Kenji Jinnai

1.8k total citations
65 papers, 1.0k citations indexed

About

Kenji Jinnai is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Kenji Jinnai has authored 65 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Cellular and Molecular Neuroscience, 31 papers in Molecular Biology and 23 papers in Neurology. Recurrent topics in Kenji Jinnai's work include Genetic Neurodegenerative Diseases (23 papers), Mitochondrial Function and Pathology (17 papers) and Parkinson's Disease Mechanisms and Treatments (10 papers). Kenji Jinnai is often cited by papers focused on Genetic Neurodegenerative Diseases (23 papers), Mitochondrial Function and Pathology (17 papers) and Parkinson's Disease Mechanisms and Treatments (10 papers). Kenji Jinnai collaborates with scholars based in Japan, United States and Taiwan. Kenji Jinnai's co-authors include Fumio Kanda, Keiichi Takahashi, S. Ono, Naonobu Futamura, Kōichi Nagao, Itaru Funakawa, Shigehisa Mitake, Yoshihiro Fukuoka, Hiroshi Kurisaki and Takuo Fujita and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Kenji Jinnai

64 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kenji Jinnai Japan 19 507 487 394 170 128 65 1.0k
Hideji Hashida Japan 19 672 1.3× 520 1.1× 434 1.1× 111 0.7× 76 0.6× 49 1.2k
K. V. Toyka Germany 15 355 0.7× 646 1.3× 575 1.5× 206 1.2× 88 0.7× 29 1.3k
Alessandro Filla Italy 24 949 1.9× 1.0k 2.1× 498 1.3× 191 1.1× 86 0.7× 45 1.6k
Arifumi Kosakai Japan 17 422 0.8× 329 0.7× 139 0.4× 258 1.5× 85 0.7× 25 1.0k
Sadayuki Matsumoto Japan 20 244 0.5× 392 0.8× 761 1.9× 173 1.0× 126 1.0× 53 1.1k
Pedro Mancías United States 15 750 1.5× 809 1.7× 380 1.0× 280 1.6× 86 0.7× 37 1.7k
Takao Omura Japan 19 363 0.7× 595 1.2× 174 0.4× 82 0.5× 190 1.5× 33 1.2k
Dominic B. Fee United States 13 391 0.8× 294 0.6× 203 0.5× 170 1.0× 33 0.3× 23 801
Ettore Salsano Italy 22 638 1.3× 426 0.9× 535 1.4× 322 1.9× 187 1.5× 72 1.6k
Hillary Lipe United States 24 593 1.2× 945 1.9× 600 1.5× 210 1.2× 65 0.5× 40 1.5k

Countries citing papers authored by Kenji Jinnai

Since Specialization
Citations

This map shows the geographic impact of Kenji Jinnai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kenji Jinnai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kenji Jinnai more than expected).

Fields of papers citing papers by Kenji Jinnai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kenji Jinnai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kenji Jinnai. The network helps show where Kenji Jinnai may publish in the future.

Co-authorship network of co-authors of Kenji Jinnai

This figure shows the co-authorship network connecting the top 25 collaborators of Kenji Jinnai. A scholar is included among the top collaborators of Kenji Jinnai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kenji Jinnai. Kenji Jinnai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Lee, Kuang‐Yung, Masayuki Nakamori, Masanori Takahashi, et al.. (2012). Muscleblind-Like 1 Knockout Mice Reveal Novel Splicing Defects in the Myotonic Dystrophy Brain. PLoS ONE. 7(3). e33218–e33218. 66 indexed citations
3.
Ono, S., Takashi Imai, Ken Takahashi, et al.. (2009). Increased type III procollagen in serum and skin of patients with amyotrophic lateral sclerosis. Acta Neurologica Scandinavica. 100(6). 377–384. 3 indexed citations
4.
Futamura, Naonobu, et al.. (2006). [Four siblings with becker muscular dystrophy (BMD) manifesting severe mental retardation].. PubMed. 46(1). 62–5. 5 indexed citations
5.
Funakawa, Itaru, et al.. (2005). [A case of esophageal achalasia followed by Parkinson's disease].. PubMed. 45(8). 607–9. 1 indexed citations
6.
Ohyanagi, Mitsumasa, et al.. (2004). The relationship between clinical stage, prognosis and myocardial damage in patients with Duchenne-type muscular dystrophy: five-year follow-up study. Annals of Nuclear Medicine. 18(3). 203–208. 10 indexed citations
7.
Jinnai, Kenji & Yoshitake Hayashi. (2001). Hemorrhage in the oculomotor nerve as a complication of leukemia. Neuropathology. 21(3). 241–244. 5 indexed citations
8.
Ono, Seiitsu, Takashi Imai, Keiichi Takahashi, et al.. (2000). Increased serum hyaluronic acid in amyotrophic lateral sclerosis: Relation to its skin content. Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders. 1(3). 213–218. 10 indexed citations
9.
Ohnishi, Akio, et al.. (2000). Dejerine-Sottas disease with a novel de novo dominant mutation, Ser 149 Arg, of the peripheral myelin protein 22. Acta Neuropathologica. 99(3). 327–330. 8 indexed citations
10.
Ono, Seiitsu, Takashi Imai, Keiichi Takahashi, et al.. (1999). Alteration in amino acids in motor neurons of the spinal cord in amyotrophic lateral sclerosis. Journal of the Neurological Sciences. 167(2). 121–126. 10 indexed citations
11.
Itoh, Kyoko, et al.. (1999). Multifocal glial nodules in a case of Duchenne muscular dystrophy with severe mental retardation. Neuropathology. 19(3). 322–327. 9 indexed citations
12.
Jinnai, Kenji, et al.. (1999). Elongation of (CTG)n repeats in myotonic dystrophy protein kinase gene in tumors associated with myotonic dystrophy patients. Muscle & Nerve. 22(9). 1271–1274. 18 indexed citations
13.
Ono, S., Fumio Kanda, Keiichi Takahashi, et al.. (1996). Neuronal loss in the medullary reticular formation in myotonic dystrophy. Neurology. 46(1). 228–231. 33 indexed citations
14.
Fukuoka, Yoshihiro, et al.. (1995). [A case of hereditary pressure-sensitive neuropathy, confirmed by a gene analysis].. PubMed. 35(6). 657–60. 3 indexed citations
15.
Ono, S., Fumio Kanda, Keiichi Takahashi, et al.. (1995). Neuronal cell loss in the dorsal raphe nucleus and the superior central nucleus in myotonic dystrophy: a clinicopathological correlation. Acta Neuropathologica. 89(2). 122–125. 11 indexed citations
16.
Jinnai, Kenji, Tetsuo Ashizawa, & M. Zouhair Atassi. (1994). Analysis of exposed regions on the main extracellular domain of mouse acetylcholine receptor α subunit in live muscle cells by binding profiles of antipeptide antibodies. Journal of Protein Chemistry. 13(8). 715–722. 4 indexed citations
17.
Atassi, M. Zouhair, et al.. (1992). Epitope-specific suppression of antibody response in experimental autoimmune myasthenia gravis by a monomethoxypolyethylene glycol conjugate of a myasthenogenic synthetic peptide.. Proceedings of the National Academy of Sciences. 89(13). 5852–5856. 37 indexed citations
18.
Ono, S., K Inoue, T Mannen, et al.. (1989). Intracytoplasmic inclusion bodies of the thalamus and the substantia nigra, and Marinesco bodies in myotonic dystrophy: a quantitative morphological study. Acta Neuropathologica. 77(4). 350–356. 32 indexed citations
19.
Jinnai, Kenji, Kazuo Chihara, Fumio Kanda, Kazuo Tada, & Toshiro Fujita. (1989). Calcitonin gene-related peptide enhances spontaneous acetylcholine release from the rat motor nerve terminal. Neuroscience Letters. 103(1). 64–68. 22 indexed citations
20.
Kanda, Fumio, Koji Inoue, Kenji Jinnai, Kanako Takahashi, & T Mannen. (1989). [A case of adrenomyeloneuropathy with localized cerebral white matter degeneration].. PubMed. 29(4). 483–7. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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