M. B. Coleman

442 total citations
17 papers, 377 citations indexed

About

M. B. Coleman is a scholar working on Genetics, Hematology and Physiology. According to data from OpenAlex, M. B. Coleman has authored 17 papers receiving a total of 377 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Genetics, 13 papers in Hematology and 6 papers in Physiology. Recurrent topics in M. B. Coleman's work include Hemoglobinopathies and Related Disorders (15 papers), Iron Metabolism and Disorders (10 papers) and Erythrocyte Function and Pathophysiology (6 papers). M. B. Coleman is often cited by papers focused on Hemoglobinopathies and Related Disorders (15 papers), Iron Metabolism and Disorders (10 papers) and Erythrocyte Function and Pathophysiology (6 papers). M. B. Coleman collaborates with scholars based in United States, Canada and Iran. M. B. Coleman's co-authors include Martin H. Steinberg, Junius G. Adams, MH Steinberg, PF Milner, SA Liebhaber, MH Steinberg, Maria W. Plonczynski, F E Cash, A. Merat and Zohreh Mostafavi‐Pour and has published in prestigious journals such as Journal of Clinical Investigation, Blood and British Journal of Haematology.

In The Last Decade

M. B. Coleman

17 papers receiving 363 citations

Peers

M. B. Coleman
MH Steinberg United States
T. H. J. Huisman United States
WA Schroeder United States
L. Perseu Italy
C Altay United States
FF Chehab United States
MH Steinberg United States
M. B. Coleman
Citations per year, relative to M. B. Coleman M. B. Coleman (= 1×) peers MH Steinberg

Countries citing papers authored by M. B. Coleman

Since Specialization
Citations

This map shows the geographic impact of M. B. Coleman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M. B. Coleman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M. B. Coleman more than expected).

Fields of papers citing papers by M. B. Coleman

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by M. B. Coleman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M. B. Coleman. The network helps show where M. B. Coleman may publish in the future.

Co-authorship network of co-authors of M. B. Coleman

This figure shows the co-authorship network connecting the top 25 collaborators of M. B. Coleman. A scholar is included among the top collaborators of M. B. Coleman based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with M. B. Coleman. M. B. Coleman is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Wilson, David M., et al.. (1999). Genetic studies suggest a multicentric origin for Hb G-coushatta [β22(B4)Glu→Ala]. Hemoglobin. 23(1). 57–67. 6 indexed citations
2.
Merritt, Diane F., Richard T. Jones, Charlotte Head, et al.. (1997). HB Seal Rock [(α2)142 Term→glu, Codon 142 TAA→GAA]: An Extended α Chain Variant Associated with Anemia, Microcytosis, and α-Thalassemia-2 (-3.7 KB). Hemoglobin. 21(4). 331–344. 11 indexed citations
3.
Coleman, M. B., Junius G. Adams, Martin H. Steinberg, & William P. Winter. (1994). A four base pair deletion 5′ to the AγT gene is associated not only with decreased expression of the AγT‐globin gene, but also of the Gγ‐globin gene in cis. American Journal of Hematology. 47(4). 307–311. 9 indexed citations
4.
Waye, John S., Barry Eng, M. B. Coleman, Martin H. Steinberg, & Blanche P. Alter. (1994). δβ-Thalassemia in an African-American: Identification of the deletion endpoints and PCR-Based diagnosis. Hemoglobin. 18(6). 389–399. 3 indexed citations
5.
Coleman, M. B., Junius G. Adams, Martin H. Steinberg, et al.. (1993). GγAγ+) hereditary persistence of fetal hemoglobin: The Gγ – 158 C → T mutation in cis to the − 175 T → C mutation of the Aγ‐globin gene results in increased Gγ‐globin synthesis. American Journal of Hematology. 42(2). 186–190. 12 indexed citations
6.
7.
Merat, A., et al.. (1993). β-Thalassemia in Southwestern Iran. Hemoglobin. 17(5). 427–437. 40 indexed citations
8.
Coleman, M. B., et al.. (1992). The -87 (C→A) β+-Thalassemia Mutation in a Black Family. Hemoglobin. 16(5). 399–401. 7 indexed citations
9.
Coleman, M. B., et al.. (1991). A four-base deletion 5' to the A gamma globin gene is a common polymorphism.. Blood. 78(9). 2473–4. 8 indexed citations
12.
Liebhaber, SA, M. B. Coleman, Junius G. Adams, F E Cash, & Martin H. Steinberg. (1987). Molecular basis for nondeletion alpha-thalassemia in American blacks. Alpha 2(116GAG----UAG).. Journal of Clinical Investigation. 80(1). 154–159. 43 indexed citations
14.
Steinberg, MH, et al.. (1984). Effects of thalassemia and microcytosis on the hematologic and vasoocclusive severity of sickle cell anemia. Blood. 63(6). 1353–1360. 144 indexed citations
15.
Steinberg, MH, et al.. (1984). Effects of thalassemia and microcytosis on the hematologic and vasoocclusive severity of sickle cell anemia. Blood. 63(6). 1353–1360. 5 indexed citations
16.
Steinberg, Martin H., et al.. (1983). The effects of alpha‐thalassaemia in HbSC disease. British Journal of Haematology. 55(3). 487–492. 21 indexed citations
17.
Balducci, Lodovico, et al.. (1978). Erythroleukemia: In vitro studies of erythropoiesis. American Journal of Hematology. 5(4). 291–295. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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