Keni Ni

483 total citations
11 papers, 377 citations indexed

About

Keni Ni is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Physiology. According to data from OpenAlex, Keni Ni has authored 11 papers receiving a total of 377 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Cellular and Molecular Neuroscience, 4 papers in Molecular Biology and 4 papers in Physiology. Recurrent topics in Keni Ni's work include Neuroscience and Neuropharmacology Research (4 papers), Alzheimer's disease research and treatments (3 papers) and Ion channel regulation and function (2 papers). Keni Ni is often cited by papers focused on Neuroscience and Neuropharmacology Research (4 papers), Alzheimer's disease research and treatments (3 papers) and Ion channel regulation and function (2 papers). Keni Ni collaborates with scholars based in Japan, Italy and United States. Keni Ni's co-authors include Nobuya Matsuoka, Junko Yarimizu, Yasuyuki Mitani, Yoshitsugu Shitaka, Hiroki Akashiba, Hiroshi Uchino, Katsuya Harada, Mayako Yamazaki, Shinji Takahashi and Hiroshi Yamada and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Keni Ni

11 papers receiving 368 citations

Peers

Keni Ni
Kaichuan Zhu Germany
Suzan Aschmies United States
Sarah Gourmaud United States
Pascal Kurosinski Switzerland
Niloufar Haque United States
Ross Jeggo United Kingdom
Krishna Bharani United States
Keni Ni
Citations per year, relative to Keni Ni Keni Ni (= 1×) peers David Demedts

Countries citing papers authored by Keni Ni

Since Specialization
Citations

This map shows the geographic impact of Keni Ni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keni Ni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keni Ni more than expected).

Fields of papers citing papers by Keni Ni

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keni Ni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keni Ni. The network helps show where Keni Ni may publish in the future.

Co-authorship network of co-authors of Keni Ni

This figure shows the co-authorship network connecting the top 25 collaborators of Keni Ni. A scholar is included among the top collaborators of Keni Ni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keni Ni. Keni Ni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Takahashi, Shinji, Ai Okamura, Mayako Yamazaki, & Keni Ni. (2019). ASP2905, a specific inhibitor of the potassium channel Kv12.2 encoded by the Kcnh3 gene, is psychoactive in mice. Behavioural Brain Research. 378. 112315–112315. 2 indexed citations
2.
Takahashi, Shinji, et al.. (2018). The KCNH3 inhibitor ASP2905 shows potential in the treatment of attention deficit/hyperactivity disorder. PLoS ONE. 13(11). e0207750–e0207750. 11 indexed citations
3.
Takahashi, Shinji, Kohei Inamura, Junko Yarimizu, et al.. (2017). Neurochemical and neuropharmacological characterization of ASP2905, a novel potent selective inhibitor of the potassium channel KCNH3. European Journal of Pharmacology. 810. 26–35. 10 indexed citations
4.
Yamazaki, Mayako, Katsuya Harada, Noriyuki Yamamoto, et al.. (2014). ASP5736, a novel 5-HT5A receptor antagonist, ameliorates positive symptoms and cognitive impairment in animal models of schizophrenia. European Neuropsychopharmacology. 24(10). 1698–1708. 31 indexed citations
5.
Mitani, Yasuyuki, Hiroki Akashiba, Junko Yarimizu, et al.. (2013). Pharmacological characterization of the novel γ-secretase modulator AS2715348, a potential therapy for Alzheimer's disease, in rodents and nonhuman primates. Neuropharmacology. 79. 412–419. 5 indexed citations
6.
Toritsuka, Michihiro, Sohei Kimoto, Melissa A. Landek‐Salgado, et al.. (2013). Deficits in microRNA-mediated Cxcr4/Cxcl12 signaling in neurodevelopmental deficits in a 22q11 deletion syndrome mouse model. Proceedings of the National Academy of Sciences. 110(43). 17552–17557. 63 indexed citations
7.
Mitani, Yasuyuki, Junko Yarimizu, Hiroshi Uchino, et al.. (2012). Differential Effects between γ-Secretase Inhibitors and Modulators on Cognitive Function in Amyloid Precursor Protein-Transgenic and Nontransgenic Mice. Journal of Neuroscience. 32(6). 2037–2050. 149 indexed citations
9.
Mitani, Yasuyuki, Junko Yarimizu, Hiroki Akashiba, et al.. (2012). Amelioration of cognitive deficits in plaque‐bearing Alzheimer's disease model mice through selective reduction of nascent soluble A42 without affecting other A pools. Journal of Neurochemistry. 125(3). 465–472. 12 indexed citations
10.
Yamada, Hiroshi, et al.. (2011). Chronic treatment with olanzapine via a novel infusion pump induces adiposity in male rats. Life Sciences. 88(17-18). 761–765. 18 indexed citations
11.
Yamada, Hiroshi, Takuma Mihara, Yuji Kondo, et al.. (2010). Two models for weight gain and hyperphagia as side effects of atypical antipsychotics in male rats: Validation with olanzapine and ziprasidone. Behavioural Brain Research. 216(2). 561–568. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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