Ling-juan Gao

581 total citations
23 papers, 457 citations indexed

About

Ling-juan Gao is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Ling-juan Gao has authored 23 papers receiving a total of 457 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 8 papers in Immunology and 7 papers in Cancer Research. Recurrent topics in Ling-juan Gao's work include Complement system in diseases (5 papers), Drug Transport and Resistance Mechanisms (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Ling-juan Gao is often cited by papers focused on Complement system in diseases (5 papers), Drug Transport and Resistance Mechanisms (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). Ling-juan Gao collaborates with scholars based in China, United States and Australia. Ling-juan Gao's co-authors include Ping‐qing Gu, Yajuan Su, Tianying Zhong, Huilin Zhang, Zhu Liu, Shuyu Guo, Fengshan Li, Lichen Teng, Lin Xiong and Yuan Mao and has published in prestigious journals such as Scientific Reports, Biochemical and Biophysical Research Communications and Life Sciences.

In The Last Decade

Ling-juan Gao

23 papers receiving 453 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ling-juan Gao China 12 289 121 106 78 55 23 457
Yoko Ino Japan 13 282 1.0× 97 0.8× 44 0.4× 87 1.1× 18 0.3× 37 510
Tara L. Sander United States 12 433 1.5× 88 0.7× 107 1.0× 73 0.9× 22 0.4× 19 598
Ye Gan China 11 190 0.7× 24 0.2× 108 1.0× 39 0.5× 33 0.6× 17 481
Hong‐Tai Tzeng Taiwan 13 181 0.6× 53 0.4× 186 1.8× 119 1.5× 7 0.1× 21 451
Rocío Soldati Argentina 12 187 0.6× 56 0.5× 264 2.5× 183 2.3× 92 1.7× 17 614
Tiefen Su China 8 314 1.1× 87 0.7× 51 0.5× 101 1.3× 45 0.8× 10 480
Susanne Stary Austria 9 232 0.8× 38 0.3× 18 0.2× 68 0.9× 13 0.2× 11 414
Ondrej Pös Slovakia 12 400 1.4× 358 3.0× 42 0.4× 79 1.0× 15 0.3× 20 696
Cédric Carli Canada 14 146 0.5× 27 0.2× 313 3.0× 145 1.9× 82 1.5× 20 547
Sami Purmonen Finland 9 201 0.7× 54 0.4× 17 0.2× 48 0.6× 28 0.5× 14 425

Countries citing papers authored by Ling-juan Gao

Since Specialization
Citations

This map shows the geographic impact of Ling-juan Gao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ling-juan Gao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ling-juan Gao more than expected).

Fields of papers citing papers by Ling-juan Gao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ling-juan Gao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ling-juan Gao. The network helps show where Ling-juan Gao may publish in the future.

Co-authorship network of co-authors of Ling-juan Gao

This figure shows the co-authorship network connecting the top 25 collaborators of Ling-juan Gao. A scholar is included among the top collaborators of Ling-juan Gao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ling-juan Gao. Ling-juan Gao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ma, Xiaoxia, Ling-juan Gao, Yulong Ma, & Xiaoguang Zhang. (2021). Antibacterial Activity and Mechanism of ZnO/Cu2+-Chitosan/Montmorillonite. Journal of Wuhan University of Technology-Mater Sci Ed. 36(4). 510–516. 4 indexed citations
2.
Liang, Jin‐Hua, Li Dong, Shu Li, et al.. (2020). The role of the globular heads of the C1q receptor in TcdA-induced human colonic epithelial cell apoptosis via a mitochondria-dependent pathway. BMC Microbiology. 20(1). 274–274. 4 indexed citations
3.
Liu, Zhu, Lichen Teng, Ling-juan Gao, et al.. (2018). The role of eukaryotic translation initiation factor 5A-1 (eIF5A-1) gene in HPV 16 E6 induces cell growth in human cervical squamous carcinoma cells. Biochemical and Biophysical Research Communications. 504(1). 6–12. 11 indexed citations
4.
Cheng, Feng, Xianwei Cui, Ling-juan Gao, et al.. (2018). Circular RNA expression profiles in placental villi from women with gestational diabetes mellitus. Biochemical and Biophysical Research Communications. 498(4). 743–750. 66 indexed citations
5.
Fang, Teng, Hongjie Ruan, Juan Xu, et al.. (2018). RBBP6 promotes human cervical carcinoma malignancy via JNK signaling pathway. Biomedicine & Pharmacotherapy. 101. 399–405. 13 indexed citations
7.
Liu, Zhu, et al.. (2017). MiR-22-3p Regulates Cell Proliferation and Inhibits Cell Apoptosis through Targeting the eIF4EBP3 Gene in Human Cervical Squamous Carcinoma Cells. International Journal of Medical Sciences. 15(2). 142–152. 44 indexed citations
9.
Liu, Zhu, Hongjie Ruan, Ping‐qing Gu, et al.. (2015). The Roles of p38 MAPK and ERK1/2 in Coplanar Polychlorinated Biphenyls-Induced Apoptosis of Human Extravillous Cytotrophoblast-Derived Transformed Cells. Cellular Physiology and Biochemistry. 36(6). 2418–2432. 14 indexed citations
10.
Zhang, Huilin, Jinrong Qiu, Ling-juan Gao, et al.. (2014). ROR1 expression correlated with poor clinical outcome in human ovarian cancer. Scientific Reports. 4(1). 5811–5811. 93 indexed citations
11.
12.
Su, Yajuan, Ling-juan Gao, Lichen Teng, et al.. (2013). Id1 enhances human ovarian cancer endothelial progenitor cell angiogenesis via PI3K/Akt and NF-κB/MMP-2 signaling pathways. Journal of Translational Medicine. 11(1). 132–132. 44 indexed citations
13.
Gao, Ling-juan, Ping‐qing Gu, Wei Zhao, et al.. (2013). The role of globular heads of the C1q receptor in HPV 16 E2-induced human cervical squamous carcinoma cell apoptosis is associated with p38 MAPK/JNK activation. Journal of Translational Medicine. 11(1). 118–118. 22 indexed citations
14.
Liu, Zhu, et al.. (2012). The Role of the Globular Heads of C1q Receptor (gC1qR) Gene in Regulating Apoptosis of Human Cervical Squamous Cell Carcinoma. Cellular Physiology and Biochemistry. 30(5). 1181–1190. 13 indexed citations
15.
Gu, Ping‐qing, et al.. (2012). The globular heads of the C1q receptor regulate apoptosis in human cervical squamous carcinoma cells via a p53-dependent pathway. Journal of Translational Medicine. 10(1). 255–255. 7 indexed citations
16.
Gu, Ping‐qing, Ling-juan Gao, Li Li, et al.. (2011). Endocrine Disruptors, Polychlorinated Biphenyls-Induced gC1qR-dependent Apoptosis in Human Trophoblast Cell Line HTR-8/SVneo. Reproductive Sciences. 19(2). 181–189. 10 indexed citations
17.
Gao, Ling-juan, Lan Ding, Shuyu Guo, et al.. (2010). Role of decay-accelerating factor in regulating survival of human cervical cancer cells. Journal of Cancer Research and Clinical Oncology. 137(1). 81–87. 10 indexed citations
18.
Gao, Ling-juan, Shuyu Guo, Ping‐qing Gu, et al.. (2009). Cooperation of decay-accelerating factor and membrane cofactor protein in regulating survival of human cervical cancer cells. BMC Cancer. 9(1). 384–384. 15 indexed citations
20.
Qiu, Wen, et al.. (2006). Gene expression profile and overexpression of apoptosis-related genes (NGFI-B and Gadd 45 γ) in early phase of Thy-1 nephritis model. Cell and Tissue Research. 326(1). 159–168. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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