Ling Xue

950 total citations
21 papers, 620 citations indexed

About

Ling Xue is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Ling Xue has authored 21 papers receiving a total of 620 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 6 papers in Cancer Research and 3 papers in Genetics. Recurrent topics in Ling Xue's work include RNA modifications and cancer (15 papers), Mitochondrial Function and Pathology (14 papers) and Cancer-related molecular mechanisms research (6 papers). Ling Xue is often cited by papers focused on RNA modifications and cancer (15 papers), Mitochondrial Function and Pathology (14 papers) and Cancer-related molecular mechanisms research (6 papers). Ling Xue collaborates with scholars based in China, United States and Uzbekistan. Ling Xue's co-authors include Min‐Xin Guan, Li Yang, Ronghua Li, Taosheng Huang, Zongqing Tan, Daren Wang, Pingping Jiang, Jun Qin Mo, Meng Wang and Haiying Li and has published in prestigious journals such as Journal of Biological Chemistry, Molecular and Cellular Biology and Scientific Reports.

In The Last Decade

Ling Xue

19 papers receiving 604 citations

Peers

Ling Xue

PHYSI

ECOLO

Xiaoyin Ma China

Akitsugu Sato Japan

Suraiya Haroon United States

Alexander J. Anderson Australia

Yong Qian China

R.N. Lightowlers United Kingdom

Per Hove Andreasen Denmark

Gilad Barshad United States

М. В. Патрушев Russia

Yanhua Qi China

Xiaoyin Ma China

Ling Xue

382 ×0.8

26 ×0.3

41 ×0.5

PHYSI

48 ×0.7

48 ×0.9

ECOLO

Citations per year, relative to Ling Xue Ling Xue (= 1×) peers Xiaoyin Ma

Countries citing papers authored by Ling Xue

Since Specialization

Citations

This map shows the geographic impact of Ling Xue's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ling Xue with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ling Xue more than expected).

Fields of papers citing papers by Ling Xue

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ling Xue. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ling Xue. The network helps show where Ling Xue may publish in the future.

Co-authorship network of co-authors of Ling Xue

This figure shows the co-authorship network connecting the top 25 collaborators of Ling Xue. A scholar is included among the top collaborators of Ling Xue based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ling Xue. Ling Xue is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wang, Caihui, et al.. (2025). The chloroplast genome elucidates the origin of mulberry in Central Asia. Frontiers in Plant Science. 16. 1592308–1592308.

Xue, Ling, Ya-Ru Chen, Xiaowen Tang, et al.. (2018). A deafness-associated mitochondrial DNA mutation altered the tRNASer(UCN) metabolism and mitochondrial function. Mitochondrion. 46. 370–379. 10 indexed citations

Zhou, Mi, Ling Xue, Yaru Chen, et al.. (2017). A hypertension-associated mitochondrial DNA mutation introduces an m1G37 modification into tRNAMet, altering its structure and function. Journal of Biological Chemistry. 293(4). 1425–1438. 58 indexed citations

Zhou, Mi, Meng Wang, Ling Xue, et al.. (2017). A hypertension-associated mitochondrial DNA mutation alters the tertiary interaction and function of tRNALeu(UUR). Journal of Biological Chemistry. 292(34). 13934–13946. 23 indexed citations

Li, Haiying, et al.. (2017). Mitochondrial tRNAThr 15909A>G mutation associated with hypertension in a Chinese Han pedigree. Biochemical and Biophysical Research Communications. 495(1). 574–581. 11 indexed citations

Wang, Hui, Heng Wang, Feng Jiang, et al.. (2016). The Study of Mitochondrial Function in A Nonsyndromic Hearing Loss Family Carrying Mitochondria tRNA(Thr) 15910C>T and 12S rRNA 1555A>G Mutations. 38(10). 1231. 1 indexed citations

Xue, Ling, Meng Wang, Haiying Li, et al.. (2016). Mitochondrial tRNA mutations in 2070 Chinese Han subjects with hypertension. Mitochondrion. 30. 208–221. 28 indexed citations

Jiang, Pingping, Meng Wang, Ling Xue, et al.. (2016). A Hypertension-Associated tRNA^Ala Mutation Alters tRNA Metabolism and Mitochondrial Function. Molecular and Cellular Biology. 36(14). 1920–1930. 49 indexed citations

Xu, Meifen, et al.. (2014). The mitochondrial tRNAMet/tRNAGlnA4401G and tRNACysG5821A mutations may be associated with hypertension in two Han Chinese families. Hereditas (Beijing). 36(2). 127–134. 1 indexed citations

10.

Xu, Yongping, Kui Xu, Ying Sun, et al.. (2014). Multiple pathogenic proteins implicated in neuronopathic Gaucher disease mice. Human Molecular Genetics. 23(15). 3943–3957. 74 indexed citations

11.

Yang, Li, Zongqing Tan, Daren Wang, et al.. (2014). Species identification through mitochondrial rRNA genetic analysis. Scientific Reports. 4(1). 4089–4089. 146 indexed citations

12.

Qin, Yanwen, Ling Xue, Pingping Jiang, et al.. (2014). Mitochondrial tRNA Variants in Chinese Subjects With Coronary Heart Disease. Journal of the American Heart Association. 3(1). e000437–e000437. 31 indexed citations

13.

Xue, Ling, Yuan Yang, & S. Wang. (2014). A novel large deletion of the DOCK8 gene in a Chinese family with autosomal‐recessive hyper‐IgE syndrome. Journal of the European Academy of Dermatology and Venereology. 29(3). 599–601. 7 indexed citations

14.

Zhang, Juanjuan, Fuxin Zhao, Qun Fu, et al.. (2013). Mitochondrial haplotypes may modulate the phenotypic manifestation of the LHON-associated m.14484T>C (MT-ND6) mutation in Chinese families. Mitochondrion. 13(6). 772–781. 21 indexed citations

15.

Wang, Xinjian, Yanwen Qin, Ling Xue, et al.. (2013). Coronary heart disease is associated with a mutation in mitochondrial tRNA. Human Molecular Genetics. 22(20). 4064–4073. 50 indexed citations

16.

Chen, Hong, Jing Zheng, Ling Xue, et al.. (2012). The 12S rRNA A1555G mutation in the mitochondrial haplogroup D5a is responsible for maternally inherited hypertension and hearing loss in two Chinese pedigrees. European Journal of Human Genetics. 20(6). 607–612. 15 indexed citations

17.

Qiu, Qiao-meng, Ronghua Li, Pingping Jiang, et al.. (2012). Mitochondrial tRNA mutations are associated with maternally inherited hypertension in two Han Chinese pedigrees. Human Mutation. 33(8). 1285–1293. 21 indexed citations

18.

Xue, Ling, Hong Chen, Yan Wang, et al.. (2011). Mutations in mitochondrial DNA associated with hypertension. Hereditas (Beijing). 33(9). 911–918. 3 indexed citations

19.

Lu, Zhongqiu, Hong Chen, Yan Wang, et al.. (2011). The tRNAMet 4435A>G mutation in the mitochondrial haplogroup G2a1 is responsible for maternally inherited hypertension in a Chinese pedigree. European Journal of Human Genetics. 19(11). 1181–1186. 34 indexed citations

20.

Tang, Xiaowen, Ronghua Li, Jing Zheng, et al.. (2010). Maternally inherited hearing loss is associated with the novel mitochondrial tRNASer(UCN) 7505T>C mutation in a Han Chinese family. Molecular Genetics and Metabolism. 100(1). 57–64. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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