Leonóra Himer

695 total citations
8 papers, 557 citations indexed

About

Leonóra Himer is a scholar working on Immunology, Physiology and Neurology. According to data from OpenAlex, Leonóra Himer has authored 8 papers receiving a total of 557 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology, 5 papers in Physiology and 2 papers in Neurology. Recurrent topics in Leonóra Himer's work include Adenosine and Purinergic Signaling (5 papers), Immune Cell Function and Interaction (4 papers) and Calcium signaling and nucleotide metabolism (2 papers). Leonóra Himer is often cited by papers focused on Adenosine and Purinergic Signaling (5 papers), Immune Cell Function and Interaction (4 papers) and Calcium signaling and nucleotide metabolism (2 papers). Leonóra Himer collaborates with scholars based in Hungary, United States and Czechia. Leonóra Himer's co-authors include Pál Pacher, Balázs Csóka, Zsolt Selmeczy, E. Sylvester Vizi, Edwin A. Deitch, György Haskó, Zoltán H. Németh, Balázs Koscsó, Z. Spolarics and Catherine Ledent and has published in prestigious journals such as The Journal of Immunology, The FASEB Journal and Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin.

In The Last Decade

Leonóra Himer

8 papers receiving 553 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Leonóra Himer Hungary	8	413	180	127	114	56	8	557
Jonathan B. Volmer United States	8	435 1.1×	110 0.6×	130 1.0×	88 0.8×	71 1.3×	8	601
Laura M. Kreckler United States	7	300 0.7×	76 0.4×	147 1.2×	69 0.6×	24 0.4×	10	428
W.A. Buurman Netherlands	7	144 0.3×	116 0.6×	83 0.7×	49 0.4×	52 0.9×	9	419
Sarah Sargin Germany	8	178 0.4×	135 0.8×	182 1.4×	31 0.3×	74 1.3×	11	459
Marzena Grdeń Poland	12	244 0.6×	62 0.3×	91 0.7×	36 0.3×	91 1.6×	23	399
Christian Karcher Australia	5	153 0.4×	63 0.3×	143 1.1×	21 0.2×	37 0.7×	10	357
German Reyes Canada	4	179 0.4×	27 0.1×	91 0.7×	20 0.2×	25 0.4×	5	318
Charles Kresge United States	12	112 0.3×	44 0.2×	225 1.8×	11 0.1×	121 2.2×	16	500
Mark D. Okusa United States	9	57 0.1×	77 0.4×	191 1.5×	71 0.6×	49 0.9×	12	407
Michael G. Rolf United Kingdom	7	225 0.5×	53 0.3×	110 0.9×	16 0.1×	23 0.4×	13	439

Countries citing papers authored by Leonóra Himer

Since Specialization

Citations

This map shows the geographic impact of Leonóra Himer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leonóra Himer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leonóra Himer more than expected).

Fields of papers citing papers by Leonóra Himer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leonóra Himer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leonóra Himer. The network helps show where Leonóra Himer may publish in the future.

Co-authorship network of co-authors of Leonóra Himer

This figure shows the co-authorship network connecting the top 25 collaborators of Leonóra Himer. A scholar is included among the top collaborators of Leonóra Himer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leonóra Himer. Leonóra Himer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

8 of 8 papers shown

1.

Vörös, Péter, Erna Sziksz, Leonóra Himer, et al.. (2013). Expression of PARK7 is increased in celiac disease. Archiv für Pathologische Anatomie und Physiologie und für Klinische Medicin. 463(3). 401–408. 10 indexed citations

2.

Himer, Leonóra, et al.. (2012). Membrane microdomain organization, calcium signal, and NFAT activation as an important axis in polarized Th cell function. Cytometry Part A. 83A(2). 185–196. 18 indexed citations

3.

Koscsó, Balázs, Balázs Csóka, Zsolt Selmeczy, et al.. (2011). Adenosine Augments IL-10 Production by Microglial Cells through an A2B Adenosine Receptor-Mediated Process. The Journal of Immunology. 188(1). 445–453. 100 indexed citations

4.

Himer, Leonóra, Attila Balog, Beáta Szebeni, et al.. (2010). Th17 cells in rheumatoid arthritis. Orvosi Hetilap. 151(25). 1003–1010. 8 indexed citations

5.

Csóka, Balázs, Zoltán H. Németh, Peter Rosenberger, et al.. (2010). A2B Adenosine Receptors Protect against Sepsis-Induced Mortality by Dampening Excessive Inflammation. The Journal of Immunology. 185(1). 542–550. 115 indexed citations

6.

Himer, Leonóra, Balázs Csóka, Zsolt Selmeczy, et al.. (2010). Adenosine A _2A receptor activation protects CD4 ⁺ T lymphocytes against activation‐induced cell death. The FASEB Journal. 24(8). 2631–2640. 60 indexed citations

7.

Csóka, Balázs, Leonóra Himer, Zsolt Selmeczy, et al.. (2008). Adenosine A _2A receptor activation inhibits T helper 1 and T helper 2 cell development and effector function. The FASEB Journal. 22(10). 3491–3499. 155 indexed citations

8.

Németh, Zoltán, David Bleich, Balázs Csóka, et al.. (2007). Adenosine receptor activation ameliorates type 1 diabetes. The FASEB Journal. 21(10). 2379–2388. 91 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact