Lanping Quan

920 total citations
21 papers, 774 citations indexed

About

Lanping Quan is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Lanping Quan has authored 21 papers receiving a total of 774 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 7 papers in Oncology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Lanping Quan's work include Wnt/β-catenin signaling in development and cancer (9 papers), Cancer-related gene regulation (9 papers) and Kruppel-like factors research (6 papers). Lanping Quan is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (9 papers), Cancer-related gene regulation (9 papers) and Kruppel-like factors research (6 papers). Lanping Quan collaborates with scholars based in China, United States and India. Lanping Quan's co-authors include Hongxia Zhu, Ningzhi Xu, Jinfeng Bai, Yihua Wang, Xiaobo Zhou, Cuiqi Zhou, Shangbin Yang, Shuang Liu, Qimin Zhan and Shun He and has published in prestigious journals such as Oncogene, International Journal of Cancer and Cellular and Molecular Life Sciences.

In The Last Decade

Lanping Quan

21 papers receiving 760 citations

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author Last Decade Papers Cites
Lanping Quan 565 210 179 131 81 21 774
Jan‐Patrik Wiksten 390 0.7× 213 1.0× 154 0.9× 127 1.0× 86 1.1× 10 646
Junhye Kwon 594 1.1× 274 1.3× 205 1.1× 99 0.8× 51 0.6× 31 854
Rinat Keren 532 0.9× 327 1.6× 160 0.9× 141 1.1× 43 0.5× 5 763
Azadeh Arabi 672 1.2× 193 0.9× 123 0.7× 59 0.5× 70 0.9× 14 795
Marisa Mariani 476 0.8× 249 1.2× 235 1.3× 148 1.1× 44 0.5× 24 758
Clare G. Fedele 513 0.9× 168 0.8× 119 0.7× 146 1.1× 45 0.6× 15 685
Martina Keith 419 0.7× 155 0.7× 115 0.6× 220 1.7× 74 0.9× 15 685
Nikolina Vlatković 626 1.1× 388 1.8× 169 0.9× 88 0.7× 69 0.9× 26 828
Gary Oxford 646 1.1× 280 1.3× 164 0.9× 161 1.2× 109 1.3× 13 878
Thomas Mund 687 1.2× 274 1.3× 92 0.5× 157 1.2× 68 0.8× 14 892

Countries citing papers authored by Lanping Quan

Since Specialization
Citations

This map shows the geographic impact of Lanping Quan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lanping Quan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lanping Quan more than expected).

Fields of papers citing papers by Lanping Quan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lanping Quan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lanping Quan. The network helps show where Lanping Quan may publish in the future.

Co-authorship network of co-authors of Lanping Quan

This figure shows the co-authorship network connecting the top 25 collaborators of Lanping Quan. A scholar is included among the top collaborators of Lanping Quan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lanping Quan. Lanping Quan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Wei, Hua Xiong, Yanmei Zou, et al.. (2016). Frequently rearranged in advanced T-cell lymphomas-1 demonstrates oncogenic properties in prostate cancer. Molecular Medicine Reports. 14(4). 3551–3558. 2 indexed citations
2.
Wang, Zaozao, Mei Liu, Hongxia Zhu, et al.. (2012). miR‐106a is frequently upregulated in gastric cancer and inhibits the extrinsic apoptotic pathway by targeting FAS. Molecular Carcinogenesis. 52(8). 634–646. 67 indexed citations
3.
4.
Zhu, Hongxia, Qifeng Wang, Chenfei Hu, et al.. (2011). High expression of survivin predicts poor prognosis in esophageal squamous cell carcinoma following radiotherapy. Tumor Biology. 32(6). 1147–1153. 27 indexed citations
5.
Zhu, Hongxia, Guo Zhang, Yan Wang, et al.. (2010). Inhibition of ErbB2 by Herceptin reduces survivin expression via the ErbB2–β‐catenin/TCF4‐survivin pathway in ErbB2‐overexpressed breast cancer cells. Cancer Science. 101(5). 1156–1162. 32 indexed citations
6.
He, Shun, Shangbin Yang, Mei Liu, et al.. (2010). Aurora kinase A induces miR-17-92 cluster through regulation of E2F1 transcription factor. Cellular and Molecular Life Sciences. 67(12). 2069–2076. 21 indexed citations
7.
Zhang, Wei, Shuang Yan, Guo Zhang, et al.. (2010). β-Catenin/TCF pathway plays a vital role in selenium induced-growth inhibition and apoptosis in esophageal squamous cell carcinoma (ESCC) cells. Cancer Letters. 296(1). 113–122. 21 indexed citations
8.
Yang, Shangbin, Shun He, Xiaobo Zhou, et al.. (2010). Suppression of Aurora-A oncogenic potential by c-Myc downregulation. Experimental & Molecular Medicine. 42(11). 759–759. 31 indexed citations
9.
Liu, Mei, Shangbin Yang, Yihua Wang, et al.. (2008). EB1 acts as an oncogene via activating β‐catenin/TCF pathway to promote cellular growth and inhibit apoptosis. Molecular Carcinogenesis. 48(3). 212–219. 34 indexed citations
10.
Wang, Yihua, Shuang Liu, Hongxia Zhu, et al.. (2008). FRAT1 overexpression leads to aberrant activation of β‐catenin/TCF pathway in esophageal squamous cell carcinoma. International Journal of Cancer. 123(3). 561–568. 34 indexed citations
11.
Yang, Shangbin, Xiaobo Zhou, Hongxia Zhu, et al.. (2007). Amplification and overexpression of Aurora-A in esophageal squamous cell carcinoma. Oncology Reports. 17(5). 1083–8. 49 indexed citations
12.
Huang, Moli, Yihua Wang, Daochun Sun, et al.. (2006). Identification of genes regulated by Wnt/β-catenin pathway and involved in apoptosis via microarray analysis. BMC Cancer. 6(1). 221–221. 43 indexed citations
13.
14.
Wang, Yihua, Xiaobo Zhou, Hongxia Zhu, et al.. (2005). Overexpression of EB1 in human esophageal squamous cell carcinoma (ESCC) may promote cellular growth by activating β-catenin/TCF pathway. Oncogene. 24(44). 6637–6645. 66 indexed citations
15.
Wang, Yihua, Hongxia Zhu, Lanping Quan, et al.. (2005). Downregulation of survivin by RNAi inhibits the growth of esophageal carcinoma cells. Cancer Biology & Therapy. 4(9). 974–978. 34 indexed citations
16.
Zhu, Hongxia, Yihua Wang, Cuiqi Zhou, et al.. (2005). [Expression of survivin and its significance in esophageal cancer].. PubMed. 27(1). 22–4. 2 indexed citations
17.
Zhou, Cuiqi, Shuang Liu, Xiaobo Zhou, et al.. (2004). Overexpression of human pituitary tumor transforming gene (hPTTG), is regulated by ?-catenin /TCF pathway in human esophageal squamous cell carcinoma. International Journal of Cancer. 113(6). 891–898. 64 indexed citations
18.
Wang, Yihua, Shuang Liu, Cuiqi Zhou, et al.. (2004). Knockdown of c-Myc expression by RNAi inhibits MCF-7 breast tumor cells growth in vitro and in vivo. Breast Cancer Research. 7(2). R220–8. 131 indexed citations
19.
Zhu, Hongxia, Cuiqi Zhou, Xiaobo Zhou, et al.. (2003). [Survivin mutants reverse the malignancy of HeLa cells].. PubMed. 22(5). 467–70. 2 indexed citations
20.
Zhou, Xiaobo, Ning Lü, Wei Zhang, et al.. (2002). [Expression and significance of beta-catenin in esophageal carcinoma].. PubMed. 21(8). 877–80. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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