Linyu Wei

619 total citations
34 papers, 432 citations indexed

About

Linyu Wei is a scholar working on Molecular Biology, Physiology and Artificial Intelligence. According to data from OpenAlex, Linyu Wei has authored 34 papers receiving a total of 432 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 8 papers in Physiology and 6 papers in Artificial Intelligence. Recurrent topics in Linyu Wei's work include Alzheimer's disease research and treatments (6 papers), Neuroscience and Neuropharmacology Research (4 papers) and Adenosine and Purinergic Signaling (4 papers). Linyu Wei is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Neuroscience and Neuropharmacology Research (4 papers) and Adenosine and Purinergic Signaling (4 papers). Linyu Wei collaborates with scholars based in China, United Kingdom and France. Linyu Wei's co-authors include Lin‐Hua Jiang, Dongliang Li, Chaokun Li, Sébastien Roger, Jian‐Zhi Wang, Mingsha Zhang, Xinjuan Li, Xuebin Yang, Guohong Wang and Jueping Cai and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and SHILAP Revista de lepidopterología.

In The Last Decade

Linyu Wei

34 papers receiving 430 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Linyu Wei China 13 149 111 72 61 51 34 432
Damien Toulorge France 9 201 1.3× 96 0.9× 88 1.2× 81 1.3× 27 0.5× 9 561
Jillian C. Belrose Canada 10 192 1.3× 138 1.2× 45 0.6× 71 1.2× 18 0.4× 16 658
Filippo Ugolini Italy 13 133 0.9× 100 0.9× 46 0.6× 178 2.9× 12 0.2× 37 530
Mahshad Kolahdouzan Canada 5 109 0.7× 108 1.0× 32 0.4× 101 1.7× 13 0.3× 5 425
Alicia Sedó Australia 12 246 1.7× 77 0.7× 35 0.5× 76 1.2× 19 0.4× 16 609
Chiara Falcicchia Italy 11 279 1.9× 94 0.8× 21 0.3× 106 1.7× 36 0.7× 21 642
Bei Miao China 14 307 2.1× 198 1.8× 31 0.4× 69 1.1× 17 0.3× 30 715
Jaume Lillo Spain 11 134 0.9× 34 0.3× 25 0.3× 45 0.7× 30 0.6× 40 351
Martha C. Rivera‐Cervantes Mexico 16 226 1.5× 68 0.6× 15 0.2× 126 2.1× 46 0.9× 24 602
Stanisław Szlufik Poland 15 181 1.2× 144 1.3× 14 0.2× 134 2.2× 34 0.7× 54 678

Countries citing papers authored by Linyu Wei

Since Specialization
Citations

This map shows the geographic impact of Linyu Wei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Linyu Wei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Linyu Wei more than expected).

Fields of papers citing papers by Linyu Wei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Linyu Wei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Linyu Wei. The network helps show where Linyu Wei may publish in the future.

Co-authorship network of co-authors of Linyu Wei

This figure shows the co-authorship network connecting the top 25 collaborators of Linyu Wei. A scholar is included among the top collaborators of Linyu Wei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Linyu Wei. Linyu Wei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wei, Linyu, Kun Shi, Jiale Liu, et al.. (2025). Improving vulnerable Calbindin1− neurons in the ventral hippocampus rescues tau-induced impairment of episodic memory. Translational Neurodegeneration. 14(1). 12–12. 1 indexed citations
2.
Wei, Linyu, Fei Sun, Mengzhu Li, et al.. (2024). A tau dephosphorylation-targeting chimera selectively recruits protein phosphatase-1 to ameliorate Alzheimer’s disease and tauopathies. Cell chemical biology. 31(10). 1787–1799.e6. 6 indexed citations
3.
Wei, Linyu, Fei Sun, Weixia Wang, et al.. (2024). Generation of tau dephosphorylation-targeting chimeras for the treatment of Alzheimer’s disease and related tauopathies. Science Bulletin. 69(8). 1137–1152. 10 indexed citations
4.
Wei, Linyu, Fei Sun, Weixia Wang, et al.. (2024). A new tau dephosphorylation-targeting chimera for the treatment of tauopathies. Acta Pharmacologica Sinica. 45(11). 2267–2276. 7 indexed citations
5.
Wei, Linyu, et al.. (2023). Local–global lightweight ViT model for mini/micro-LED-chip defect recognition. Engineering Applications of Artificial Intelligence. 123. 106247–106247. 9 indexed citations
6.
Yin, Yaling, Linyu Wei, Emily A. Caseley, et al.. (2023). Leveraging the ATP‐P2X7 receptor signalling axis to alleviate traumatic CNS damage and related complications. Medicinal Research Reviews. 43(5). 1346–1373. 12 indexed citations
7.
Zheng, Jie, Fei Liu, Yang Gao, et al.. (2021). A novel dephosphorylation targeting chimera selectively promoting tau removal in tauopathies. Signal Transduction and Targeted Therapy. 6(1). 269–269. 49 indexed citations
8.
Wei, Linyu, Dongliang Li, Sébastien Roger, et al.. (2019). Adenosine Triphosphate Release and P2 Receptor Signaling in Piezo1 Channel-Dependent Mechanoregulation. Frontiers in Pharmacology. 10. 1304–1304. 50 indexed citations
9.
Ye, Jinwang, Yaling Yin, Huanhuan Liu, et al.. (2019). Tau inhibits PKA by nuclear proteasome‐dependent PKAR2α elevation with suppressed CREB/GluA1 phosphorylation. Aging Cell. 19(1). e13055–e13055. 24 indexed citations
10.
Wei, Linyu, Sharifah Alawieyah Syed Mortadza, Jing Yan, et al.. (2018). ATP-activated P2X7 receptor in the pathophysiology of mood disorders and as an emerging target for the development of novel antidepressant therapeutics. Neuroscience & Biobehavioral Reviews. 87. 192–205. 39 indexed citations
11.
Jiang, Lin‐Hua, Linyu Wei, & Sharifah Alawieyah Syed Mortadza. (2018). Melastatin-related transient receptor potential 2 channel in Aβ42-induced neuroinflammation: implications to Alzheimer’s disease mechanism and development of therapeutics. Neural Regeneration Research. 13(3). 419–419. 3 indexed citations
12.
Wei, Linyu, Emily A. Caseley, Dongliang Li, & Lin‐Hua Jiang. (2016). ATP-induced P2X Receptor-Dependent Large Pore Formation: How Much Do We Know?. Frontiers in Pharmacology. 7. 5–5. 20 indexed citations
13.
Li, Dongliang, Xinjuan Li, Linyu Wei, et al.. (2015). Hydrogen sulfide intervention in focal cerebral ischemia/reperfusion injury in rats. SHILAP Revista de lepidopterología. 10(6). 932–932. 26 indexed citations
14.
15.
Wei, Linyu, et al.. (2014). Motor preparation attenuates neural variability and beta-band LFP in parietal cortex. Scientific Reports. 4(1). 6809–6809. 5 indexed citations
16.
Li, Chaokun, Linyu Wei, Linlin Shan, et al.. (2014). Stable Cell Line of Human SH-SY5Y Uniformly Expressing TWIK-Related Acid-Sensitive Potassium Channel and eGFP Fusion. Applied Biochemistry and Biotechnology. 172(6). 3253–3262. 2 indexed citations
17.
Li, Chaokun, et al.. (2014). Mifepristone modulates serotonin transporter function. Neural Regeneration Research. 9(6). 646–646. 2 indexed citations
18.
Li, Xinjuan, Hua Han, Linyu Wei, et al.. (2013). Progesterone treatment before experimental hypoxia-ischemia enhances the expression of glucose transporter proteins GLUT1 and GLUT3 in neonatal rats. Neuroscience Bulletin. 29(3). 287–294. 19 indexed citations
19.
Wei, Linyu, et al.. (2013). Parietal Cortical Neuronal Activity Is Selective for Express Saccades. Journal of Neuroscience. 33(2). 814–823. 21 indexed citations
20.
Li, Xinjuan, Shuang Li, Xinqiang Li, Linyu Wei, & Dongliang Li. (2012). [Effects of taurine on the ultrastructure and P2X7 receptor expression in brain following traumatic brain injury in rats].. PubMed. 28(4). 301–3, 308. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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