Lamont G. Weide

1.5k citations
34 papers · 1.1k indexed · h-index 15
Topics
Pancreatic function and diabetes (17 papers)Diabetes and associated disorders (11 papers)Diabetes Management and Research (8 papers)

In The Last Decade

Lamont G. Weide

32 papers receiving 1.1k citations

Peers

Lamont G. Weide
Comparison fields: 5 of 72
  • Surgery 881
  • Endocrinology, Diabetes and Metabolism 558
  • Genetics 435
  • Molecular Biology 215
  • Pharmacology 155
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Citations per field
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Citations per year

Countries citing papers authored by Lamont G. Weide

Since Specialization
Citations

This map shows the geographic impact of Lamont G. Weide's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Lamont G. Weide with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Lamont G. Weide more than expected).

Fields of papers citing papers by Lamont G. Weide

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Lamont G. Weide. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Lamont G. Weide. The network helps show where Lamont G. Weide may publish in the future.

Co-authorship network of co-authors of Lamont G. Weide

This figure shows the co-authorship network connecting the top 25 collaborators of Lamont G. Weide. A scholar is included among the top collaborators of Lamont G. Weide based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Lamont G. Weide. Lamont G. Weide is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 32
2 155
3 63
4 31
5 12
6 32
7 1
8
Experimental evidence for the origin of ductal-type adenocarcinoma from the islets of Langerhans.
50
9 2
10 5
11 6
12
The role of pancreatic islets in experimental pancreatic carcinogenicity.
22
13
Comparison of two microbiological culture techniques used to monitor islet cell isolation contamination.
1
14 67
15 30
16 14
17 9
18 164
19
Intraportal islet allografts: the use of a stimulation index to represent functional results.
5
20 288

About Lamont G. Weide

Lamont G. Weide is a scholar working on Transplantation, Endocrinology, Diabetes and Metabolism and Surgery, having authored 34 papers that have together received 1.1k indexed citations. Recurring topics across this work include Pancreatic function and diabetes (17 papers), Diabetes and associated disorders (11 papers) and Diabetes Management and Research (8 papers). The work is most often cited by research in Transplantation (119 citations), Endocrinology, Diabetes and Metabolism (558 citations) and Surgery (881 citations). Lamont G. Weide has collaborated with scholars based in United States, United Kingdom and Japan. Frequent co-authors include Paul E. Lacy, Piero Marchetti, M. Wayne Flye, Charles B. Anderson, Christopher S. McCullough, Ronald L. Gingerich, Julio V. Santiago, David W. Scharp, Luca Falqui and Philip E. Cryer. Their work appears in journals such as Gastroenterology, Diabetes Care and Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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