi. (2010). The Inorganic Pyrophosphate Transporter ANK Preserves the Differentiated Phenotype of Articular Chondrocyte. Journal of Biological Chemistry. 285(14). 10572–10582. 23 indexed citations
18.
Cailotto, F., Marie‐Nathalie Kolopp‐Sarda, Véronique Latger‐Cannard, et al.. (2008). C-reactive protein (CRP) increases VEGF-A expression in monocytic cells via a PI3-kinase and ERK 1/2 signaling dependent pathway. Atherosclerosis. 200(2). 286–293. 35 indexed citations
19.
Cailotto, F., Arnaud Bianchi, Sylvie Sébillaud, et al.. (2007). Inorganic pyrophosphate generation by transforming growth factor-beta-1 is mainly dependent on ANK induction by Ras/Raf-1/extracellular signal-regulated kinase pathways in chondrocytes. Arthritis Research & Therapy. 9(6). R122–R122. 38 indexed citations
20.
Herbeth, Bernard, Frédéric Fumeron, Roberte Aubert, et al.. (2005). Polymorphism of the 5-HT2A receptor gene and food intakes in children and adolescents: the Stanislas Family Study. American Journal of Clinical Nutrition. 82(2). 467–470. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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