M. Löffler

21 papers receiving 628 citations

Peers

M. Löffler
Comparison fields: 5 of 90
  • Physiology 41
  • Clinical Biochemistry 48
  • Cancer Research 104
  • Molecular Biology 444
  • Biological Psychiatry 12
Replace Elizabeth A. Carrey with:
Elizabeth A. Carrey United Kingdom
Sally McCormick United States
Alexis Salas-Burgos Chile
J. Logan Irvin United States
Geen-Dong Chang Taiwan
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Citations per field
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Citations per year

Countries citing papers authored by M. Löffler

Since Specialization
Citations

This map shows the geographic impact of M. Löffler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. Löffler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. Löffler more than expected).

Fields of papers citing papers by M. Löffler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. Löffler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. Löffler. The network helps show where M. Löffler may publish in the future.

Co-authorship network

The 25 scholars most cited alongside M. Löffler, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with M. Löffler Line = papers co-authored together M. Löffler links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 201821
2 201655
3 201011
4 20063
5 2005205
6 2004103
7 200419
8 200224
9 20023
10 19982
11 199728
12 199437
13 199213
14 199110
15 19876
16 198731
17
Uridine, but not cytidine can sustain growth of Ehrlich ascites tumor cells in glucose-deprived medium with altered proliferation kinetics.
198515
18
Characterization of the deoxynucleoside-dependent reversal of hypoxia-induced inhibition of cell cycle progression in Ehrlich ascites tumor cells.
198513
19 198214
20 19815

About M. Löffler

M. Löffler is a scholar working on Biotechnology, Cancer Research, Clinical Biochemistry, Physiology and Biochemistry, having authored 21 papers that have together received 640 indexed citations. Recurring topics across this work include Biochemical and Molecular Research (11 papers), Cancer, Hypoxia, and Metabolism (6 papers), Cancer Research and Treatments (4 papers), HIV/AIDS drug development and treatment (4 papers), Neonatal Health and Biochemistry (3 papers), Metabolism and Genetic Disorders (3 papers), Pneumocystis jirovecii pneumonia detection and treatment (3 papers) and Polyamine Metabolism and Applications (2 papers). The work is most often cited by research in Physiology (41 citations), Clinical Biochemistry (48 citations), Cancer Research (104 citations), Molecular Biology (444 citations) and Biological Psychiatry (12 citations). M. Löffler has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Elke Zameitat, Anthony M. Marinaki, H. Anne Simmonds, F Schneider, Wolfgang Knecht, Øystein Åmellem, Jean‐Baptiste Coutelis, Jure Piškur, Zoran Gojković and Kasper Møller. Their work appears in journals such as Nucleosides Nucleotides & Nucleic Acids, Cell Proliferation, Experimental Cell Research, Molecular Genetics and Genomics and British Journal of Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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