M. C. Capogrossi

3.2k total citations
42 papers, 2.5k citations indexed

About

M. C. Capogrossi is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Physiology. According to data from OpenAlex, M. C. Capogrossi has authored 42 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 8 papers in Cardiology and Cardiovascular Medicine and 7 papers in Physiology. Recurrent topics in M. C. Capogrossi's work include Cardiac electrophysiology and arrhythmias (6 papers), Ion channel regulation and function (6 papers) and Virus-based gene therapy research (5 papers). M. C. Capogrossi is often cited by papers focused on Cardiac electrophysiology and arrhythmias (6 papers), Ion channel regulation and function (6 papers) and Virus-based gene therapy research (5 papers). M. C. Capogrossi collaborates with scholars based in Italy, United States and Switzerland. M. C. Capogrossi's co-authors include Giulio Pompilio, Patrizia Nigro, Edward G. Lakatta, Chiara Cencioni, Michael D. Stern, Monica Napolitano, Harold A. Spurgeon, Alessandra Magenta, Fabio Martelli and Pasquale Fasanaro and has published in prestigious journals such as Circulation, Journal of Clinical Investigation and Nature Medicine.

In The Last Decade

M. C. Capogrossi

41 papers receiving 2.5k citations

Peers

M. C. Capogrossi
M. C. Capogrossi
Citations per year, relative to M. C. Capogrossi M. C. Capogrossi (= 1×) peers Michael P. Czubryt

Countries citing papers authored by M. C. Capogrossi

Since Specialization
Citations

This map shows the geographic impact of M. C. Capogrossi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. C. Capogrossi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. C. Capogrossi more than expected).

Fields of papers citing papers by M. C. Capogrossi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. C. Capogrossi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. C. Capogrossi. The network helps show where M. C. Capogrossi may publish in the future.

Co-authorship network of co-authors of M. C. Capogrossi

This figure shows the co-authorship network connecting the top 25 collaborators of M. C. Capogrossi. A scholar is included among the top collaborators of M. C. Capogrossi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. C. Capogrossi. M. C. Capogrossi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
D'agostino, M, Daniela Lulli, Naomi De Luca, et al.. (2025). miR-200c inhibition and catalase accelerate diabetic wound healing. Journal of Biomedical Science. 32(1). 21–21. 8 indexed citations
2.
Magenta, Alessandra, Mariangela Pia Dagostino, Luca Di Vito, et al.. (2019). The Oxidative Stress-Induced miR-200c Is Upregulated in Psoriasis and Correlates with Disease Severity and Determinants of Cardiovascular Risk. Oxidative Medicine and Cellular Longevity. 2019. 1–12. 31 indexed citations
3.
Piccioni, Andrea, Eleonora Gaetani, Mariangela Palladino, et al.. (2014). Sonic hedgehog gene therapy increases the ability of the dystrophic skeletal muscle to regenerate after injury. Gene Therapy. 21(4). 413–421. 24 indexed citations
4.
Nigro, Patrizia, Giulio Pompilio, & M. C. Capogrossi. (2013). Cyclophilin A: a key player for human disease. Cell Death and Disease. 4(10). e888–e888. 351 indexed citations
5.
Cencioni, Chiara, M. C. Capogrossi, & Monica Napolitano. (2012). The SDF-1/CXCR4 axis in stem cell preconditioning. Cardiovascular Research. 94(3). 400–407. 124 indexed citations
6.
Magenta, Alessandra, Chiara Cencioni, Pasquale Fasanaro, et al.. (2011). miR-200c is upregulated by oxidative stress and induces endothelial cell apoptosis and senescence via ZEB1 inhibition. Cell Death and Differentiation. 18(10). 1628–1639. 376 indexed citations
7.
Marchetti, Carlo, Anna Di Carlo, Francesco Facchiano, et al.. (2011). High mobility group box 1 is a novel substrate of dipeptidyl peptidase-IV. Diabetologia. 55(1). 236–244. 48 indexed citations
8.
Colussi, Claudia, Cristina Banfi, Maura Brioschi, et al.. (2010). Proteomic profile of differentially expressed plasma proteins from dystrophic mice and following suberoylanilide hydroxamic acid treatment. PROTEOMICS - CLINICAL APPLICATIONS. 4(1). 71–83. 30 indexed citations
9.
Cappuzzello, Claudia, Roberta Melchionna, Arduino A. Mangoni, et al.. (2007). Role of rat α adducin in angiogenesis: Null effect of the F316Y polymorphism. Cardiovascular Research. 75(3). 608–617. 9 indexed citations
10.
Abbate, Antonio, Federica Limana, M. C. Capogrossi, et al.. (2006). Cyclo-oxygenase-2 (COX-2) inhibition reduces apoptosis in acute myocardial infarction. APOPTOSIS. 11(6). 1061–1063. 20 indexed citations
11.
Matsumoto, Kenji, et al.. (2006). Papilloma protein E6 abrogates shear stress-dependent survival in human endothelial cells: Evidence for specialized functions of paxillin. Cardiovascular Research. 70(3). 578–588. 8 indexed citations
12.
Pompilio, Giulio, Aldo Cannata, Maurizio Pesce, M. C. Capogrossi, & Paolo Biglioli. (2005). Long-lasting improvement of myocardial perfusion and chronic refractory angina after autologous intramyocardial PBSC transplantation. Cytotherapy. 7(6). 494–496. 5 indexed citations
13.
Melillo, G, Federica Serino, Corrado Cirielli, & M. C. Capogrossi. (2004). Gene Therapy in Peripheral Artery Disease. PubMed. 4(3). 295–300. 4 indexed citations
14.
Mangoni, Antonella, Giovanna Zambruno, Gaia Spinetti, et al.. (2002). Adenovirus-mediated VEGF165 gene transfer enhances wound healing by promoting angiogenesis in CD1 diabetic mice. Gene Therapy. 9(19). 1271–1277. 104 indexed citations
15.
Gaetano, Carlo, Assunta Catalano, Roberta Palumbo, et al.. (2000). Transcriptionally active drugs improve adenovirus vector performance in vitro and in vivo. Gene Therapy. 7(19). 1624–1630. 46 indexed citations
16.
Kovesdi, Imre, et al.. (1999). Vascular permeability effect of adenovirus-mediated vascular endothelial growth factor gene transfer to the rabbit and rat skeletal muscle. Journal of Thoracic and Cardiovascular Surgery. 118(2). 339–347. 23 indexed citations
17.
Cirielli, Corrado, M. C. Capogrossi, & Antonino Passaniti. (1997). Anti-tumor gene therapy. Journal of Neuro-Oncology. 31(1-2). 217–223. 4 indexed citations
18.
Gambassi, G, et al.. (1994). Temperature modulates calcium homeostasis and ventricular arrhythmias in myocardial preparations. Cardiovascular Research. 28(3). 391–399. 22 indexed citations
19.
Пили, Роберто, Stefano Corda, Antonino Passaniti, et al.. (1993). Endothelial cell Ca2+ increases upon tumor cell contact and modulates cell-cell adhesion.. Journal of Clinical Investigation. 92(6). 3017–3022. 18 indexed citations
20.
Stern, Michael D., M. C. Capogrossi, & Edward G. Lakatta. (1988). Spontaneous calcium release from the sarcoplasmic reticulum in myocardial cells: mechanisms and consequences. Cell Calcium. 9(5-6). 247–256. 100 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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