Leila Khamzina

759 total citations
7 papers, 618 citations indexed

About

Leila Khamzina is a scholar working on Molecular Biology, Surgery and Hepatology. According to data from OpenAlex, Leila Khamzina has authored 7 papers receiving a total of 618 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 2 papers in Surgery and 2 papers in Hepatology. Recurrent topics in Leila Khamzina's work include Protein Kinase Regulation and GTPase Signaling (2 papers), Metabolism, Diabetes, and Cancer (2 papers) and Pancreatic function and diabetes (2 papers). Leila Khamzina is often cited by papers focused on Protein Kinase Regulation and GTPase Signaling (2 papers), Metabolism, Diabetes, and Cancer (2 papers) and Pancreatic function and diabetes (2 papers). Leila Khamzina collaborates with scholars based in Canada, United States and Germany. Leila Khamzina's co-authors include Alain Veilleux, Sébastien Bergeron, André Marette, Jack R. Wands, P Borgeat, Marc Pouliot, Shaun R. McColl, Patrick P. McDonald, Philip A. Gruppuso and Franz Ludwig Dumoulin and has published in prestigious journals such as The Journal of Immunology, Gastroenterology and Endocrinology.

In The Last Decade

Leila Khamzina

7 papers receiving 598 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Leila Khamzina Canada 6 337 192 161 128 71 7 618
Shuichi Otabe Japan 14 244 0.7× 208 1.1× 323 2.0× 174 1.4× 158 2.2× 22 700
Kelly Hester United States 10 258 0.8× 148 0.8× 193 1.2× 101 0.8× 41 0.6× 23 536
Su Sung Kim South Korea 9 274 0.8× 164 0.9× 248 1.5× 169 1.3× 86 1.2× 13 662
Leylla Badeanlou United States 7 314 0.9× 188 1.0× 217 1.3× 70 0.5× 62 0.9× 7 611
Sangwon Byun United States 13 395 1.2× 93 0.5× 275 1.7× 168 1.3× 69 1.0× 17 695
Toshihiko Hashinaga Japan 10 168 0.5× 184 1.0× 283 1.8× 85 0.7× 119 1.7× 13 511
Ching-Chyuan Hsieh United States 13 369 1.1× 191 1.0× 74 0.5× 36 0.3× 57 0.8× 15 666
Sylvia Jacob Germany 16 311 0.9× 155 0.8× 251 1.6× 131 1.0× 135 1.9× 36 657
Sandra Slusher United States 7 359 1.1× 133 0.7× 167 1.0× 177 1.4× 138 1.9× 8 653
Elizabeth Tomlinson United States 7 884 2.6× 158 0.8× 300 1.9× 342 2.7× 100 1.4× 8 1.3k

Countries citing papers authored by Leila Khamzina

Since Specialization
Citations

This map shows the geographic impact of Leila Khamzina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Leila Khamzina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Leila Khamzina more than expected).

Fields of papers citing papers by Leila Khamzina

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Leila Khamzina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Leila Khamzina. The network helps show where Leila Khamzina may publish in the future.

Co-authorship network of co-authors of Leila Khamzina

This figure shows the co-authorship network connecting the top 25 collaborators of Leila Khamzina. A scholar is included among the top collaborators of Leila Khamzina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Leila Khamzina. Leila Khamzina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Khamzina, Leila, Alain Veilleux, Sébastien Bergeron, & André Marette. (2004). Increased Activation of the Mammalian Target of Rapamycin Pathway in Liver and Skeletal Muscle of Obese Rats: Possible Involvement in Obesity-Linked Insulin Resistance. Endocrinology. 146(3). 1473–1481. 469 indexed citations
2.
Dumoulin, Franz Ludwig, Annette von dem Bussche, Jisu Li, et al.. (2003). Hepatitis C Virus NS2 Protein Inhibits Gene Expression from Different Cellular and Viral Promoters in Hepatic and Nonhepatic Cell Lines. Virology. 305(2). 260–266. 52 indexed citations
3.
Khamzina, Leila, Philip A. Gruppuso, & Jack R. Wands. (2003). Insulin signaling through insulin receptor substrate 1 and 2 in normal liver development. Gastroenterology. 125(2). 572–585. 26 indexed citations
4.
Khamzina, Leila & Pierre Borgeat. (1998). Correlation of α-Fetoprotein Expression in Normal Hepatocytes during Development with Tyrosine Phosphorylation and Insulin Receptor Expression. Molecular Biology of the Cell. 9(5). 1093–1105. 9 indexed citations
5.
Khamzina, Leila, et al.. (1995). Alpha-fetoprotein (AFP) expression in clones of McA-RH 7777 rat hepatoma: correlation with the occurrence of homogeneously staining regions on chromosome 14.. PubMed. 55(16). 3615–22. 6 indexed citations
6.
Pouliot, Marc, Patrick P. McDonald, Leila Khamzina, P Borgeat, & Shaun R. McColl. (1994). Granulocyte-macrophage colony-stimulating factor enhances 5-lipoxygenase levels in human polymorphonuclear leukocytes.. The Journal of Immunology. 152(2). 851–858. 52 indexed citations
7.
Khamzina, Leila, et al.. (1988). Phenotypic variability of cultured rat hepatoma cell population in respect to alpha‐fetoprotein synthesis. International Journal of Cancer. 42(4). 633–637. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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