Luis A. Williams

4.0k total citations · 3 hit papers
20 papers, 2.4k citations indexed

About

Luis A. Williams is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Genetics. According to data from OpenAlex, Luis A. Williams has authored 20 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 9 papers in Cellular and Molecular Neuroscience and 7 papers in Genetics. Recurrent topics in Luis A. Williams's work include Neurogenetic and Muscular Disorders Research (7 papers), Amyotrophic Lateral Sclerosis Research (6 papers) and Pluripotent Stem Cells Research (6 papers). Luis A. Williams is often cited by papers focused on Neurogenetic and Muscular Disorders Research (7 papers), Amyotrophic Lateral Sclerosis Research (6 papers) and Pluripotent Stem Cells Research (6 papers). Luis A. Williams collaborates with scholars based in United States, United Kingdom and Germany. Luis A. Williams's co-authors include Kevin Eggan, Steve S.W. Han, Brandi N. Davis‐Dusenbery, Lisa M. Farmer, Richard D. Vierstra, Sophia L. Stone, Judy Callis, Evangelos Kiskinis, Joseph R. Klim and Brian J. Wainger and has published in prestigious journals such as Cell, Nature Communications and Neuron.

In The Last Decade

Luis A. Williams

20 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Axonal Transport of TDP-43 mRNA Granules Is Impaired by ALS-Causing Mutations

2014 497 citations Nael H. Alami, Rebecca B. Smith et al. Neuron profile →
Intrinsic Membrane Hyperexcitability of Amyotrophic Lateral Sclerosis Patient-Derived Motor Neurons

2014 481 citations Brian J. Wainger, Evangelos Kiskinis et al. Cell Reports profile →
ALS-implicated protein TDP-43 sustains levels of STMN2, a mediator of motor neuron growth and repair

2019 345 citations Joseph R. Klim, Luis A. Williams et al. Nature Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Luis A. Williams United States	16	1.6k	978	684	467	397	20	2.4k
Joseph R. Herdy United States	11	1.0k 0.7×	434 0.4×	257 0.4×	312 0.7×	108 0.3×	16	1.5k
Christopher Grunseich United States	24	2.0k 1.3×	321 0.3×	430 0.6×	639 1.4×	76 0.2×	52	2.7k
Christopher J. Donnelly United States	23	2.3k 1.4×	1.4k 1.4×	942 1.4×	803 1.7×	44 0.1×	35	3.4k
Jennifer C. Moore United States	28	1.7k 1.1×	449 0.5×	326 0.5×	443 0.9×	39 0.1×	46	2.4k
Daniela C. Zarnescu United States	28	1.9k 1.2×	692 0.7×	413 0.6×	410 0.9×	136 0.3×	46	2.7k
Xiufang Guo United States	19	1.0k 0.7×	249 0.3×	173 0.3×	558 1.2×	71 0.2×	41	1.5k
Timothy LaVaute United States	17	1.6k 1.0×	130 0.1×	441 0.6×	466 1.0×	83 0.2×	17	2.8k
Betsy A. Hosler United States	20	954 0.6×	1.2k 1.2×	736 1.1×	260 0.6×	35 0.1×	23	2.2k
Ilmin Kwon South Korea	16	1.3k 0.8×	485 0.5×	306 0.4×	255 0.5×	142 0.4×	25	2.0k
Anselme L. Perrier France	24	2.7k 1.7×	344 0.4×	269 0.4×	1.4k 3.1×	68 0.2×	48	3.4k

Countries citing papers authored by Luis A. Williams

Since Specialization

Citations

This map shows the geographic impact of Luis A. Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luis A. Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luis A. Williams more than expected).

Fields of papers citing papers by Luis A. Williams

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Luis A. Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luis A. Williams. The network helps show where Luis A. Williams may publish in the future.

Co-authorship network of co-authors of Luis A. Williams

This figure shows the co-authorship network connecting the top 25 collaborators of Luis A. Williams. A scholar is included among the top collaborators of Luis A. Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Luis A. Williams. Luis A. Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Zhang, Hongkang, Christopher A. Werley, Steven J. Ryan, et al.. (2023). A phenotypic screening platform for chronic pain therapeutics using all-optical electrophysiology. Pain. 165(4). 922–940. 3 indexed citations

Schifferer, Martina, Luis A. Williams, Alexandra Stolz, et al.. (2023). Spatial proteomics reveals secretory pathway disturbances caused by neuropathy-associated TECPR2. Nature Communications. 14(1). 870–870. 8 indexed citations

Simkin, Dina, Bernabé I. Bustos, Christina M. Ambrosi, et al.. (2022). Homozygous might be hemizygous: CRISPR/Cas9 editing in iPSCs results in detrimental on-target defects that escape standard quality controls. Stem Cell Reports. 17(4). 993–1008. 36 indexed citations

Simkin, Dina, Christina M. Ambrosi, Luis A. Williams, et al.. (2022). ‘Channeling’ therapeutic discovery for epileptic encephalopathy through iPSC technologies. Trends in Pharmacological Sciences. 43(5). 392–405. 14 indexed citations

Williams, Luis A., Michael P. Murphy, Christopher A. Werley, et al.. (2019). Scalable Measurements of Intrinsic Excitability in Human iPS Cell-Derived Excitatory Neurons Using All-Optical Electrophysiology. Neurochemical Research. 44(3). 714–725. 11 indexed citations

Klim, Joseph R., Luis A. Williams, Francesco Limone, et al.. (2019). ALS-implicated protein TDP-43 sustains levels of STMN2, a mediator of motor neuron growth and repair. Nature Neuroscience. 22(2). 167–179. 345 indexed citations breakdown →

Vogt, Miriam A., Philip Knuckles, Adrian Higginbottom, et al.. (2018). TDP-43 induces p53-mediated cell death of cortical progenitors and immature neurons. Scientific Reports. 8(1). 8097–8097. 40 indexed citations

Thams, Sebastian, Emily R. Lowry, Krista J. Spiller, et al.. (2018). A Stem Cell-Based Screening Platform Identifies Compounds that Desensitize Motor Neurons to Endoplasmic Reticulum Stress. Molecular Therapy. 27(1). 87–101. 37 indexed citations

Werley, Christopher A., et al.. (2017). All‐Optical Electrophysiology for Disease Modeling and Pharmacological Characterization of Neurons. Current Protocols in Pharmacology. 78(1). 11.20.1–11.20.24. 22 indexed citations

10.

Tripathi, Pratibha, Natalia Rodríguez‐Muela, Joseph R. Klim, et al.. (2017). Reactive Astrocytes Promote ALS-like Degeneration and Intracellular Protein Aggregation in Human Motor Neurons by Disrupting Autophagy through TGF-β1. Stem Cell Reports. 9(2). 667–680. 99 indexed citations

11.

Koppes, Abigail N., et al.. (2014). Neurite outgrowth on electrospun PLLA fibers is enhanced by exogenous electrical stimulation. Journal of Neural Engineering. 11(4). 46002–46002. 54 indexed citations

12.

Wainger, Brian J., Evangelos Kiskinis, Ole Wiskow, et al.. (2014). Intrinsic Membrane Hyperexcitability of Amyotrophic Lateral Sclerosis Patient-Derived Motor Neurons. Cell Reports. 7(1). 1–11. 481 indexed citations breakdown →

13.

Ichida, Justin K., Julia TCW, Luis A. Williams, et al.. (2014). Notch inhibition allows oncogene-independent generation of iPS cells. Nature Chemical Biology. 10(8). 632–639. 54 indexed citations

14.

Davis‐Dusenbery, Brandi N., Luis A. Williams, Joseph R. Klim, & Kevin Eggan. (2014). How to make spinal motor neurons. Development. 141(3). 491–501. 111 indexed citations

15.

Alami, Nael H., Rebecca B. Smith, Mónica A. Carrasco, et al.. (2014). Axonal Transport of TDP-43 mRNA Granules Is Impaired by ALS-Causing Mutations. Neuron. 81(3). 536–543. 497 indexed citations breakdown →

16.

Williams, Luis A., Brandi N. Davis‐Dusenbery, & Kevin Eggan. (2012). SnapShot: Directed Differentiation of Pluripotent Stem Cells. Cell. 149(5). 1174–1174.e1. 54 indexed citations

17.

Simon, Marissa K., et al.. (2012). Positive- and negative-acting regulatory elements contribute to the tissue-specific expression of INNER NO OUTER, a YABBY-type transcription factor gene in Arabidopsis. BMC Plant Biology. 12(1). 214–214. 22 indexed citations

18.

Han, Steve S.W., Luis A. Williams, & Kevin Eggan. (2011). Constructing and Deconstructing Stem Cell Models of Neurological Disease. Neuron. 70(4). 626–644. 116 indexed citations

19.

Stone, Sophia L., Luis A. Williams, Lisa M. Farmer, Richard D. Vierstra, & Judy Callis. (2006). KEEP ON GOING, a RING E3 Ligase Essential for Arabidopsis Growth and Development, Is Involved in Abscisic Acid Signaling. The Plant Cell. 18(12). 3415–3428. 332 indexed citations

20.

Meister, Robert J., Luis A. Williams, Mona M. Monfared, et al.. (2004). Definition and interactions of a positive regulatory element of the Arabidopsis INNER NO OUTER promoter. The Plant Journal. 37(3). 426–438. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact