KT Preissner

417 total citations
14 papers, 375 citations indexed

About

KT Preissner is a scholar working on Hematology, Cancer Research and Genetics. According to data from OpenAlex, KT Preissner has authored 14 papers receiving a total of 375 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Hematology, 5 papers in Cancer Research and 3 papers in Genetics. Recurrent topics in KT Preissner's work include Blood Coagulation and Thrombosis Mechanisms (6 papers), Protease and Inhibitor Mechanisms (5 papers) and Platelet Disorders and Treatments (4 papers). KT Preissner is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (6 papers), Protease and Inhibitor Mechanisms (5 papers) and Platelet Disorders and Treatments (4 papers). KT Preissner collaborates with scholars based in Germany, Austria and Italy. KT Preissner's co-authors include G. Müller‐Berghaus, E M Anders, Inés Martín-Padura, Adriana Zanetti, G Conforti, Elisabetta Dejana, Sibylle Hess, Elena Ghibaudi, Hal Morgenstern and Lars E. French and has published in prestigious journals such as Blood, Cardiovascular Research and American Journal of Ophthalmology.

In The Last Decade

KT Preissner

14 papers receiving 361 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
KT Preissner Germany 7 195 154 143 87 83 14 375
M. Pucci Italy 8 118 0.6× 252 1.6× 81 0.6× 32 0.4× 161 1.9× 16 394
Sallouha Aïdoudi France 6 144 0.7× 37 0.2× 160 1.1× 37 0.4× 126 1.5× 8 351
Merete Thune Wiiger Norway 11 233 1.2× 96 0.6× 39 0.3× 47 0.5× 201 2.4× 20 515
Nancy E. Kirschbaum United States 9 249 1.3× 51 0.3× 160 1.1× 66 0.8× 310 3.7× 13 635
V L Woods United States 11 468 2.4× 57 0.4× 334 2.3× 132 1.5× 89 1.1× 15 724
Hidehiro Nomura Japan 8 134 0.7× 408 2.6× 113 0.8× 51 0.6× 146 1.8× 13 548
Shunichi Ishigami Japan 6 93 0.5× 83 0.5× 36 0.3× 41 0.5× 105 1.3× 25 343
JP Rosa France 9 469 2.4× 36 0.2× 270 1.9× 134 1.5× 143 1.7× 14 650
Pere Fábregas Spain 10 145 0.7× 208 1.4× 57 0.4× 66 0.8× 212 2.6× 19 462
Shima Uneda Japan 11 100 0.5× 124 0.8× 30 0.2× 50 0.6× 264 3.2× 17 495

Countries citing papers authored by KT Preissner

Since Specialization
Citations

This map shows the geographic impact of KT Preissner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by KT Preissner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites KT Preissner more than expected).

Fields of papers citing papers by KT Preissner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by KT Preissner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by KT Preissner. The network helps show where KT Preissner may publish in the future.

Co-authorship network of co-authors of KT Preissner

This figure shows the co-authorship network connecting the top 25 collaborators of KT Preissner. A scholar is included among the top collaborators of KT Preissner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with KT Preissner. KT Preissner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Wygrecka, Małgorzata, Ewa Jabłońska, Ingrid Henneke, et al.. (2015). Coagulation factor XII mediates fibrotic response to lung injury. Pneumologie. 69(5). 2 indexed citations
2.
Mangold, Andreas, Thomas Scherz, Sherin Alias, et al.. (2014). P726Coronary NET burden and DNase activity in ST-elevation acute coronary syndrome are predictors of infarct size. Cardiovascular Research. 103(suppl 1). S133.1–S133. 1 indexed citations
3.
Wygrecka, Małgorzata, Djuro Kosanovic, Frank Petersen, et al.. (2013). Mast cells and fibroblasts work in concert to aggravate pulmonary fibrosis: role of transmembrane stem cell factor (SCF) and PAR-2/PKCalpha/Raf-1/p44/42 signaling pathway. Pneumologie. 67(S 01). 4 indexed citations
4.
Kwapiszewska, Grażyna, Philipp Markart, Ralph T. Schermuly, et al.. (2009). Potential Role of Protease-Activated Receptor-2 in Pathogenesis of Pulmonary Hypertension.. A1862–A1862. 1 indexed citations
5.
Brownlee, Michael, A. Jonczyk, Arne Sutter, KT Preissner, & HP Hammes. (1996). Subcutaneous injection of a cyclic peptide antagonist of vitronectin receptor-type integrins inhibits retinal neovascularization. American Journal of Ophthalmology. 122(3). 464–465. 9 indexed citations
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Zanetti, Adriana, G Conforti, Sibylle Hess, et al.. (1994). Clustering of vitronectin and RGD peptides on microspheres leads to engagement of integrins on the luminal aspect of endothelial cell membrane. Blood. 84(4). 1116–1123. 3 indexed citations
8.
Zanetti, Adriana, G Conforti, Sibylle Hess, et al.. (1994). Clustering of vitronectin and RGD peptides on microspheres leads to engagement of integrins on the luminal aspect of endothelial cell membrane. Blood. 84(4). 1116–1123. 55 indexed citations
9.
Tschopp, J, et al.. (1993). Human megakaryocytes express clusterin and package it without apolipoprotein A-1 into alpha-granules. Blood. 82(1). 118–125. 39 indexed citations
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Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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