M. R. Kindt
- Surgery top 10%
- Endocrinology, Diabetes and Metabolism top 5%
- Molecular Biology
- Cancer Research
- Cardiology and Cardiovascular Medicine
- Co-authors
- D J RaderH. Bryan BrewerP LohseH Bryan BrewerLoren A. ZechDaniel J. RaderKatsunori IkewakiThomas Fairwell
- Topics
- Peroxisome Proliferator-Activated Receptors (5 papers)Lipoproteins and Cardiovascular Health (5 papers)Diabetes, Cardiovascular Risks, and Lipoproteins (4 papers)
- Partner nations
- United StatesItalyGermany
In The Last Decade
M. R. Kindt
12 papers receiving 435 citations
Peers
Comparison fields: 5 of 49
- Surgery 294
- Endocrinology, Diabetes and Metabolism 275
- Molecular Biology 137
- Cancer Research 120
- Cardiology and Cardiovascular Medicine 69
Countries citing papers authored by M. R. Kindt
This map shows the geographic impact of M. R. Kindt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. R. Kindt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. R. Kindt more than expected).
Fields of papers citing papers by M. R. Kindt
This network shows the impact of papers produced by M. R. Kindt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. R. Kindt. The network helps show where M. R. Kindt may publish in the future.
Co-authorship network of co-authors of M. R. Kindt
This figure shows the co-authorship network connecting the top 25 collaborators of M. R. Kindt. A scholar is included among the top collaborators of M. R. Kindt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. R. Kindt. M. R. Kindt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | LES GRANDS-PARENTS: AGENTS D'UNE SOCIALISATION SPÉCIFIQUE DE LEURS PETITS-ENFANTS CONSOMMATEURS? | 2 |
| 2 | 103 | |
| 3 | 63 | |
| 4 | 14 | |
| 5 | 23 | |
| 6 | 32 | |
| 7 | 53 | |
| 8 | 13 | |
| 9 | 40 | |
| 10 | In vivo protein metabolism utilizing stable isotopes and mass spectrometry: a new approach to the study of mutant proteins in humans. | 6 |
| 11 | 60 | |
| 12 | 34 |
About M. R. Kindt
M. R. Kindt is a scholar working on Cancer Research, Endocrinology, Diabetes and Metabolism and Cell Biology, having authored 12 papers that have together received 443 indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (5 papers), Lipoproteins and Cardiovascular Health (5 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (4 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (275 citations), Cancer Research (120 citations) and Surgery (294 citations). M. R. Kindt has collaborated with scholars based in United States, Italy and Germany. Frequent co-authors include D J Rader, H. Bryan Brewer, P Lohse, H Bryan Brewer, Loren A. Zech, Daniel J. Rader, Katsunori Ikewaki, Thomas Fairwell, Juergen R. Schaefer and Glenda D. Talley. Their work appears in journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.