Kun‐Tu Yeh

4.7k citations
151 papers · 3.7k indexed · h-index 35

Impact in

Papers in

    • Cancer-related gene regulation 22
    • Epigenetics and DNA Methylation 15
    • RNA modifications and cancer 14
    • Glycosylation and Glycoproteins Research 10
    • Cancer-related Molecular Pathways 10

Kun‐Tu Yeh

149 papers receiving 3.7k citations

Peers

Kun‐Tu Yeh
Comparison fields: 5 of 122
  • Endocrine and Autonomic Systems 689
  • Aging 160
  • Cancer Research 751
  • Molecular Biology 2.0k
  • Oncology 783
Replace Brian J. Altman with:
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Citations per field
00.5×1.5×2.3×
Brian J. Altman · 1×
Citations per year

Countries citing papers authored by Kun‐Tu Yeh

Since Specialization
Citations

This map shows the geographic impact of Kun‐Tu Yeh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kun‐Tu Yeh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kun‐Tu Yeh more than expected).

Fields of papers citing papers by Kun‐Tu Yeh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kun‐Tu Yeh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kun‐Tu Yeh. The network helps show where Kun‐Tu Yeh may publish in the future.

Co-authors

The 25 scholars most cited alongside Kun‐Tu Yeh, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kun‐Tu Yeh Line = papers co-authored together Kun‐Tu Yeh links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 151 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005337
2 2008141
3 2005106
4 201498
5 200982
6 201179
7 200670
8 201867
9 201466
10 200661
11 201159
12 201355
13 201354
14 201153
15 201652
16 200951
17 200850
18 201449
19
Epigenetic changes of tumor suppressor genes, P15, P16, VHL and P53 in oral cancer.
200347
20 200646

About Kun‐Tu Yeh

Kun‐Tu Yeh is a scholar working on Molecular Biology, Oncology, Cancer Research, Pulmonary and Respiratory Medicine and Pathology and Forensic Medicine, having authored 151 papers that have together received 3.7k indexed citations. Recurring topics across this work include Cancer-related gene regulation (22 papers), Epigenetics and DNA Methylation (15 papers), RNA modifications and cancer (14 papers), Genetic factors in colorectal cancer (11 papers), Glycosylation and Glycoproteins Research (10 papers), Cancer-related Molecular Pathways (10 papers), Lung Cancer Treatments and Mutations (10 papers) and Cancer-related molecular mechanisms research (9 papers). The work is most often cited by research in Endocrine and Autonomic Systems (689 citations), Aging (160 citations), Cancer Research (751 citations), Molecular Biology (2.0k citations) and Oncology (783 citations). Kun‐Tu Yeh has collaborated with scholars based in Taiwan, United States and Singapore. Frequent co-authors include Jan‐Gowth Chang, Ya‐Sian Chang, Ming‐Feng Hou, Shou‐Jen Kuo, Shou‐Tung Chen, Kong‐Bung Choo, Yueh‐Min Lin, Jui‐Chang Chen, Shu‐Hui Lin and Ming‐Yu Yang. Their work appears in journals such as Clinica Chimica Acta, Medical Oncology, International Journal of Molecular Sciences, PLoS ONE and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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