Keiichi Ebina

765 total citations
55 papers, 617 citations indexed

About

Keiichi Ebina is a scholar working on Molecular Biology, Immunology and Biochemistry. According to data from OpenAlex, Keiichi Ebina has authored 55 papers receiving a total of 617 indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 21 papers in Immunology and 13 papers in Biochemistry. Recurrent topics in Keiichi Ebina's work include Antioxidant Activity and Oxidative Stress (13 papers), Atherosclerosis and Cardiovascular Diseases (11 papers) and Antifungal resistance and susceptibility (7 papers). Keiichi Ebina is often cited by papers focused on Antioxidant Activity and Oxidative Stress (13 papers), Atherosclerosis and Cardiovascular Diseases (11 papers) and Antifungal resistance and susceptibility (7 papers). Keiichi Ebina collaborates with scholars based in Japan. Keiichi Ebina's co-authors include Katsushi Yokota, Takeshi Kumagai, Akira Sato, Hisatake Kondo, Hiroyuki Sakagami, Osamu Sakaguchi, Junken Aoki, Takayuki Nagata, Kiyoshi Nagata and Hikaru Yamanaka and has published in prestigious journals such as European Journal of Pharmacology, Biochimica et Biophysica Acta (BBA) - General Subjects and Peptides.

In The Last Decade

Keiichi Ebina

54 papers receiving 614 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Keiichi Ebina Japan 15 286 175 102 89 75 55 617
Dan Hao China 14 475 1.7× 100 0.6× 33 0.3× 34 0.4× 103 1.4× 58 1.1k
J. Rapacz United States 21 452 1.6× 193 1.1× 145 1.4× 53 0.6× 91 1.2× 52 1.3k
A. C. Looman Germany 11 405 1.4× 231 1.3× 42 0.4× 28 0.3× 70 0.9× 17 768
Judith Hasler‐Rapacz United States 17 298 1.0× 135 0.8× 60 0.6× 39 0.4× 71 0.9× 33 859
John V. Stokes United States 18 142 0.5× 156 0.9× 17 0.2× 39 0.4× 88 1.2× 46 744
Daniel W. Siemsen United States 14 341 1.2× 286 1.6× 13 0.1× 78 0.9× 49 0.7× 27 781
Haitao Hu China 22 520 1.8× 227 1.3× 47 0.5× 346 3.9× 140 1.9× 88 1.2k
Shu‐Jun Chiu Taiwan 13 249 0.9× 108 0.6× 18 0.2× 36 0.4× 81 1.1× 13 512
Julia Sehm Germany 7 341 1.2× 50 0.3× 46 0.5× 51 0.6× 29 0.4× 7 652

Countries citing papers authored by Keiichi Ebina

Since Specialization
Citations

This map shows the geographic impact of Keiichi Ebina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Keiichi Ebina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Keiichi Ebina more than expected).

Fields of papers citing papers by Keiichi Ebina

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Keiichi Ebina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Keiichi Ebina. The network helps show where Keiichi Ebina may publish in the future.

Co-authorship network of co-authors of Keiichi Ebina

This figure shows the co-authorship network connecting the top 25 collaborators of Keiichi Ebina. A scholar is included among the top collaborators of Keiichi Ebina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Keiichi Ebina. Keiichi Ebina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Uwamizu, Akiharu, Daisuke Uta, Junken Aoki, et al.. (2024). Biotinylated Heptapeptides with D-amino Acids Suppress Allergic Reactions by Inhibiting Mast Cell Activation and Antagonizing the Histamine Receptor. International Journal of Peptide Research and Therapeutics. 31(1).
2.
Sato, Akira, et al.. (2022). Estrogen Sulfotransferase is Highly Expressed in Vascular Endothelial Cells Overlying Atherosclerotic Plaques. The Protein Journal. 41(1). 179–188. 3 indexed citations
3.
Sato, Akira, et al.. (2021). Royal Jelly Proteins Inhibit Macrophage Proliferation: Interactions with Native- and Oxidized-Low Density Lipoprotein. The Protein Journal. 40(5). 699–708. 7 indexed citations
4.
Sato, Akira, et al.. (2021). Interaction of Native- and Oxidized-Low-Density Lipoprotein with Human Estrogen Sulfotransferase. The Protein Journal. 40(2). 192–204. 3 indexed citations
5.
Sato, Akira, et al.. (2018). The interaction of β2-glycoprotein I with lysophosphatidic acid in platelet aggregation and blood clotting. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1866(12). 1232–1241. 4 indexed citations
6.
Sato, Akira, et al.. (2017). Angiotensin II induces the aggregation of native and oxidized low-density lipoprotein. European Biophysics Journal. 47(1). 1–9. 8 indexed citations
8.
Sato, Akira, et al.. (2015). Biotinylated heptapeptides substituted with a d-amino acid as platelet-activating factor inhibitors. European Journal of Pharmacology. 764. 202–207. 5 indexed citations
9.
Sato, Akira, et al.. (2015). Angiotensin peptides attenuate platelet-activating factor-induced inflammatory activity in rats. Peptides. 73. 60–66. 4 indexed citations
10.
Sato, Akira & Keiichi Ebina. (2013). Common mechanism in endothelin-3 and PAF receptor function for anti-inflammatory responses. European Journal of Pharmacology. 718(1-3). 30–33. 8 indexed citations
11.
Sato, Akira, et al.. (2013). An endothelin-3-related synthetic biotinylated pentapeptide as a novel inhibitor of platelet-activating factor. European Journal of Pharmacology. 714(1-3). 142–147. 6 indexed citations
12.
Sato, Akira, Junken Aoki, & Keiichi Ebina. (2012). Synthetic Biotinylated Peptide Compound, BP21, Specifically Recognizes Lysophosphatidylcholine Micelles. Chemical Biology & Drug Design. 80(3). 417–425. 12 indexed citations
13.
Kumagai, Takeshi, et al.. (2001). Oxidized low-density lipoprotein-binding specificity of Asp-hemolysin from Aspergillus fumigatus. Biochimica et Biophysica Acta (BBA) - General Subjects. 1568(3). 183–188. 30 indexed citations
14.
Kumagai, Takeshi, et al.. (1999). Cytotoxic Activity and Cytokine Gene Induction of Asp-hemolysin to Murine Macrophages.. Nippon Ishinkin Gakkai Zasshi. 40(4). 217–222. 27 indexed citations
15.
Kumagai, Takeshi, et al.. (1999). Binding of Asp-hemolysin from Aspergillus fumigatus to Oxidized Low Density Lipoprotein.. Biological and Pharmaceutical Bulletin. 22(5). 549–550. 18 indexed citations
16.
Kumagai, Takeshi, et al.. (1998). Oxidized low density lipoprotein inhibits the hemolytic activity of Asp-hemolysin fromAspergillus fumigatus. FEMS Microbiology Letters. 167(2). 275–280. 21 indexed citations
17.
Kumagai, Takeshi, et al.. (1996). Apolipoprotein B Inhibits the Hemolytic Activity of Asp-Hemolysin from Aspergillus fumigatus.. Biological and Pharmaceutical Bulletin. 19(4). 547–550. 17 indexed citations
18.
Sakagami, Hiroyuki, Keiichi Ebina, & Hisatake Kondo. (1994). Re-examination of the ontogeny in the gene expression of DARPP-32 in the rat brain. Molecular Brain Research. 25(1-2). 67–72. 6 indexed citations
19.
Ebina, Keiichi, Katsushi Yokota, & Osamu Sakaguchi. (1983). :XVI. Biological Properties of Asp-hemolysin as a Parasite Factor. Nippon Ishinkin Gakkai Zasshi. 24(3). 245–252. 1 indexed citations
20.
Ebina, Keiichi, Katsushi Yokota, & Osamu Sakaguchi. (1983). . Japanese Journal of Medical Mycology. 24(3). 245–252. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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