L. A. WILDBLOOD
- Small Animals top 2%
- Helminth infection and control 5
- Parasitology top 5%
- Parasites and Host Interactions 3
- Animal Science and Zoology top 10%
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- Tuberculosis Research and Epidemiology 2
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- Parasite Biology and Host Interactions 3
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- Mycobacterium research and diagnosis 4
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- Pharmacological Effects of Natural Compounds 2
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- Adenosine and Purinergic Signaling 1
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- High Entropy Alloys Studies 1
- Co-authors
- Douglas JonesJacqueline B. MatthewsAlasdair J. NisbetJohn M. SharpKaren StevensonTom N. McNeillyNeil F. InglisIain J. McKendrick
- Journals
- Veterinary Immunology and Immunopathology (4 papers)Parasite Immunology (3 papers)Veterinary Research Communications (1 paper)
- Partner nations
- United KingdomNorwayUnited States
In The Last Decade
L. A. WILDBLOOD
12 papers receiving 364 citations
Peers
Comparison fields: 5 of 40
- Small Animals 214
- Parasitology 164
- Animal Science and Zoology 44
- Infectious Diseases 80
- Ecology 103
Countries citing papers authored by L. A. WILDBLOOD
This map shows the geographic impact of L. A. WILDBLOOD's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by L. A. WILDBLOOD with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites L. A. WILDBLOOD more than expected).
Fields of papers citing papers by L. A. WILDBLOOD
This network shows the impact of papers produced by L. A. WILDBLOOD. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by L. A. WILDBLOOD. The network helps show where L. A. WILDBLOOD may publish in the future.
Co-authorship network
The 25 scholars most cited alongside L. A. WILDBLOOD, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 90 | |
| 2 | 2011 | 23 | |
| 3 | 2010 | 25 | |
| 4 | 2010 | 25 | |
| 5 | 2008 | 15 | |
| 6 | 2007 | 41 | |
| 7 | 2006 | 8 | |
| 8 | 2005 | 27 | |
| 9 | 2001 | 33 | |
| 10 | 2000 | 16 | |
| 11 | 1998 | 64 | |
| 12 | 1997 | 4 |
About L. A. WILDBLOOD
L. A. WILDBLOOD is a scholar working on Small Animals, Parasitology and Pharmacology, having authored 12 papers that have together received 371 indexed citations. Recurring topics across this work include Helminth infection and control (5 papers), Mycobacterium research and diagnosis (4 papers), Parasite Biology and Host Interactions (3 papers), Parasites and Host Interactions (3 papers), Tuberculosis Research and Epidemiology (2 papers), Pharmacological Effects of Natural Compounds (2 papers), Adenosine and Purinergic Signaling (1 paper) and High Entropy Alloys Studies (1 paper). The work is most often cited by research in Small Animals (214 citations), Parasitology (164 citations) and Animal Science and Zoology (44 citations). L. A. WILDBLOOD has collaborated with scholars based in United Kingdom, Norway and United States. Frequent co-authors include Douglas Jones, Jacqueline B. Matthews, Alasdair J. Nisbet, John M. Sharp, Karen Stevenson, Tom N. McNeilly, Neil F. Inglis, Iain J. McKendrick, David J. Bartley and Yvonne Bartley. Their work appears in journals such as Veterinary Immunology and Immunopathology, Parasite Immunology, Veterinary Research Communications, Vaccine and Journal of Clinical Microbiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.