M. E. Cooper

2.5k total citations · 1 hit paper
13 papers, 2.0k citations indexed

About

M. E. Cooper is a scholar working on Cardiology and Cardiovascular Medicine, Endocrinology, Diabetes and Metabolism and Nephrology. According to data from OpenAlex, M. E. Cooper has authored 13 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cardiology and Cardiovascular Medicine, 5 papers in Endocrinology, Diabetes and Metabolism and 4 papers in Nephrology. Recurrent topics in M. E. Cooper's work include Blood Pressure and Hypertension Studies (5 papers), Renin-Angiotensin System Studies (4 papers) and Chronic Kidney Disease and Diabetes (3 papers). M. E. Cooper is often cited by papers focused on Blood Pressure and Hypertension Studies (5 papers), Renin-Angiotensin System Studies (4 papers) and Chronic Kidney Disease and Diabetes (3 papers). M. E. Cooper collaborates with scholars based in Australia, Denmark and United States. M. E. Cooper's co-authors include Dick de Zeeuw, B. M. Brenner, S Oren, I. Tikkanen, R. W. E. Watts, Steen Neldam, R J Viskoper, C. E. Mogensen, Terri J. Allen and Geoffrey Boner and has published in prestigious journals such as Diabetes, Kidney International and The American Journal of Medicine.

In The Last Decade

M. E. Cooper

13 papers receiving 1.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Randomised controlled trial of dual blockade of renin-angiotensin system in patients with hypertension, microalbuminuria, and non-insulin dependent diabetes: the candesartan and lisinopril microalbuminuria (CALM) study

2000 745 citations C. E. Mogensen, Steen Neldam et al. BMJ profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
M. E. Cooper Australia	12	1.1k	842	771	229	202	13	2.0k
H.-H. Parving Denmark	18	769 0.7×	989 1.2×	735 1.0×	231 1.0×	116 0.6×	30	2.1k
Hans Henrik Parving Denmark	11	743 0.7×	582 0.7×	1.2k 1.6×	264 1.2×	151 0.7×	12	2.9k
Allan Kofoed‐Enevoldsen Denmark	20	914 0.8×	962 1.1×	1.0k 1.3×	280 1.2×	240 1.2×	49	2.6k
C. E. Mogensen Denmark	14	1.4k 1.2×	1.1k 1.3×	1.3k 1.6×	162 0.7×	146 0.7×	17	2.6k
U. Smidt Denmark	25	1.4k 1.2×	1.3k 1.5×	1.2k 1.5×	249 1.1×	137 0.7×	38	2.7k
Mari-Anne Gall Denmark	17	725 0.6×	797 0.9×	667 0.9×	253 1.1×	103 0.5×	26	2.1k
E. R. Mathiesen Denmark	15	730 0.6×	1.3k 1.5×	1.1k 1.4×	141 0.6×	165 0.8×	30	2.3k
G C Viberti United Kingdom	13	701 0.6×	989 1.2×	1.3k 1.7×	176 0.8×	183 0.9×	21	2.4k
Eberhard Ritz Germany	12	502 0.4×	513 0.6×	864 1.1×	317 1.4×	123 0.6×	12	1.8k
Richard E. Gilbert Australia	24	681 0.6×	839 1.0×	758 1.0×	579 2.5×	165 0.8×	44	2.2k

Countries citing papers authored by M. E. Cooper

Since Specialization

Citations

This map shows the geographic impact of M. E. Cooper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. E. Cooper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. E. Cooper more than expected).

Fields of papers citing papers by M. E. Cooper

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. E. Cooper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. E. Cooper. The network helps show where M. E. Cooper may publish in the future.

Co-authorship network of co-authors of M. E. Cooper

This figure shows the co-authorship network connecting the top 25 collaborators of M. E. Cooper. A scholar is included among the top collaborators of M. E. Cooper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. E. Cooper. M. E. Cooper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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13 of 13 papers shown

Sourris, Karly C., Jasmine G. Lyons, Sonia Dougherty, et al.. (2013). Plasma advanced glycation end products (AGEs) and NF-κB activity are independent determinants of diastolic and pulse pressure. Clinical Chemistry and Laboratory Medicine (CCLM). 52(1). 129–38. 16 indexed citations

Thomas, Merlin C., M. E. Cooper, Kasper Rossing, & H.‐H. Parving. (2006). Anaemia in diabetes: is there a rationale to TREAT?. Diabetologia. 49(6). 1151–1157. 67 indexed citations

Boner, Geoffrey, M. E. Cooper, B. M. Brenner, et al.. (2005). Adverse effects of left ventricular hypertrophy in the reduction of endpoints in NIDDM with the angiotensin II antagonist losartan (RENAAL) study. Diabetologia. 48(10). 1980–1987. 32 indexed citations

Tikellis, Chris, C. I. Johnston, Josephine M. Forbes, et al.. (2004). Identification of angiotensin converting enzyme 2 in the rodent retina. Current Eye Research. 29(6). 419–427. 66 indexed citations

Brenner, B. M., M. E. Cooper, & Dick de Zeeuw. (2002). Effects of losartan on renal and cardiovascular outcomes in patients with type 2 diabetes and nephropathy. ACC Current Journal Review. 11(1). 26–26. 416 indexed citations

Cooper, M. E., Geoffrey Boner, Zemin Cao, et al.. (2001). Irbesartan normalises the deficiency in glomerular nephrin expression in a model of diabetes and hypertension. Diabetologia. 44(7). 874–877. 213 indexed citations

Mogensen, C. E., Steen Neldam, I. Tikkanen, et al.. (2000). Randomised controlled trial of dual blockade of renin-angiotensin system in patients with hypertension, microalbuminuria, and non-insulin dependent diabetes: the candesartan and lisinopril microalbuminuria (CALM) study. BMJ. 321(7274). 1440–1444. 745 indexed citations breakdown →

Cooper, M. E., Vicki Thallas, Josephine M. Forbes, et al.. (2000). The cross-link breaker, N-phenacylthiazolium bromide prevents vascular advanced glycation end-product accumulation. Diabetologia. 43(5). 660–664. 89 indexed citations

Vranes, Dimitria, M. E. Cooper, & Rodney J. Dilley. (1999). Cellular Mechanisms of Diabetic Vascular Hypertrophy. Microvascular Research. 57(1). 8–18. 53 indexed citations

10.

Gilbert, Richard E., Angela Cox, Linlin Wu, et al.. (1998). Expression of transforming growth factor-beta1 and type IV collagen in the renal tubulointerstitium in experimental diabetes: effects of ACE inhibition.. Diabetes. 47(3). 414–422. 184 indexed citations

11.

Cooper, M. E.. (1997). Antihypertensive Therapy and Diabetic Microvascular Disease. Clinical and Experimental Hypertension. 19(5-6). 769–778. 4 indexed citations

12.

Cooper, M. E., et al.. (1992). Antihypertensive therapy in a model combining spontaneous hypertension with diabetes. Kidney International. 41(4). 898–903. 45 indexed citations

13.

Seeman, Ego, M. E. Cooper, John L. Hopper, et al.. (1988). Effect of early menopause on bone mass in normal women and patients with qsteoporosis. The American Journal of Medicine. 85(2). 213–216. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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