M Panzica

927 total citations · 1 hit paper
10 papers, 822 citations indexed

About

M Panzica is a scholar working on Molecular Biology, Hepatology and Oncology. According to data from OpenAlex, M Panzica has authored 10 papers receiving a total of 822 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Hepatology and 3 papers in Oncology. Recurrent topics in M Panzica's work include Liver physiology and pathology (4 papers), Digestive system and related health (2 papers) and Pancreatic function and diabetes (2 papers). M Panzica is often cited by papers focused on Liver physiology and pathology (4 papers), Digestive system and related health (2 papers) and Pancreatic function and diabetes (2 papers). M Panzica collaborates with scholars based in United States, Netherlands and Germany. M Panzica's co-authors include Janet E. Mead, L Braun, N Fausto, Graeme I. Bell, R Mikumo, N.L. Thompson, Paul Yaswen, Christos J. Petropoulos, Nelson Fausto and Nancy L. Thompson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Biochemical Journal.

In The Last Decade

M Panzica

10 papers receiving 805 citations

Hit Papers

Transforming growth factor beta mRNA increases during liv... 1988 2026 2000 2013 1988 100 200 300 400

Peers

M Panzica
Comparison fields: 5 of 69
  • Hepatology 445
  • Molecular Biology 364
  • Surgery 305
  • Oncology 177
  • Epidemiology 155
Replace Wendy Garrison with:
Wendy Garrison United States
Yukio Kume Japan
Emma Du United States
Alexandra A. Henrion France
George G. Skouteris Germany
Yuji Matsuzawa Japan
Michèle Egger Switzerland
Chenzhi Hao China
Jamil Haque United States
F. Mihaimeed United Kingdom
Wendy Garrison United States View profile →
Citations per field, relative to M Panzica
M Panzica · 1×
Citations per year, relative to M Panzica
M Panzica · 1×

Countries citing papers authored by M Panzica

Since Specialization
Citations

This map shows the geographic impact of M Panzica's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M Panzica with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M Panzica more than expected).

Fields of papers citing papers by M Panzica

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M Panzica. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M Panzica. The network helps show where M Panzica may publish in the future.

Co-authorship network of co-authors of M Panzica

This figure shows the co-authorship network connecting the top 25 collaborators of M Panzica. A scholar is included among the top collaborators of M Panzica based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M Panzica. M Panzica is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
# Work Indexed citations
1 5
2
Expression of a highly conserved oncofetal gene, TA1/E16, in human colon carcinoma and other primary cancers: homology to Schistosoma mansoni amino acid permease and Caenorhabditis elegans gene products.
74
3
TA1, a highly conserved oncofetal complementary DNA from rat hepatoma, encodes an integral membrane protein associated with liver development, carcinogenesis, and cell activation.
66
4 19
5
Spatiotemporal expression of two cell-cell adhesion molecule 105 isoforms during liver development.
16
6 87
7
Transforming growth factor beta mRNA increases during liver regeneration: a possible paracrine mechanism of growth regulation. breakdown →
427
8
Proto-oncogene expression and growth factors during liver regeneration.
47
9 11
10
Cell lineages in liver carcinogenesis: possible clues from studies of the distribution of alpha-fetoprotein RNA sequences in cell populations isolated from normal, regenerating, and preneoplastic rat livers.
70

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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