Luca Posa
- Cellular and Molecular Neuroscience top 5%
- Clinical Psychology top 5%
- Pharmacology top 5%
- Physiology
- Organic Chemistry
- Co-authors
- Gabriella GobbiDanilo De GregorioStefano ComaiJustine P. EnnsRafael Ochoa‐SanchezMartha López-CanulRyan J. McLaughlinSabatino Maione
- Topics
- Pain Mechanisms and Treatments (9 papers)Circadian rhythm and melatonin (8 papers)Neurotransmitter Receptor Influence on Behavior (7 papers)
- Partner nations
- CanadaItalyUnited States
In The Last Decade
Luca Posa
22 papers receiving 822 citations
Hit Papers
Peers
Comparison fields: 5 of 82
- Cellular and Molecular Neuroscience 401
- Clinical Psychology 290
- Pharmacology 281
- Physiology 156
- Organic Chemistry 132
Countries citing papers authored by Luca Posa
This map shows the geographic impact of Luca Posa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luca Posa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luca Posa more than expected).
Fields of papers citing papers by Luca Posa
This network shows the impact of papers produced by Luca Posa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luca Posa. The network helps show where Luca Posa may publish in the future.
Co-authorship network of co-authors of Luca Posa
This figure shows the co-authorship network connecting the top 25 collaborators of Luca Posa. A scholar is included among the top collaborators of Luca Posa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Luca Posa. Luca Posa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 4 | |
| 3 | 4 | |
| 4 | 3 | |
| 5 | 10 | |
| 6 | 8 | |
| 7 | 89 | |
| 8 | 5 | |
| 9 | 26 | |
| 10 | 21 | |
| 11 | 21 | |
| 12 | 22 | |
| 13 | 2 | |
| 14 | 68 | |
| 15 | Cannabidiol modulates serotonergic transmission and reverses both allodynia and anxiety-like behavior in a model of neuropathic painbreakdown → | 276 |
| 16 | 4 | |
| 17 | 0 | |
| 18 | 81 | |
| 19 | 88 | |
| 20 | 19 |
About Luca Posa
Luca Posa is a scholar working on Endocrine and Autonomic Systems, Cellular and Molecular Neuroscience and Physiology, having authored 23 papers that have together received 834 indexed citations. Recurring topics across this work include Pain Mechanisms and Treatments (9 papers), Circadian rhythm and melatonin (8 papers) and Neurotransmitter Receptor Influence on Behavior (7 papers). The work is most often cited by research in Biological Psychiatry (78 citations), Cellular and Molecular Neuroscience (401 citations) and Endocrine and Autonomic Systems (125 citations). Luca Posa has collaborated with scholars based in Canada, Italy and United States. Frequent co-authors include Gabriella Gobbi, Danilo De Gregorio, Stefano Comai, Justine P. Enns, Rafael Ochoa‐Sanchez, Martha López-Canul, Ryan J. McLaughlin, Sabatino Maione, Nicolás A. Núñez and Annalida Bedini. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Pain.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.