Linna Peng

1.3k total citations
12 papers, 332 citations indexed

About

Linna Peng is a scholar working on Molecular Biology, Surgery and Cancer Research. According to data from OpenAlex, Linna Peng has authored 12 papers receiving a total of 332 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 3 papers in Surgery and 3 papers in Cancer Research. Recurrent topics in Linna Peng's work include Cancer-related gene regulation (3 papers), RNA Research and Splicing (3 papers) and Esophageal Cancer Research and Treatment (3 papers). Linna Peng is often cited by papers focused on Cancer-related gene regulation (3 papers), RNA Research and Splicing (3 papers) and Esophageal Cancer Research and Treatment (3 papers). Linna Peng collaborates with scholars based in China, United States and Laos. Linna Peng's co-authors include Fei Chen, Shibin Hu, Aixia Song, Zhenning Wang, Congling Xu, Yingying Luo, Wen Tan, Dongxin Lin, Chen Wu and Qionghua Cui and has published in prestigious journals such as Nature Communications, Molecular Cell and Gastroenterology.

In The Last Decade

Linna Peng

12 papers receiving 331 citations

Peers

Linna Peng
Shang‐Min Zhang United States
Jiang Xu China
Ke Zheng China
Linna Peng
Citations per year, relative to Linna Peng Linna Peng (= 1×) peers Julien Izotte

Countries citing papers authored by Linna Peng

Since Specialization
Citations

This map shows the geographic impact of Linna Peng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Linna Peng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Linna Peng more than expected).

Fields of papers citing papers by Linna Peng

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Linna Peng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Linna Peng. The network helps show where Linna Peng may publish in the future.

Co-authorship network of co-authors of Linna Peng

This figure shows the co-authorship network connecting the top 25 collaborators of Linna Peng. A scholar is included among the top collaborators of Linna Peng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Linna Peng. Linna Peng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Hu, Shibin, Linna Peng, Aixia Song, et al.. (2023). INTAC endonuclease and phosphatase modules differentially regulate transcription by RNA polymerase II. Molecular Cell. 83(10). 1588–1604.e5. 30 indexed citations
2.
Song, Aixia, Shibin Hu, Linna Peng, et al.. (2022). Coordinated regulation of RNA polymerase II pausing and elongation progression by PAF1. Science Advances. 8(13). eabm5504–eabm5504. 35 indexed citations
3.
Hu, Shibin, Linna Peng, Congling Xu, et al.. (2021). SPT5 stabilizes RNA polymerase II, orchestrates transcription cycles, and maintains the enhancer landscape. Molecular Cell. 81(21). 4425–4439.e6. 63 indexed citations
4.
Chu, Jiahui, Xiangjie Niu, Jiang Chang, et al.. (2020). Metabolic remodeling by TIGAR overexpression is a therapeutic target in esophageal squamous-cell carcinoma. Theranostics. 10(8). 3488–3502. 27 indexed citations
5.
Yao, Jiacheng, Qionghua Cui, Wenyi Fan, et al.. (2020). Single-cell transcriptomic analysis in a mouse model deciphers cell transition states in the multistep development of esophageal cancer. Nature Communications. 11(1). 3715–3715. 88 indexed citations
6.
Zhao, Ting, Linna Peng, Pan Li, et al.. (2019). Novel hyaluronic acid-modified temperature-sensitive nanoparticles for synergistic chemo-photothermal therapy. Carbohydrate Polymers. 214. 221–233. 30 indexed citations
7.
Peng, Linna, Sijin Cheng, Yuan Lin, et al.. (2018). CCGD-ESCC: A Comprehensive Database for Genetic Variants Associated with Esophageal Squamous Cell Carcinoma in Chinese Population. Genomics Proteomics & Bioinformatics. 16(4). 262–268. 14 indexed citations
8.
Li, Jun, Wenle Tan, Linna Peng, et al.. (2018). Integrative analysis of gene expression profiles reveals specific signaling pathways associated with pancreatic duct adenocarcinoma. Cancer Communications. 38(1). 13–13. 7 indexed citations
9.
Cui, Qionghua, Linna Peng, Lixuan Wei, et al.. (2017). Genetic variant repressing ADH1A expression confers susceptibility to esophageal squamous-cell carcinoma. Cancer Letters. 421. 43–50. 11 indexed citations
10.
Wei, Lixuan, Mingming Shao, Yanjie Zhao, et al.. (2017). Functional role of PLCE1 intronic insertion variant associated with susceptibility to esophageal squamous cell carcinoma. Carcinogenesis. 39(2). 191–201. 6 indexed citations
11.
Zhao, Yanjie, Lixuan Wei, Mingming Shao, et al.. (2017). BRCA1-Associated Protein Increases Invasiveness of Esophageal Squamous Cell Carcinoma. Gastroenterology. 153(5). 1304–1319.e5. 19 indexed citations
12.
Wang, Lingling, Yanjie Zhao, Jun Li, et al.. (2016). Data analysis in the post‐genome‐wide association study era. Chronic Diseases and Translational Medicine. 2(4). 231–234. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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