Kwang‐Yu Chang

2.0k total citations
58 papers, 1.3k citations indexed

About

Kwang‐Yu Chang is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Kwang‐Yu Chang has authored 58 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 21 papers in Oncology and 17 papers in Cancer Research. Recurrent topics in Kwang‐Yu Chang's work include Cancer, Hypoxia, and Metabolism (10 papers), Glioma Diagnosis and Treatment (9 papers) and Histone Deacetylase Inhibitors Research (6 papers). Kwang‐Yu Chang is often cited by papers focused on Cancer, Hypoxia, and Metabolism (10 papers), Glioma Diagnosis and Treatment (9 papers) and Histone Deacetylase Inhibitors Research (6 papers). Kwang‐Yu Chang collaborates with scholars based in Taiwan, United States and United Kingdom. Kwang‐Yu Chang's co-authors include Jang‐Yang Chang, Jian‐Ying Chuang, Wen‐Chang Chang, Chia-Jui Yen, Tsung‐I Hsu, Chia-Hung Chien, Wei‐Ting Hsueh, Chien‐Feng Li, Shang‐Hung Chen and Jing‐Ping Liou and has published in prestigious journals such as Journal of Clinical Oncology, SHILAP Revista de lepidopterología and Immunity.

In The Last Decade

Kwang‐Yu Chang

57 papers receiving 1.3k citations

Peers

Kwang‐Yu Chang
Mei Dong China
Elie Traer United States
Ye Song China
Joshua C. Anderson United States
Quintin Pan United States
Kwang‐Yu Chang
Citations per year, relative to Kwang‐Yu Chang Kwang‐Yu Chang (= 1×) peers Katrin E. Tagscherer

Countries citing papers authored by Kwang‐Yu Chang

Since Specialization
Citations

This map shows the geographic impact of Kwang‐Yu Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kwang‐Yu Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kwang‐Yu Chang more than expected).

Fields of papers citing papers by Kwang‐Yu Chang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kwang‐Yu Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kwang‐Yu Chang. The network helps show where Kwang‐Yu Chang may publish in the future.

Co-authorship network of co-authors of Kwang‐Yu Chang

This figure shows the co-authorship network connecting the top 25 collaborators of Kwang‐Yu Chang. A scholar is included among the top collaborators of Kwang‐Yu Chang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kwang‐Yu Chang. Kwang‐Yu Chang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Shen, Chih‐Jie, Amandeep Thakur, I‐Chung Chen, et al.. (2025). Contribution of Prostaglandin E2‐Induced Neuronal Excitation to Drug Resistance in Glioblastoma Countered by a Novel Blood–Brain Barrier Crossing Celecoxib Derivative. Advanced Science. 12(38). e06336–e06336. 1 indexed citations
2.
Chiang, Nai‐Jung, Li-Yuan Bai, Chang‐Fang Chiu, et al.. (2025). A phase I study of liposomal Irinotecan (ONIVYDE®) in combination with TAS-102 (LONSURF®) in refractory solid tumors. Investigational New Drugs. 43(3). 709–718. 1 indexed citations
3.
Yang, Wen‐Bin, Chia-Hung Chien, Jian‐Ying Chuang, et al.. (2024). CXCR7 activation evokes the anti-PD-L1 antibody against glioblastoma by remodeling CXCL12-mediated immunity. Cell Death and Disease. 15(6). 434–434. 2 indexed citations
4.
Chang, Wen‐Chang, et al.. (2023). The complex role of eicosanoids in the brain: Implications for brain tumor development and therapeutic opportunities. Biochimica et Biophysica Acta (BBA) - Reviews on Cancer. 1878(5). 188957–188957. 4 indexed citations
5.
Chien, Chia-Hung, Wen‐Bin Yang, Jian‐Ying Chuang, et al.. (2022). SH3GLB1-related autophagy mediates mitochondrial metabolism to acquire resistance against temozolomide in glioblastoma. Journal of Experimental & Clinical Cancer Research. 41(1). 220–220. 11 indexed citations
6.
Tsai, Yu-Ting, Pin‐Yuan Chen, Chiung‐Yuan Ko, et al.. (2022). Reprogramming of arachidonate metabolism confers temozolomide resistance to glioblastoma through enhancing mitochondrial activity in fatty acid oxidation. Journal of Biomedical Science. 29(1). 21–21. 40 indexed citations
7.
Debele, Tilahun Ayane, Ping-Ching Wu, Yu‐Feng Wei, et al.. (2021). Transferrin Modified GSH Sensitive Hyaluronic Acid Derivative Micelle to Deliver HSP90 Inhibitors to Enhance the Therapeutic Efficacy of Brain Cancers. Cancers. 13(10). 2375–2375. 8 indexed citations
8.
Yang, Wen‐Bin, Tsung‐I Hsu, Jing‐Ping Liou, et al.. (2021). Histone deacetylase 6 acts upstream of DNA damage response activation to support the survival of glioblastoma cells. Cell Death and Disease. 12(10). 884–884. 23 indexed citations
9.
Shen, Chih‐Jie, Kwang‐Yu Chang, Bo-Wen Lin, et al.. (2020). Oleic acid-induced NOX4 is dependent on ANGPTL4 expression to promote human colorectal cancer metastasis. Theranostics. 10(16). 7083–7099. 49 indexed citations
10.
Chien, Chia-Hung, Jian‐Ying Chuang, Shun‐Tai Yang, et al.. (2019). Enrichment of superoxide dismutase 2 in glioblastoma confers to acquisition of temozolomide resistance that is associated with tumor-initiating cell subsets. Journal of Biomedical Science. 26(1). 77–77. 29 indexed citations
11.
Chien, Chia-Hung, Wei‐Ting Hsueh, Jian‐Ying Chuang, & Kwang‐Yu Chang. (2019). Role of autophagy in therapeutic resistance of glioblastoma. Journal of Cancer Metastasis and Treatment. 2019. 12 indexed citations
12.
Chang, Kwang‐Yu, et al.. (2018). Abstract 4887: Mitochondrial SOD2 is the mainstay to protect the stemness-featured glioblastoma cells against drug-induced reactive oxygen stress. Cancer Research. 78(13_Supplement). 4887–4887. 3 indexed citations
13.
Chang, Kwang‐Yu, Tsung‐I Hsu, Cheng‐Keng Chuang, et al.. (2017). Stress stimuli induce cancer-stemness gene expression via Sp1 activation leading to therapeutic resistance in glioblastoma. Biochemical and Biophysical Research Communications. 493(1). 14–19. 25 indexed citations
14.
Chang, Kwang‐Yu, et al.. (2014). Nephrotic Syndrome Associated with Thymoma. SHILAP Revista de lepidopterología. 1 indexed citations
15.
Chen, Tzu‐Ju, Sung‐Wei Lee, Li‐Ching Lin, et al.. (2014). Cyclin-dependent kinase 4 overexpression is mostly independent of gene amplification and constitutes an independent prognosticator for nasopharyngeal carcinoma. Tumor Biology. 35(7). 7209–7216. 9 indexed citations
16.
Li, Chien‐Feng, Sung‐Wei Lee, Tzu‐Ju Chen, et al.. (2013). Overexpression of stathmin 1 confers an independent prognostic indicator in nasopharyngeal carcinoma. Tumor Biology. 35(3). 2619–2629. 41 indexed citations
18.
Chang, Kwang‐Yu, et al.. (2011). H+-myo-Inositol Transporter SLC2A13 as a Potential Marker for Cancer Stem Cells in an Oral Squamous Cell Carcinoma. Current Cancer Drug Targets. 11(8). 966–975. 19 indexed citations
19.
Chien, Chia‐Chi, Ivan M. Kempson, Y. Hwu, et al.. (2011). Complete microscale profiling of tumor microangiogenesis. Biotechnology Advances. 31(3). 396–401. 10 indexed citations
20.
Lu, Chin‐Li, et al.. (2010). Upper aerodigestive tract lymphoma in Taiwan. Journal of Clinical Pathology. 63(10). 888–893. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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