Lisha Xiang

3.4k total citations · 4 hit papers
40 papers, 2.1k citations indexed

About

Lisha Xiang is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Lisha Xiang has authored 40 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 16 papers in Cancer Research and 14 papers in Oncology. Recurrent topics in Lisha Xiang's work include Cancer, Hypoxia, and Metabolism (12 papers), Cancer Cells and Metastasis (5 papers) and Cancer Research and Treatments (4 papers). Lisha Xiang is often cited by papers focused on Cancer, Hypoxia, and Metabolism (12 papers), Cancer Cells and Metastasis (5 papers) and Cancer Research and Treatments (4 papers). Lisha Xiang collaborates with scholars based in China, United States and United Kingdom. Lisha Xiang's co-authors include Gregg L. Semenza, Daniele M. Gilkes, Naoharu Takano, Weibo Luo, John W. Bullen, Debangshu Samanta, Houjie Liang, Haiquan Lu, Hongxia Hu and Pallavi Chaturvedi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Scientific Reports.

In The Last Decade

Lisha Xiang

37 papers receiving 2.1k citations

Hit Papers

Hypoxia-inducible factors and RAB22A mediate formation of... 2014 2026 2018 2022 2014 2015 2015 2019 100 200 300 400

Peers

Lisha Xiang
Debangshu Samanta United States
Nicolas Skuli United States
Pallavi Chaturvedi United States
Haiquan Lu United States
Anaïs Oudin Luxembourg
Lindsey K. Boroughs United States
Lisha Xiang
Citations per year, relative to Lisha Xiang Lisha Xiang (= 1×) peers Naoharu Takano

Countries citing papers authored by Lisha Xiang

Since Specialization
Citations

This map shows the geographic impact of Lisha Xiang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lisha Xiang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lisha Xiang more than expected).

Fields of papers citing papers by Lisha Xiang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lisha Xiang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lisha Xiang. The network helps show where Lisha Xiang may publish in the future.

Co-authorship network of co-authors of Lisha Xiang

This figure shows the co-authorship network connecting the top 25 collaborators of Lisha Xiang. A scholar is included among the top collaborators of Lisha Xiang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lisha Xiang. Lisha Xiang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xiang, Lisha, Yang Yang, Lulu Zhang, et al.. (2025). A20 attenuates oxidized self-DNA-mediated inflammation in acute kidney injury. Signal Transduction and Targeted Therapy. 10(1). 154–154. 3 indexed citations
2.
He, Tingting, et al.. (2025). Influence factor and mechanism of FLASH effect. Frontiers in Oncology. 15. 1669228–1669228.
3.
Yu, Yuewen, Lisha Xiang, Xuanwei Zhang, et al.. (2023). Pure Organic AIE Nanoscintillator for X‐ray Mediated Type I and Type II Photodynamic Therapy. Advanced Science. 10(26). e2302395–e2302395. 49 indexed citations
4.
Yu, Yuewen, Lisha Xiang, Xuanwei Zhang, et al.. (2023). Pure Organic AIE Nanoscintillator for X‐ray Mediated Type I and Type II Photodynamic Therapy (Adv. Sci. 26/2023). Advanced Science. 10(26).
5.
Chen, Miao, Ruiyang Ren, Weimin Lin, et al.. (2021). Exploring the oncostatin M (OSM) feed-forward signaling of glioblastoma via STAT3 in pan-cancer analysis. Cancer Cell International. 21(1). 565–565. 21 indexed citations
6.
Xiang, Lisha & Gregg L. Semenza. (2018). Hypoxia-inducible factors promote breast cancer stem cell specification and maintenance in response to hypoxia or cytotoxic chemotherapy. Advances in cancer research. 141. 175–212. 56 indexed citations
7.
Ju, Julia A., Inês Godet, Hasini Jayatilaka, et al.. (2017). Hypoxia Selectively Enhances Integrin α5β1 Receptor Expression in Breast Cancer to Promote Metastasis. Molecular Cancer Research. 15(6). 723–734. 101 indexed citations
8.
Geng, Peiliang, Jianjun Li, Hongtao Li, et al.. (2016). Genetic polymorphisms in C-reactive protein increase cancer susceptibility. Scientific Reports. 6(1). 17161–17161. 6 indexed citations
9.
Geng, Peiliang, Jianjun Li, Ning Wang, et al.. (2015). Genetic Contribution of Polymorphisms in Glutathione S-Transferases to Brain Tumor Risk. Molecular Neurobiology. 53(3). 1730–1740. 11 indexed citations
10.
Geng, Peiliang, Juanjuan Ou, Ganfeng Xie, et al.. (2015). Mitogen-Activated Protein Kinase Kinase 4 Gene Polymorphism and Cancer Risk. Medicine. 94(44). e0938–e0938. 1 indexed citations
11.
Geng, Peiliang, Juanjuan Ou, Jian‐Jun Li, et al.. (2015). A Comprehensive Analysis of Influence ERCC Polymorphisms Confer on the Development of Brain Tumors. Molecular Neurobiology. 53(4). 2705–2714. 16 indexed citations
12.
Geng, Peiliang, Jianjun Li, Ning Wang, et al.. (2015). Genetic association between TRAIL-R1 Thr209Arg and cancer susceptibility. Scientific Reports. 5(1). 10382–10382. 5 indexed citations
13.
Geng, Peiliang, Juanjuan Ou, Jianjun Li, et al.. (2015). Genetic Association Between NFKBIA -881A>G Polymorphism and Cancer Susceptibility. Medicine. 94(31). e1024–e1024. 4 indexed citations
14.
Takano, Naoharu, Yasmeen Sarfraz, Daniele M. Gilkes, et al.. (2014). Decreased Expression of Cystathionine β-Synthase Promotes Glioma Tumorigenesis. Molecular Cancer Research. 12(10). 1398–1406. 53 indexed citations
15.
Geng, Peiliang, Lisha Xiang, Ning Wang, et al.. (2014). Distinct Role of CD86 Polymorphisms (rs1129055, rs17281995) in Risk of Cancer: Evidence from a Meta-Analysis. PLoS ONE. 9(11). e109131–e109131. 6 indexed citations
16.
Wang, Ting, Daniele M. Gilkes, Naoharu Takano, et al.. (2014). Hypoxia-inducible factors and RAB22A mediate formation of microvesicles that stimulate breast cancer invasion and metastasis. Proceedings of the National Academy of Sciences. 111(31). E3234–42. 412 indexed citations breakdown →
17.
Hu, Hongxia, Naoharu Takano, Lisha Xiang, et al.. (2014). Hypoxia-inducible factors enhance glutamate signaling in cancer cells. Oncotarget. 5(19). 8853–8868. 58 indexed citations
18.
Geng, Peiliang, Jianjun Li, Juanjuan Ou, et al.. (2014). Association of Fas -1377 G/A Polymorphism with Susceptibility to Cancer. PLoS ONE. 9(2). e88748–e88748. 3 indexed citations
19.
Xiang, Lisha, Daniele M. Gilkes, Pallavi Chaturvedi, et al.. (2013). Ganetespib blocks HIF-1 activity and inhibits tumor growth, vascularization, stem cell maintenance, invasion, and metastasis in orthotopic mouse models of triple-negative breast cancer. Journal of Molecular Medicine. 92(2). 151–164. 91 indexed citations
20.
Xiang, Lisha, Ganfeng Xie, Chen Liu, et al.. (2013). Knock-down of glutaminase 2 expression decreases glutathione, NADH, and sensitizes cervical cancer to ionizing radiation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1833(12). 2996–3005. 87 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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