M Pagliari

612 total citations
26 papers, 391 citations indexed

About

M Pagliari is a scholar working on Hematology, Surgery and Genetics. According to data from OpenAlex, M Pagliari has authored 26 papers receiving a total of 391 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Hematology, 7 papers in Surgery and 6 papers in Genetics. Recurrent topics in M Pagliari's work include Platelet Disorders and Treatments (18 papers), Blood groups and transfusion (13 papers) and Heparin-Induced Thrombocytopenia and Thrombosis (7 papers). M Pagliari is often cited by papers focused on Platelet Disorders and Treatments (18 papers), Blood groups and transfusion (13 papers) and Heparin-Induced Thrombocytopenia and Thrombosis (7 papers). M Pagliari collaborates with scholars based in Italy, Netherlands and Iran. M Pagliari's co-authors include Flora Peyvandi, Luciano Baronciani, Giovanna Cozzi, Ilaria Mancini, Cristina Novembrino, Francesca Stufano, Francesco Blasi, Stefano Aliberti, Valentina De Zan and Andrea Artoni and has published in prestigious journals such as Blood, PLoS ONE and Thrombosis and Haemostasis.

In The Last Decade

M Pagliari

26 papers receiving 381 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
M Pagliari Italy 10 211 115 79 64 62 26 391
C. Bélizna France 8 65 0.3× 52 0.5× 64 0.8× 43 0.7× 40 0.6× 16 343
Eleonora Petito Italy 11 126 0.6× 77 0.7× 44 0.6× 46 0.7× 22 0.4× 25 306
Ana Belén Moreno‐Castaño Spain 12 61 0.3× 65 0.6× 74 0.9× 42 0.7× 42 0.7× 27 315
Jan Zlamal Germany 8 161 0.8× 89 0.8× 40 0.5× 18 0.3× 37 0.6× 25 287
Eva Leinøe Denmark 10 232 1.1× 17 0.1× 36 0.5× 21 0.3× 17 0.3× 31 338
Catherine Trichet France 9 169 0.8× 27 0.2× 34 0.4× 14 0.2× 12 0.2× 13 292
Muhua Cao China 11 72 0.3× 27 0.2× 139 1.8× 52 0.8× 20 0.3× 18 377
M. Le Besnerais France 11 54 0.3× 24 0.2× 48 0.6× 36 0.6× 28 0.5× 23 290
Andrea Duminuco Italy 11 128 0.6× 29 0.3× 32 0.4× 24 0.4× 15 0.2× 60 344
Sebastian Schubert Germany 10 65 0.3× 31 0.3× 112 1.4× 57 0.9× 10 0.2× 20 325

Countries citing papers authored by M Pagliari

Since Specialization
Citations

This map shows the geographic impact of M Pagliari's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M Pagliari with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M Pagliari more than expected).

Fields of papers citing papers by M Pagliari

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M Pagliari. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M Pagliari. The network helps show where M Pagliari may publish in the future.

Co-authorship network of co-authors of M Pagliari

This figure shows the co-authorship network connecting the top 25 collaborators of M Pagliari. A scholar is included among the top collaborators of M Pagliari based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M Pagliari. M Pagliari is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mollica, Luca, Luciano Baronciani, Giovanna Cozzi, et al.. (2024). Type 2M/2A von Willebrand disease: a shared phenotype between type 2M and 2A. Blood Advances. 8(7). 1725–1736. 8 indexed citations
2.
Baronciani, Luciano, M Pagliari, Giovanna Cozzi, et al.. (2023). Genetic determinants of enhanced von Willebrand factor clearance from plasma. Journal of Thrombosis and Haemostasis. 21(5). 1112–1122. 14 indexed citations
3.
Baronciani, Luciano, Francesca Stufano, Giovanna Cozzi, et al.. (2023). A comparative study in patients with type 2 von Willebrand disease using 4 different platelet-dependent von Willebrand factor assays. Research and Practice in Thrombosis and Haemostasis. 7(3). 100139–100139. 6 indexed citations
4.
Maino, Alberto, Saeed Sadeghian, Ilaria Mancini, et al.. (2023). Opium as a risk factor for early-onset coronary artery disease: Results from the Milano-Iran (MIran) study. PLoS ONE. 18(4). e0283707–e0283707. 7 indexed citations
5.
Agosti, Pasquale, Ilaria Mancini, Saeed Sadeghian, et al.. (2023). Factor V Leiden but not the factor II 20210G>A mutation is a risk factor for premature coronary artery disease: a case-control study in Iran. Research and Practice in Thrombosis and Haemostasis. 7(1). 100048–100048. 5 indexed citations
6.
Baronciani, Luciano, et al.. (2023). Reply to “Comment on: A comparative study in type 2 von Willebrand disease patients using four different platelet-dependent von Willebrand factor assays”. Research and Practice in Thrombosis and Haemostasis. 7(6). 102174–102174. 1 indexed citations
7.
Baronciani, Luciano, M Pagliari, Giovanna Cozzi, et al.. (2022). Phenotypic and genetic characterizations of the Milan cohort of von Willebrand disease type 2. Blood Advances. 6(13). 4031–4040. 17 indexed citations
8.
Pagliari, M, Luciano Baronciani, Chiara Cordiglieri, et al.. (2022). The dominant p.Thr274Pro mutation in the von Willebrand factor propeptide causes the von Willebrand disease type 1 phenotype in two unrelated patients. Haemophilia. 28(2). 292–300. 1 indexed citations
9.
Pagliari, M, Ilaria Mancini, Emanuela Pappalardo, et al.. (2021). Role of ADAMTS13, VWF and F8 genes in deep vein thrombosis. PLoS ONE. 16(10). e0258675–e0258675. 6 indexed citations
10.
Pagliari, M, Ilaria Mancini, Ida Martinelli, et al.. (2020). ADAMTS13 activity, high VWF and FVIII levels in the pathogenesis of deep vein thrombosis. Thrombosis Research. 197. 132–137. 15 indexed citations
11.
Mancini, Ilaria, Luciano Baronciani, Andrea Artoni, et al.. (2020). The ADAMTS13‐von Willebrand factor axis in COVID‐19 patients. Journal of Thrombosis and Haemostasis. 19(2). 513–521. 149 indexed citations
12.
Ferrari, Barbara, et al.. (2019). Risk of diagnostic delay in congenital thrombotic thrombocytopenic purpura. Journal of Thrombosis and Haemostasis. 17(4). 666–669. 7 indexed citations
13.
Fasulo, Maria Rosaria, Eugenia Biguzzi, Maria Abbattista, et al.. (2017). The ISTH Bleeding Assessment Tool and the risk of future bleeding. Journal of Thrombosis and Haemostasis. 16(1). 125–130. 28 indexed citations
14.
Pagliari, M, Luca A. Lotta, Hugoline G. de Haan, et al.. (2016). Next-Generation Sequencing and In Vitro Expression Study of ADAMTS13 Single Nucleotide Variants in Deep Vein Thrombosis. PLoS ONE. 11(11). e0165665–e0165665. 7 indexed citations
15.
Stufano, Francesca, Luciano Baronciani, M Pagliari, et al.. (2015). Evaluation of an heterogeneous group of patients with von Willebrand disease using an assay alternative to ristocetin induced platelet agglutination. Journal of Thrombosis and Haemostasis. 13(10). 1806–1814. 8 indexed citations
16.
Pagliari, M, Safwat Abdel‐Azeim, Edrisse Chermak, et al.. (2015). The D173G mutation in ADAMTS-13 causes a severe form of congenital thrombotic thrombocytopenic purpura. Thrombosis and Haemostasis. 115(1). 51–62. 15 indexed citations
17.
Bucciarelli, Paolo, Simona Maria Siboni, Francesca Stufano, et al.. (2014). Predictors of von Willebrand disease diagnosis in individuals with borderline von Willebrand factor plasma levels. Journal of Thrombosis and Haemostasis. 13(2). 228–236. 14 indexed citations
18.
Scaglione, Giovanni Luca, Stefano Lancellotti, Massimiliano Papi, et al.. (2013). The type 2B p.R1306W natural mutation of von Willebrand factor dramatically enhances the multimer sensitivity to shear stress. Journal of Thrombosis and Haemostasis. 11(9). 1688–1698. 13 indexed citations
19.
Pagliari, M, Luciano Baronciani, Silvia La Marca, et al.. (2013). A synonymous (c.3390C>T) or a splice‐site (c.3380‐2A>G) mutation causes exon 26 skipping in four patients with von Willebrand disease (2A/IIE). Journal of Thrombosis and Haemostasis. 11(7). 1251–1259. 14 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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