Ke-Fei Shen

1.9k total citations
23 papers, 1.6k citations indexed

About

Ke-Fei Shen is a scholar working on Cellular and Molecular Neuroscience, Physiology and Molecular Biology. According to data from OpenAlex, Ke-Fei Shen has authored 23 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Cellular and Molecular Neuroscience, 19 papers in Physiology and 18 papers in Molecular Biology. Recurrent topics in Ke-Fei Shen's work include Neuropeptides and Animal Physiology (20 papers), Pain Mechanisms and Treatments (19 papers) and Receptor Mechanisms and Signaling (10 papers). Ke-Fei Shen is often cited by papers focused on Neuropeptides and Animal Physiology (20 papers), Pain Mechanisms and Treatments (19 papers) and Receptor Mechanisms and Signaling (10 papers). Ke-Fei Shen collaborates with scholars based in United States. Ke-Fei Shen's co-authors include Stanley M. Crain, P.A. Schwartzkroin, Guoguang Chen, Alcmène Chalazonitis and Robert W. Ledeen and has published in prestigious journals such as Brain Research, Pain and Annals of the New York Academy of Sciences.

In The Last Decade

Ke-Fei Shen

23 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ke-Fei Shen United States	19	1.2k	921	808	206	164	23	1.6k
Mingting Tian United States	7	1.0k 0.9×	504 0.5×	756 0.9×	344 1.7×	126 0.8×	9	1.6k
Kim Fisher Canada	17	605 0.5×	887 1.0×	394 0.5×	296 1.4×	138 0.8×	21	1.4k
Wendy Walwyn United States	26	1.3k 1.1×	789 0.9×	985 1.2×	121 0.6×	73 0.4×	42	1.9k
Jan Hendrich United Kingdom	8	562 0.5×	620 0.7×	688 0.9×	64 0.3×	93 0.6×	8	1.3k
W.A. Prado Brazil	18	345 0.3×	527 0.6×	293 0.4×	171 0.8×	175 1.1×	48	1.1k
A. Ableitner Germany	10	656 0.6×	402 0.4×	366 0.5×	56 0.3×	85 0.5×	14	918
Shaun Gorman United Kingdom	5	401 0.3×	582 0.6×	696 0.9×	45 0.2×	68 0.4×	7	1.1k
Wojciech Margas United States	14	657 0.6×	311 0.3×	552 0.7×	68 0.3×	43 0.3×	33	1.0k
Margit Szikszay Hungary	16	460 0.4×	391 0.4×	348 0.4×	105 0.5×	102 0.6×	39	811
J.W. Villiger New Zealand	18	500 0.4×	221 0.2×	328 0.4×	106 0.5×	92 0.6×	33	847

Countries citing papers authored by Ke-Fei Shen

Since Specialization

Citations

This map shows the geographic impact of Ke-Fei Shen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ke-Fei Shen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ke-Fei Shen more than expected).

Fields of papers citing papers by Ke-Fei Shen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ke-Fei Shen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ke-Fei Shen. The network helps show where Ke-Fei Shen may publish in the future.

Co-authorship network of co-authors of Ke-Fei Shen

This figure shows the co-authorship network connecting the top 25 collaborators of Ke-Fei Shen. A scholar is included among the top collaborators of Ke-Fei Shen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ke-Fei Shen. Ke-Fei Shen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Crain, Stanley M. & Ke-Fei Shen. (2008). Low doses of cyclic AMP-phosphodiesterase inhibitors rapidly evoke opioid receptor-mediated thermal hyperalgesia in naïve mice which is converted to prominent analgesia by cotreatment with ultra-low-dose naltrexone. Brain Research. 1231. 16–24. 10 indexed citations

Crain, Stanley M. & Ke-Fei Shen. (2007). Naloxone rapidly evokes endogenous kappa opioid receptor-mediated hyperalgesia in naïve mice pretreated briefly with GM1 ganglioside or in chronic morphine-dependent mice. Brain Research. 1167. 31–41. 14 indexed citations

Crain, Stanley M. & Ke-Fei Shen. (2003). Neuraminidase inhibitor, oseltamivir blocks GM1 ganglioside-regulated excitatory opioid receptor-mediated hyperalgesia, enhances opioid analgesia and attenuates tolerance in mice. Brain Research. 995(2). 260–266. 47 indexed citations

Crain, Stanley M. & Ke-Fei Shen. (2001). Acute thermal hyperalgesia elicited by low-dose morphine in normal mice is blocked by ultra-low-dose naltrexone, unmasking potent opioid analgesia. Brain Research. 888(1). 75–82. 130 indexed citations

Shen, Ke-Fei & Stanley M. Crain. (2001). Cholera toxin-B subunit blocks excitatory opioid receptor-mediated hyperalgesic effects in mice, thereby unmasking potent opioid analgesia and attenuating opioid tolerance/dependence. Brain Research. 919(1). 20–30. 42 indexed citations

Crain, Stanley M. & Ke-Fei Shen. (2000). Antagonists of excitatory opioid receptor functions enhance morphine's analgesic potency and attenuate opioid tolerance/dependence liability. Pain. 84(2). 121–131. 243 indexed citations

Crain, Stanley M. & Ke-Fei Shen. (2000). Enhanced analgesic potency and reduced tolerance of morphine in 129/SvEv mice: evidence for a deficiency in GM1 ganglioside-regulated excitatory opioid receptor functions. Brain Research. 856(1-2). 227–235. 43 indexed citations

Crain, Stanley M. & Ke-Fei Shen. (1998). GM1 Ganglioside‐induced Modulation of Opioid Receptor‐mediated Functions. Annals of the New York Academy of Sciences. 845(1). 106–125. 40 indexed citations

Shen, Ke-Fei & Stanley M. Crain. (1997). Ultra-low doses of naltrexone or etorphine increase morphine's antinociceptive potency and attenuate tolerance/dependence in mice. Brain Research. 757(2). 176–190. 104 indexed citations

10.

Crain, Stanley M. & Ke-Fei Shen. (1996). Etorphine elicits anomalous excitatory opioid effects on sensory neurons treated with GM1 ganglioside or pertussis toxin in contrast to its potent inhibitory effects on naive or chronic morphine-treated cells. Brain Research. 741(1-2). 275–283. 7 indexed citations

11.

Crain, Stanley M. & Ke-Fei Shen. (1996). Modulatory effects of Gs-coupled excitatory opioid receptor functions on opioid analgesia, tolerance, and dependence. Neurochemical Research. 21(11). 1347–1351. 65 indexed citations

12.

Shen, Ke-Fei & Stanley M. Crain. (1995). Biphalin, an enkephalin analog with unexpectedly high antinociceptive potency and low dependence liability in vivo, selectively antagonizes excitatory opioid receptor functions of sensory neurons in culture. Brain Research. 701(1-2). 158–166. 24 indexed citations

13.

Shen, Ke-Fei & Stanley M. Crain. (1995). Specific N- or C-terminus modified dynorphin and β-endorphin peptides can selectively block excitatory opioid receptor functions in sensory neurons and unmask potent inhibitory effects of opioid agonists. Brain Research. 673(1). 30–38. 16 indexed citations

14.

Shen, Ke-Fei & Stanley M. Crain. (1994). Antagonists at excitatory opioid receptors on sensory neurons in culture increase potency and specificity of opiate analgesics and attenuate development of tolerance/dependence. Brain Research. 636(2). 286–297. 67 indexed citations

15.

Shen, Ke-Fei & Stanley M. Crain. (1992). Chronic selective activation of excitatory opioid receptor functions in sensory neurons results in opioid ‘dependence’ without tolerance. Brain Research. 597(1). 74–83. 47 indexed citations

16.

Shen, Ke-Fei, Stanley M. Crain, & Robert W. Ledeen. (1991). Brief treatment of sensory ganglion neurons with GM1 ganglioside enhances the efficacy of opioid excitatory effects on the action potential. Brain Research. 559(1). 130–138. 36 indexed citations

17.

Crain, Stanley M. & Ke-Fei Shen. (1990). Opioids can evoke direct receptor-mediated excitatory effects on sensory neurons. Trends in Pharmacological Sciences. 11(2). 77–81. 292 indexed citations

18.

Shen, Ke-Fei & Stanley M. Crain. (1990). Cholera toxin-B subunit blocks excitatory effects of opioids on sensory neuron action potentials indicating that GM1 ganglioside may regulate Gs-linked opioid receptor functions. Brain Research. 531(1-2). 1–7. 62 indexed citations

19.

Shen, Ke-Fei & P.A. Schwartzkroin. (1988). Effects of temperature alterations on population and cellular activities in hippocampal slices from mature and immature rabbit. Brain Research. 475(2). 305–316. 72 indexed citations

20.

Chen, Guoguang, Alcmène Chalazonitis, Ke-Fei Shen, & Stanley M. Crain. (1988). Inhibitor of cyclic AMP-dependent protein kinase blocks opioid-induced prolongation of the action potential of mouse sensory ganglion neurons in dissociated cell cultures. Brain Research. 462(2). 372–377. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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