Laura Kivinen
- Co-authors
- Marikki LaihoUlf ErikssonKari AlitaloJuha KlefströmBirgitta OlofssonVladimir JoukovVijay KumarAri Ristimäki
- Topics
- Cancer-related Molecular Pathways (7 papers)Cancer, Hypoxia, and Metabolism (4 papers)TGF-β signaling in diseases (2 papers)
- Journals
- OncogeneExperimental Cell ResearchPubMed
- Partner nations
- FinlandUnited StatesSweden
In The Last Decade
Laura Kivinen
8 papers receiving 554 citations
Peers
Comparison fields: 5 of 60
- Molecular Biology 454
- Oncology 287
- Cancer Research 153
- Surgery 38
- Cell Biology 38
Countries citing papers authored by Laura Kivinen
This map shows the geographic impact of Laura Kivinen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Laura Kivinen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Laura Kivinen more than expected).
Fields of papers citing papers by Laura Kivinen
This network shows the impact of papers produced by Laura Kivinen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Laura Kivinen. The network helps show where Laura Kivinen may publish in the future.
Co-authorship network of co-authors of Laura Kivinen
This figure shows the co-authorship network connecting the top 25 collaborators of Laura Kivinen. A scholar is included among the top collaborators of Laura Kivinen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Laura Kivinen. Laura Kivinen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 52 | |
| 2 | Ras- and mitogen-activated protein kinase kinase-dependent and -independent pathways in p21Cip1/Waf1 induction by fibroblast growth factor-2, platelet-derived growth factor, and transforming growth factor-beta1. | 30 |
| 3 | 56 | |
| 4 | 379 | |
| 5 | p21ras-mediated decrease of the retinoblastoma protein in fibroblasts occurs through growth factor-dependent mechanisms. | 3 |
| 6 | Cell cycle dependent effects of u.v.-radiation on p53 expression and retinoblastoma protein phosphorylation. | 33 |
| 7 | 5 | |
| 8 | Human retinoblastoma gene product prevents c-Ha-ras oncogene mediated cellular transformation of mouse fibroblasts. | 7 |
About Laura Kivinen
Laura Kivinen is a scholar working on Cancer Research, Oncology and Biotechnology, having authored 8 papers that have together received 565 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (7 papers), Cancer, Hypoxia, and Metabolism (4 papers) and TGF-β signaling in diseases (2 papers). The work is most often cited by research in Oncology (287 citations), Cancer Research (153 citations) and Molecular Biology (454 citations). Laura Kivinen has collaborated with scholars based in Finland, United States and Sweden. Frequent co-authors include Marikki Laiho, Ulf Eriksson, Kari Alitalo, Juha Klefström, Birgitta Olofsson, Vladimir Joukov, Vijay Kumar, Ari Ristimäki, Karri Paavonen and Yuji Gunji. Their work appears in journals such as Oncogene, Experimental Cell Research and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.