Leonard B. Maggi

4.7k total citations · 1 hit paper
38 papers, 3.2k citations indexed

About

Leonard B. Maggi is a scholar working on Molecular Biology, Cancer Research and Immunology. According to data from OpenAlex, Leonard B. Maggi has authored 38 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 10 papers in Cancer Research and 8 papers in Immunology. Recurrent topics in Leonard B. Maggi's work include RNA regulation and disease (11 papers), RNA modifications and cancer (8 papers) and RNA Research and Splicing (7 papers). Leonard B. Maggi is often cited by papers focused on RNA regulation and disease (11 papers), RNA modifications and cancer (8 papers) and RNA Research and Splicing (7 papers). Leonard B. Maggi collaborates with scholars based in United States, Canada and Italy. Leonard B. Maggi's co-authors include Jason D. Weber, Brandon Faubert, Erika L. Pearce, Gerritje J. W. van der Windt, Julianna Blagih, Alejandro V. Villarino, David O’Sullivan, Jing Qiu, Chih‐Hao Chang and Stanley Ching‐Cheng Huang and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Leonard B. Maggi

37 papers receiving 3.2k citations

Hit Papers

Posttranscriptional Control of T Cell Effector Function b... 2013 2026 2017 2021 2013 500 1000 1.5k

Peers

Leonard B. Maggi
Laura L. McCormick United States
Joseph Barbi United States
K. Venuprasad United States
Jillian Nicholl Australia
Raymond Davis United States
Peter Schow United States
Laura L. McCormick United States
Leonard B. Maggi
Citations per year, relative to Leonard B. Maggi Leonard B. Maggi (= 1×) peers Laura L. McCormick

Countries citing papers authored by Leonard B. Maggi

Since Specialization
Citations

This map shows the geographic impact of Leonard B. Maggi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leonard B. Maggi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leonard B. Maggi more than expected).

Fields of papers citing papers by Leonard B. Maggi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leonard B. Maggi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leonard B. Maggi. The network helps show where Leonard B. Maggi may publish in the future.

Co-authorship network of co-authors of Leonard B. Maggi

This figure shows the co-authorship network connecting the top 25 collaborators of Leonard B. Maggi. A scholar is included among the top collaborators of Leonard B. Maggi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leonard B. Maggi. Leonard B. Maggi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sharma, Ashish, Patrick W. Sheehan, Leonard B. Maggi, et al.. (2025). Reconstructed cell-type-specific rhythms in human brain link Alzheimer’s pathology, circadian stress, and ribosomal disruption. Neuron. 113(17). 2822–2838.e7. 3 indexed citations
2.
Kung, Che-Pei, Kyle A. Cottrell, Raleigh D. Kladney, et al.. (2021). Correction to: Evaluating the therapeutic potential of ADAR1 inhibition for triple-negative breast cancer. Oncogene. 40(11). 2147–2147. 3 indexed citations
3.
Kung, Che-Pei, Kyle A. Cottrell, Raleigh D. Kladney, et al.. (2020). Evaluating the therapeutic potential of ADAR1 inhibition for triple-negative breast cancer. Oncogene. 40(1). 189–202. 55 indexed citations
4.
Mahajan, Nitin, Melissa L. Bates, Chakrapani Tripathi, et al.. (2019). The snoRNA target of t(4;14) in multiple myeloma regulates ribosome biogenesis. FASEB BioAdvances. 1(7). 404–414. 18 indexed citations
5.
Govindan, Ramaswamy, Siddhartha Devarakonda, Deepali Jain, et al.. (2019). Cancer Genomics for the Clinician. 3 indexed citations
6.
Kung, Che-Pei, Leonard B. Maggi, & Jason D. Weber. (2018). The Role of RNA Editing in Cancer Development and Metabolic Disorders. Frontiers in Endocrinology. 9. 762–762. 69 indexed citations
7.
Chang, Stephanie H., Reza Ghasemi, Ryuji Higashikubo, et al.. (2016). Deficiency of the adaptor protein SLy1 results in a natural killer cell ribosomopathy affecting tumor clearance. OncoImmunology. 5(12). e1238543–e1238543. 6 indexed citations
8.
Saporita, Anthony J., et al.. (2014). ARF and p53 Coordinate Tumor Suppression of an Oncogenic IFN-β-STAT1-ISG15 Signaling Axis. Cell Reports. 7(2). 514–526. 46 indexed citations
9.
Maggi, Leonard B., et al.. (2014). ARF tumor suppression in the nucleolus. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1842(6). 831–839. 56 indexed citations
10.
Chang, Chih‐Hao, Jonathan D. Curtis, Leonard B. Maggi, et al.. (2013). Posttranscriptional Control of T Cell Effector Function by Aerobic Glycolysis. Cell. 153(6). 1239–1251. 1623 indexed citations breakdown →
11.
Chu, Liang, Mack Y. Su, Leonard B. Maggi, et al.. (2012). Multiple myeloma–associated chromosomal translocation activates orphan snoRNA ACA11 to suppress oxidative stress. Journal of Clinical Investigation. 122(8). 2793–2806. 75 indexed citations
12.
Kladney, Raleigh D., et al.. (2012). Cathepsin K-Cre Causes Unexpected Germline Deletion of Genes in Mice. PLoS ONE. 7(7). e42005–e42005. 26 indexed citations
13.
Maggi, Leonard B., et al.. (2009). Nucleophosmin protein expression level, but not threonine 198 phosphorylation, is essential in growth and proliferation. Oncogene. 28(36). 3209–3220. 19 indexed citations
14.
Saporita, Anthony J., Leonard B. Maggi, Anthony J. Apicelli, & Jason D. Weber. (2007). Therapeutic Targets in the ARF Tumor Suppressor Pathway. Current Medicinal Chemistry. 14(17). 1815–1827. 34 indexed citations
15.
Moran, Jason M., et al.. (2006). Role of MAPK in the Regulation of Double-Stranded RNA- and Encephalomyocarditis Virus-Induced Cyclooxygenase-2 Expression by Macrophages. The Journal of Immunology. 177(5). 3413–3420. 50 indexed citations
16.
Yu, Yue, Leonard B. Maggi, Anthony J. Apicelli, et al.. (2006). Nucleophosmin Is Essential for Ribosomal Protein L5 Nuclear Export. Molecular and Cellular Biology. 26(10). 3798–3809. 177 indexed citations
17.
Maggi, Leonard B. & Jason D. Weber. (2005). Nucleolar Adaptation in Human Cancer. Cancer Investigation. 23(7). 599–608. 66 indexed citations
18.
Maggi, Leonard B., Jason M. Moran, R. Mark L. Buller, & John A. Corbett. (2003). ERK Activation Is Required for Double-stranded RNA- and Virus-induced Interleukin-1 Expression by Macrophages. Journal of Biological Chemistry. 278(19). 16683–16689. 39 indexed citations
19.
Maggi, Leonard B., Jason M. Moran, Anna L. Scarim, et al.. (2002). Novel Role for Calcium-independent Phospholipase A2in the Macrophage Antiviral Response of Inducible Nitric-oxide Synthase Expression. Journal of Biological Chemistry. 277(41). 38449–38455. 36 indexed citations
20.
Maggi, Leonard B., et al.. (2002). Role of Interferon Regulatory Factor-1 in Double-stranded RNA-induced iNOS Expression by Mouse Islets. Journal of Biological Chemistry. 277(1). 359–365. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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