M. J. Dunne

563 total citations
19 papers, 485 citations indexed

About

M. J. Dunne is a scholar working on Surgery, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, M. J. Dunne has authored 19 papers receiving a total of 485 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Surgery, 8 papers in Molecular Biology and 4 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in M. J. Dunne's work include Pancreatic function and diabetes (8 papers), Ion channel regulation and function (7 papers) and Adenosine and Purinergic Signaling (3 papers). M. J. Dunne is often cited by papers focused on Pancreatic function and diabetes (8 papers), Ion channel regulation and function (7 papers) and Adenosine and Purinergic Signaling (3 papers). M. J. Dunne collaborates with scholars based in United Kingdom and United States. M. J. Dunne's co-authors include Ole H. Petersen, Susanne G. Straub, R. F. L. James, Geoffrey W.G. Sharp, R M Shepherd, Karen E. Cosgrove, Mary Collins, Frank L. Iber, Michael J. Owen and Wendy M. Macfarlane and has published in prestigious journals such as The EMBO Journal, Nature Methods and Diabetes.

In The Last Decade

M. J. Dunne

18 papers receiving 456 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
M. J. Dunne United Kingdom 10 252 244 127 94 77 19 485
J.J. Calvo Spain 13 158 0.6× 216 0.9× 70 0.6× 66 0.7× 34 0.4× 45 481
Jean‐Michel Garel France 13 179 0.7× 60 0.2× 67 0.5× 168 1.8× 32 0.4× 28 406
M Okamoto Japan 14 137 0.5× 122 0.5× 104 0.8× 30 0.3× 23 0.3× 32 541
Yanhong Wang China 11 182 0.7× 103 0.4× 41 0.3× 48 0.5× 75 1.0× 36 500
Etelvina Andreu Spain 9 244 1.0× 258 1.1× 151 1.2× 35 0.4× 19 0.2× 18 501
Elia Martha Pérez-Armendáriz Mexico 13 365 1.4× 155 0.6× 85 0.7× 49 0.5× 10 0.1× 21 574
G Tamburrano Italy 13 153 0.6× 197 0.8× 370 2.9× 24 0.3× 23 0.3× 32 566
Akihiro Yamauchi Japan 15 170 0.7× 66 0.3× 54 0.4× 184 2.0× 24 0.3× 29 481
Rachel V. Richardson United Kingdom 10 228 0.9× 82 0.3× 81 0.6× 36 0.4× 16 0.2× 10 458
M. Lange Germany 12 83 0.3× 95 0.4× 189 1.5× 85 0.9× 13 0.2× 19 482

Countries citing papers authored by M. J. Dunne

Since Specialization
Citations

This map shows the geographic impact of M. J. Dunne's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. J. Dunne with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. J. Dunne more than expected).

Fields of papers citing papers by M. J. Dunne

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. J. Dunne. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. J. Dunne. The network helps show where M. J. Dunne may publish in the future.

Co-authorship network of co-authors of M. J. Dunne

This figure shows the co-authorship network connecting the top 25 collaborators of M. J. Dunne. A scholar is included among the top collaborators of M. J. Dunne based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. J. Dunne. M. J. Dunne is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Li, Haiyin, et al.. (2025). SpaceBar enables single-cell-resolution clone tracing with imaging-based spatial transcriptomics. Nature Methods. 23(2). 328–333.
2.
Dunne, M. J., et al.. (2001). Electrophysiology of the -cell and mechanisms of inhibition of insulin release.. 1 indexed citations
3.
Dunne, M. J., et al.. (2000). Persistent hyperinsulinaemic hyperglycaemia of infancy-derived cells; implications for beta-cells that replicate in vitro. Journal of Molecular Endocrinology. 24(3). 313–320. 4 indexed citations
4.
Dunne, M. J.. (2000). Ions, genes and insulin release: from basic science to clinical disease Based on the 1998 R. D. Lawrence Lecture. Diabetic Medicine. 17(2). 91–104. 15 indexed citations
5.
Macfarlane, Wendy M., R M Shepherd, Karen E. Cosgrove, et al.. (2000). Glucose modulation of insulin mRNA levels is dependent on transcription factor PDX-1 and occurs independently of changes in intracellular Ca2+.. Diabetes. 49(3). 418–423. 57 indexed citations
6.
McClenaghan, Neville H., Karen E. Cosgrove, R M Shepherd, et al.. (1999). ATP-sensitive potassium channels and efaroxan-induced insulin release in the electrofusion-derived BRIN-BD11 beta-cell line.. Diabetes. 48(12). 2349–2357. 29 indexed citations
7.
Straub, Susanne G., R. F. L. James, M. J. Dunne, & Geoffrey W.G. Sharp. (1998). Glucose activates both K(ATP) channel-dependent and K(ATP) channel-independent signaling pathways in human islets.. Diabetes. 47(5). 758–763. 71 indexed citations
8.
Harding, E. A., Cheikhou Kane, Roger F.L. James, N J M London, & M. J. Dunne. (1997). Modulation of Three Types of Potassium Selective Channels by NAD and Other Pyridine Nucleotides in Human Pancreatic β-Cells. Advances in experimental medicine and biology. 426. 43–50. 3 indexed citations
9.
Dunne, M. J., et al.. (1997). Nature's KATP-Channel Knockout. Physiology. 12(5). 197–203. 6 indexed citations
10.
Dunne, M. J.. (1994). Phorbol myristate acetate and ATP-sensitive potassium channels in insulin-secreting cells. American Journal of Physiology-Cell Physiology. 267(2). C501–C506. 23 indexed citations
11.
Jaggar, Jonathan H., et al.. (1993). Interaction of diazoxide and cromakalim with ATP-regulated K+ channels in rodent and clonal insulin-secreting cells. Journal of Molecular Endocrinology. 10(1). 59–70. 5 indexed citations
13.
Dunne, M. J. & Ole H. Petersen. (1991). Potassium selective ion channels in insulin-secreting cells: physiology, pharmacology and their role in stimulus-secretion coupling. Biochimica et Biophysica Acta (BBA) - Reviews on Biomembranes. 1071(1). 67–82. 139 indexed citations
14.
Dunne, M. J.. (1990). Nutrient and pharmacological stimulation of insulin‐secreting cells: marked differences in the onset of electrical activity. Experimental Physiology. 75(6). 771–777. 8 indexed citations
15.
Dunne, M. J., David I. Yule, D.V. Gallacher, & Ole H. Petersen. (1989). Cromakalim (BRL 34915) and diazoxide activate ATP-regulated potassium channels in insulin-secreting cells. Pflügers Archiv - European Journal of Physiology. 414(S1). S154–S155. 18 indexed citations
16.
Seckl, Jonathan R., et al.. (1988). Opioid-mediated inhibition of oxytocin during insulin-induced hypoglycemic stimulation of vasopressin in man. European Journal of Endocrinology. 118(1). 77–81. 8 indexed citations
17.
Dunne, M. J., et al.. (1987). Gallbladder volume and emptying in insulin-requiring male diabetics. Digestive Diseases and Sciences. 32(8). 824–828. 32 indexed citations
18.
Collins, Mary, A. Maija Kissonerghis, M. J. Dunne, et al.. (1985). Transcripts from an aberrantly re-arranged human T-cell receptor beta-chain gene.. The EMBO Journal. 4(5). 1211–1215. 25 indexed citations
19.
Collins, Mary, Peter N. Goodfellow, M. J. Dunne, et al.. (1984). A human T-cell antigen receptor beta chain gene maps to chromosome 7.. The EMBO Journal. 3(10). 2347–2349. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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