Kenrick A. Waite

2.7k total citations
9 papers, 143 citations indexed

About

Kenrick A. Waite is a scholar working on Molecular Biology, Epidemiology and Cell Biology. According to data from OpenAlex, Kenrick A. Waite has authored 9 papers receiving a total of 143 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 7 papers in Epidemiology and 7 papers in Cell Biology. Recurrent topics in Kenrick A. Waite's work include Ubiquitin and proteasome pathways (9 papers), Autophagy in Disease and Therapy (7 papers) and Endoplasmic Reticulum Stress and Disease (7 papers). Kenrick A. Waite is often cited by papers focused on Ubiquitin and proteasome pathways (9 papers), Autophagy in Disease and Therapy (7 papers) and Endoplasmic Reticulum Stress and Disease (7 papers). Kenrick A. Waite collaborates with scholars based in United States. Kenrick A. Waite's co-authors include Jeroen Roelofs, Soyeon Park, Stella Y. Lee and Alina De La Mota‐Peynado and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and SHILAP Revista de lepidopterología.

In The Last Decade

Kenrick A. Waite

9 papers receiving 142 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Kenrick A. Waite United States	5	117	90	82	11	8	9	143
Matías González-Quiroz Chile	4	57 0.5×	23 0.3×	44 0.5×	5 0.5×	7 0.9×	4	96
Di-Ao Liu United States	3	74 0.6×	29 0.3×	18 0.2×	3 0.3×	9 1.1×	4	97
Kevin Leiss Germany	4	74 0.6×	38 0.4×	18 0.2×	3 0.3×	7 0.9×	7	100
Shree Padma Metur United States	7	59 0.5×	62 0.7×	25 0.3×		9 1.1×	9	124
Han-Yi Huang Taiwan	3	209 1.8×	49 0.5×	47 0.6×	24 2.2×	31 3.9×	5	260
Ze Cheng United States	5	195 1.7×	14 0.2×	21 0.3×	16 1.5×	12 1.5×	5	223
Palak Sekhri United States	6	44 0.4×	31 0.3×	19 0.2×	6 0.5×	14 1.8×	9	104
Franziska Ottens Germany	6	151 1.3×	11 0.1×	25 0.3×	10 0.9×	9 1.1×	7	196
Svenja Ulferts Germany	7	103 0.9×	12 0.1×	40 0.5×	3 0.3×	8 1.0×	8	141
Mara Sannai Germany	5	67 0.6×	9 0.1×	59 0.7×	10 0.9×	13 1.6×	5	109

Countries citing papers authored by Kenrick A. Waite

Since Specialization

Citations

This map shows the geographic impact of Kenrick A. Waite's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenrick A. Waite with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenrick A. Waite more than expected).

Fields of papers citing papers by Kenrick A. Waite

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kenrick A. Waite. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenrick A. Waite. The network helps show where Kenrick A. Waite may publish in the future.

Co-authorship network of co-authors of Kenrick A. Waite

This figure shows the co-authorship network connecting the top 25 collaborators of Kenrick A. Waite. A scholar is included among the top collaborators of Kenrick A. Waite based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenrick A. Waite. Kenrick A. Waite is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Waite, Kenrick A., et al.. (2024). Proteasome condensate formation is driven by multivalent interactions with shuttle factors and ubiquitin chains. Proceedings of the National Academy of Sciences. 121(10). e2310756121–e2310756121. 6 indexed citations

2.

Waite, Kenrick A. & Jeroen Roelofs. (2022). Proteasome granule formation is regulated through mitochondrial respiration and kinase signaling. Journal of Cell Science. 135(17). 5 indexed citations

3.

Waite, Kenrick A. & Jeroen Roelofs. (2022). Proteaphagy and the Trafficking of Proteasomes under Nutrient Stress Conditions. SHILAP Revista de lepidopterología. 1(1). 21–24. 1 indexed citations

4.

Waite, Kenrick A., et al.. (2021). Proteaphagy is specifically regulated and requires factors dispensable for general autophagy. Journal of Biological Chemistry. 298(1). 101494–101494. 21 indexed citations

5.

Waite, Kenrick A., et al.. (2021). Proteasome activator Blm10 levels and autophagic degradation directly impact the proteasome landscape. Journal of Biological Chemistry. 296. 100468–100468. 4 indexed citations

6.

Waite, Kenrick A., et al.. (2020). Tagging the proteasome active site β5 causes tag specific phenotypes in yeast. Scientific Reports. 10(1). 18133–18133. 4 indexed citations

7.

Roelofs, Jeroen, et al.. (2018). Native Gel Approaches in Studying Proteasome Assembly and Chaperones. Methods in molecular biology. 1844. 237–260. 14 indexed citations

8.

Roelofs, Jeroen, et al.. (2017). Nitrogen Starvation and Rapamycin both Induce Autophagic Degradation of Proteasome Complexes. The FASEB Journal. 31(S1). 1 indexed citations

9.

Waite, Kenrick A., et al.. (2015). Starvation Induces Proteasome Autophagy with Different Pathways for Core and Regulatory Particles. Journal of Biological Chemistry. 291(7). 3239–3253. 87 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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