LaMonica V. Stewart

1.0k total citations
28 papers, 793 citations indexed

About

LaMonica V. Stewart is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, LaMonica V. Stewart has authored 28 papers receiving a total of 793 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 11 papers in Cancer Research and 8 papers in Pulmonary and Respiratory Medicine. Recurrent topics in LaMonica V. Stewart's work include Prostate Cancer Treatment and Research (8 papers), Cancer, Lipids, and Metabolism (6 papers) and Peroxisome Proliferator-Activated Receptors (6 papers). LaMonica V. Stewart is often cited by papers focused on Prostate Cancer Treatment and Research (8 papers), Cancer, Lipids, and Metabolism (6 papers) and Peroxisome Proliferator-Activated Receptors (6 papers). LaMonica V. Stewart collaborates with scholars based in United States, United Kingdom and Ireland. LaMonica V. Stewart's co-authors include Nancy L. Weigel, Tunde Akinyeke, Tara C. Polek, David Danielpour, Emuejevoke Olokpa, Michael W. Kattan, Sarah E. Blutt, Mary L. Thomas, Elizabeth J. Ryu and Elizabeth A. Allegretto and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Cancer Research.

In The Last Decade

LaMonica V. Stewart

27 papers receiving 767 citations

Peers

LaMonica V. Stewart
Bojie Dai United States
Boris Shorning United Kingdom
Tara C. Polek United States
Y Omoto Japan
M. Vijay Kumar United States
Bryan M. Gillard United States
Bojie Dai United States
LaMonica V. Stewart
Citations per year, relative to LaMonica V. Stewart LaMonica V. Stewart (= 1×) peers Bojie Dai

Countries citing papers authored by LaMonica V. Stewart

Since Specialization
Citations

This map shows the geographic impact of LaMonica V. Stewart's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by LaMonica V. Stewart with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites LaMonica V. Stewart more than expected).

Fields of papers citing papers by LaMonica V. Stewart

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by LaMonica V. Stewart. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by LaMonica V. Stewart. The network helps show where LaMonica V. Stewart may publish in the future.

Co-authorship network of co-authors of LaMonica V. Stewart

This figure shows the co-authorship network connecting the top 25 collaborators of LaMonica V. Stewart. A scholar is included among the top collaborators of LaMonica V. Stewart based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with LaMonica V. Stewart. LaMonica V. Stewart is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Roberts, Ken, Sarah Williams‐Blangero, LaMonica V. Stewart, et al.. (2025). Increasing the genomic workforce through research capacity building: Designing evaluation plans for maximum impact. The American Journal of Human Genetics. 112(5). 967–974. 1 indexed citations
2.
Kanagasabai, Thanigaivelan, et al.. (2024). Co‐targeting SKP2 and KDM5B inhibits prostate cancer progression by abrogating AKT signaling with induction of senescence and apoptosis. The Prostate. 84(9). 877–887. 3 indexed citations
3.
Chen, Zhenbang, et al.. (2024). The KDM5 inhibitor PBIT reduces proliferation of castration-resistant prostate cancer cells via cell cycle arrest and the induction of senescence. Experimental Cell Research. 437(1). 113991–113991. 2 indexed citations
4.
Williams, Stephen D., et al.. (2022). Hypoxia-Inducible Expression of Annexin A6 Enhances the Resistance of Triple-Negative Breast Cancer Cells to EGFR and AR Antagonists. Cells. 11(19). 3007–3007. 10 indexed citations
5.
Olokpa, Emuejevoke, et al.. (2017). Crosstalk between the Androgen Receptor and PPAR Gamma Signaling Pathways in the Prostate. PPAR Research. 2017. 1–13. 22 indexed citations
6.
Olokpa, Emuejevoke, et al.. (2016). The Androgen Receptor Regulates PPARγ Expression and Activity in Human Prostate Cancer Cells. Journal of Cellular Physiology. 231(12). 2664–2672. 33 indexed citations
7.
Akinyeke, Tunde & LaMonica V. Stewart. (2011). Troglitazone suppresses c-Myc levels in human prostate cancer cells via a PPARγ-independent mechanism. Cancer Biology & Therapy. 11(12). 1046–1058. 49 indexed citations
8.
Stewart, LaMonica V., et al.. (2010). The PPARγ ligand ciglitazone regulates androgen receptor activation differently in androgen-dependent versus androgen-independent human prostate cancer cells. Experimental Cell Research. 316(20). 3478–3488. 28 indexed citations
9.
Rennel, E S, Alexander H. R. Varey, LaMonica V. Stewart, et al.. (2009). VEGF121b, a new member of the VEGFxxxb family of VEGF-A splice isoforms, inhibits neovascularisation and tumour growth in vivo. British Journal of Cancer. 101(7). 1183–1193. 43 indexed citations
11.
Stewart, LaMonica V., et al.. (2005). Vitamin D receptor agonists induce prostatic acid phosphatase to reduce cell growth and HER-2 signaling in LNCaP-derived human prostate cancer cells. The Journal of Steroid Biochemistry and Molecular Biology. 97(1-2). 37–46. 12 indexed citations
12.
Stewart, LaMonica V. & Nancy L. Weigel. (2005). Role of insulin‐like growth factor binding proteins in 1α,25‐dihydroxyvitamin D3‐induced growth inhibition of human prostate cancer cells. The Prostate. 64(1). 9–19. 26 indexed citations
14.
Stewart, LaMonica V. & Nancy L. Weigel. (2004). Vitamin D and Prostate Cancer. Experimental Biology and Medicine. 229(4). 277–284. 111 indexed citations
15.
Stewart, LaMonica V., et al.. (2003). Regulation of trespin expression by modulators of cell growth, differentiation, and apoptosis in prostatic epithelial cells. Experimental Cell Research. 284(2). 301–313. 12 indexed citations
16.
Chipuk, Jerry E., et al.. (2002). Identification and Characterization of A Novel Rat Ov-Serpin Family Member, Trespin. Journal of Biological Chemistry. 277(29). 26412–26421. 6 indexed citations
17.
Polek, Tara C., LaMonica V. Stewart, Elizabeth J. Ryu, et al.. (2002). p53 Is Required for 1,25-Dihydroxyvitamin D3-Induced G0 Arrest But Is Not Required for G1 Accumulation or Apoptosis of LNCaP Prostate Cancer Cells. Endocrinology. 144(1). 50–60. 51 indexed citations
18.
Blutt, Sarah E., Tara C. Polek, LaMonica V. Stewart, Michael W. Kattan, & Nancy L. Weigel. (2000). A calcitriol analogue, EB1089, inhibits the growth of LNCaP tumors in nude mice.. PubMed. 60(4). 779–82. 77 indexed citations
19.
Lucia, M. Scott, Michael B. Sporn, Anita B. Roberts, LaMonica V. Stewart, & David Danielpour. (1998). The role of transforming growth factor-β1, -β2, and -β3 in androgen-responsive growth of NRP-152 rat prostatic epithelial cells. Journal of Cellular Physiology. 175(2). 184–192. 41 indexed citations
20.
Stewart, LaMonica V. & Mary L. Thomas. (1997). Retinoids Differentially Regulate the Proliferation of Colon Cancer Cell Lines. Experimental Cell Research. 233(2). 321–329. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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