Lisa Stadmeyer

1.3k total citations · 1 hit paper
14 papers, 1.1k citations indexed

About

Lisa Stadmeyer is a scholar working on Molecular Biology, Rheumatology and Nephrology. According to data from OpenAlex, Lisa Stadmeyer has authored 14 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 4 papers in Rheumatology and 1 paper in Nephrology. Recurrent topics in Lisa Stadmeyer's work include TGF-β signaling in diseases (7 papers), Bone Metabolism and Diseases (5 papers) and RNA Research and Splicing (3 papers). Lisa Stadmeyer is often cited by papers focused on TGF-β signaling in diseases (7 papers), Bone Metabolism and Diseases (5 papers) and RNA Research and Splicing (3 papers). Lisa Stadmeyer collaborates with scholars based in United States. Lisa Stadmeyer's co-authors include Ernesto Canalis, Vaida Glatt, Mary Bouxsein, Anna Smerdel‐Ramoya, Stefano Zanotti, Deena Durant, Freddy Radtke, Sheila Rydziel, Renata C. Pereira and Aris N. Economides and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Development.

In The Last Decade

Lisa Stadmeyer

14 papers receiving 1.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Age-Related Changes in Trabecular Architecture Differ in Female and Male C57BL/6J Mice

2007 501 citations Vaida Glatt, Ernesto Canalis et al. Journal of Bone and Mineral Research profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Lisa Stadmeyer United States	12	737	240	196	136	129	14	1.1k
Tujun Weng China	16	899 1.2×	266 1.1×	269 1.4×	194 1.4×	116 0.9×	30	1.3k
Masahiro Kanematsu Japan	11	733 1.0×	225 0.9×	364 1.9×	91 0.7×	92 0.7×	14	1.0k
Hiroshi Tsurukami Japan	17	671 0.9×	439 1.8×	387 2.0×	201 1.5×	85 0.7×	29	1.2k
Mark L. Johnson United States	11	673 0.9×	223 0.9×	164 0.8×	166 1.2×	84 0.7×	16	938
David E. Maridas United States	13	558 0.8×	157 0.7×	177 0.9×	79 0.6×	190 1.5×	24	974
Narelle E. McGregor Australia	19	816 1.1×	189 0.8×	584 3.0×	142 1.0×	98 0.8×	36	1.2k
Eveline Boudin Belgium	16	584 0.8×	229 1.0×	233 1.2×	310 2.3×	41 0.3×	43	920
Elizabeth C. Wahl United States	15	407 0.6×	278 1.2×	194 1.0×	147 1.1×	70 0.5×	22	763
Thomas H. Ambrosi United States	10	408 0.6×	135 0.6×	170 0.9×	58 0.4×	246 1.9×	22	1.0k
M.-C. Faugere United States	11	533 0.7×	310 1.3×	344 1.8×	155 1.1×	56 0.4×	20	1.1k

Countries citing papers authored by Lisa Stadmeyer

Since Specialization

Citations

This map shows the geographic impact of Lisa Stadmeyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lisa Stadmeyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lisa Stadmeyer more than expected).

Fields of papers citing papers by Lisa Stadmeyer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lisa Stadmeyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lisa Stadmeyer. The network helps show where Lisa Stadmeyer may publish in the future.

Co-authorship network of co-authors of Lisa Stadmeyer

This figure shows the co-authorship network connecting the top 25 collaborators of Lisa Stadmeyer. A scholar is included among the top collaborators of Lisa Stadmeyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lisa Stadmeyer. Lisa Stadmeyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Zanotti, Stefano, Lisa Stadmeyer, Anna Smerdel‐Ramoya, Deena Durant, & Ernesto Canalis. (2009). Misexpression of CCAAT/enhancer binding protein beta causes osteopenia. Journal of Endocrinology. 201(2). 263–274. 37 indexed citations

Pearsall, R. Scott, Ernesto Canalis, Milton Cornwall-Brady, et al.. (2008). A soluble activin Type IIA receptor induces bone formation and improves skeletal integrity. Proceedings of the National Academy of Sciences. 105(19). 7082–7087. 7 indexed citations

Zanotti, Stefano, Anna Smerdel‐Ramoya, Lisa Stadmeyer, & Ernesto Canalis. (2008). Activation of the ERK pathway in osteoblastic cells, role of gremlin and BMP‐2. Journal of Cellular Biochemistry. 104(4). 1421–1426. 23 indexed citations

Nagatomo, Kanako J., Kevin A. Tompkins, Hanson Fong, et al.. (2008). Transgenic Overexpression of Gremlin Results in Developmental Defects in Enamel and Dentin in Mice. Connective Tissue Research. 49(6). 391–400. 11 indexed citations

Smerdel‐Ramoya, Anna, Stefano Zanotti, Lisa Stadmeyer, Deena Durant, & Ernesto Canalis. (2008). Skeletal Overexpression of Connective Tissue Growth Factor Impairs Bone Formation and Causes Osteopenia. Endocrinology. 149(9). 4374–4381. 41 indexed citations

Raz, Regina, Sigmar Stricker, Florian Witte, et al.. (2008). The mutationROR2W749X, linked to human BDB, is a recessive mutation in the mouse, causing brachydactyly, mediating patterning of joints and modeling recessive Robinow syndrome. Development. 135(9). 1713–1723. 21 indexed citations

Zanotti, Stefano, Anna Smerdel‐Ramoya, Lisa Stadmeyer, et al.. (2008). Notch Inhibits Osteoblast Differentiation and Causes Osteopenia. Endocrinology. 149(8). 3890–3899. 166 indexed citations

Gazzerro, Elisabetta, Anna Smerdel‐Ramoya, Stefano Zanotti, et al.. (2007). Conditional Deletion of Gremlin Causes a Transient Increase in Bone Formation and Bone Mass. Journal of Biological Chemistry. 282(43). 31549–31557. 90 indexed citations

Rydziel, Sheila, Lisa Stadmeyer, Stefano Zanotti, et al.. (2007). Nephroblastoma Overexpressed (Nov) Inhibits Osteoblastogenesis and Causes Osteopenia. Journal of Biological Chemistry. 282(27). 19762–19772. 60 indexed citations

10.

Glatt, Vaida, Ernesto Canalis, Lisa Stadmeyer, & Mary Bouxsein. (2007). Age-Related Changes in Trabecular Architecture Differ in Female and Male C57BL/6J Mice. Journal of Bone and Mineral Research. 22(8). 1197–1207. 501 indexed citations breakdown →

11.

Gazzerro, Elisabetta, et al.. (2006). Twisted gastrulation, a bone morphogenetic protein agonist/antagonist, is not required for post-natal skeletal function. Bone. 39(6). 1252–1260. 19 indexed citations

12.

Pereira, Renata C., et al.. (2006). CCAAT/Enhancer-binding protein homologous protein (CHOP) decreases bone formation and causes osteopenia. Bone. 40(3). 619–626. 40 indexed citations

13.

Pereira, Renata C., Lisa Stadmeyer, Stefan J. Marciniak, David Ron, & Ernesto Canalis. (2005). C/EBP homologous protein is necessary for normal osteoblastic function. Journal of Cellular Biochemistry. 97(3). 633–640. 36 indexed citations

14.

Durant, Deena, Renata C. Pereira, Lisa Stadmeyer, & Ernesto Canalis. (2004). Transgenic mice expressing selected insulin-like growth factor-binding protein-5 fragments do not exhibit enhanced bone formation. Growth Hormone & IGF Research. 14(4). 319–327. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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