Leslie A. Goldstein

3.0k citations
26 papers · 2.6k indexed · h-index 23

Leslie A. Goldstein

26 papers receiving 2.5k citations

Peers

Leslie A. Goldstein
Comparison fields: 5 of 102
  • Immunology and Allergy 429
  • Cell Biology 539
  • Cancer Research 457
  • Oncology 688
  • Immunology 502
Replace Chloé C. Féral with:
Chloé C. Féral France
Alexander Y. Tsygankov United States
Parmesh Dutt United States
Mario Gonzalez–Gronow United States
Michael L. Lu United States
K R Auger United States
John T. Price Australia
Corinne Wendling France
Jenny Jongstra‐Bilen Canada
Francis J. Eng United States
Leslie A. Goldstein relative to Chloé C. Féral France Chloé C. Féral's profile →
Citations per field
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Chloé C. Féral · 1×
Citations per year

Countries citing papers authored by Leslie A. Goldstein

Since Specialization
Citations

This map shows the geographic impact of Leslie A. Goldstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leslie A. Goldstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leslie A. Goldstein more than expected).

Fields of papers citing papers by Leslie A. Goldstein

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leslie A. Goldstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leslie A. Goldstein. The network helps show where Leslie A. Goldstein may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Leslie A. Goldstein, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Leslie A. Goldstein Line = papers co-authored together Leslie A. Goldstein links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 201828
2 201385
3 201343
4 2010301
5 201035
6 201052
7 2008152
8 200828
9 2006161
10 200562
11 200488
12 2002145
13
Granzyme B-mediated degradation of T-cell receptor zeta chain.
200239
14 2001110
15 200129
16 200027
17 1997130
18 1997124
19 199446
20 1989273

About Leslie A. Goldstein

Leslie A. Goldstein is a scholar working on Immunology and Allergy, Cancer Research and Epidemiology, having authored 26 papers that have together received 2.6k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (15 papers), Autophagy in Disease and Therapy (8 papers), Protease and Inhibitor Mechanisms (6 papers), Mitochondrial Function and Pathology (4 papers), RNA Interference and Gene Delivery (4 papers), Peptidase Inhibition and Analysis (4 papers), Hepatitis B Virus Studies (3 papers) and Cell Adhesion Molecules Research (3 papers). The work is most often cited by research in Immunology and Allergy (429 citations), Cell Biology (539 citations) and Cancer Research (457 citations). Leslie A. Goldstein has collaborated with scholars based in United States, Italy and Ireland. Frequent co-authors include Hannah Rabinowich, Wen‐Chi Hou, Jie Han, Brian Gastman, Eugene C. Butcher, Louis J. Picker, David Zhou, Jie Han, Caisheng Lu and Wen‐Tien Chen. Their work appears in journals such as Cell, Journal of Biological Chemistry and The Journal of Experimental Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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