Kazuo Todoki

574 total citations
30 papers, 485 citations indexed

About

Kazuo Todoki is a scholar working on Physiology, Surgery and Molecular Biology. According to data from OpenAlex, Kazuo Todoki has authored 30 papers receiving a total of 485 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Physiology, 7 papers in Surgery and 5 papers in Molecular Biology. Recurrent topics in Kazuo Todoki's work include Nitric Oxide and Endothelin Effects (9 papers), Neuropeptides and Animal Physiology (5 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (4 papers). Kazuo Todoki is often cited by papers focused on Nitric Oxide and Endothelin Effects (9 papers), Neuropeptides and Animal Physiology (5 papers) and Cardiovascular, Neuropeptides, and Oxidative Stress Research (4 papers). Kazuo Todoki collaborates with scholars based in Japan and United States. Kazuo Todoki's co-authors include Eiichiro Okabe, Fumihiko Yoshino, Haruo Itô, Shunsuke Takahashi, Yuko Nakamura, Hitoshi Matsumoto, Masaichi‐Chang‐il Lee, Kyo Kobayashi, Hirofumi Shoji and Hiroe Ito and has published in prestigious journals such as Journal of Pharmacology and Experimental Therapeutics, Anesthesiology and Journal of Dental Research.

In The Last Decade

Kazuo Todoki

28 papers receiving 468 citations

Peers

Kazuo Todoki
Cristina Antoniali Brazil
César Tadeu Spadella Brazil
Graf H Germany
Antonia Patruno Italy
Lihui Tang China
Yeşim Ünlüçerçi Türkiye
Won-Jae Kim South Korea
Yuko Imai Japan
Chuan Cai China
Cristina Gomes de Macedo Brazil
Cristina Antoniali Brazil
Kazuo Todoki
Citations per year, relative to Kazuo Todoki Kazuo Todoki (= 1×) peers Cristina Antoniali

Countries citing papers authored by Kazuo Todoki

Since Specialization
Citations

This map shows the geographic impact of Kazuo Todoki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kazuo Todoki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kazuo Todoki more than expected).

Fields of papers citing papers by Kazuo Todoki

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kazuo Todoki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kazuo Todoki. The network helps show where Kazuo Todoki may publish in the future.

Co-authorship network of co-authors of Kazuo Todoki

This figure shows the co-authorship network connecting the top 25 collaborators of Kazuo Todoki. A scholar is included among the top collaborators of Kazuo Todoki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kazuo Todoki. Kazuo Todoki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Haishima, Yuji, Chie Hasegawa, Kazuo Todoki, et al.. (2016). A biological study establishing the endotoxin limit of biomaterials for bone regeneration in cranial and femoral implantation of rats. Journal of Biomedical Materials Research Part B Applied Biomaterials. 105(6). 1514–1524. 4 indexed citations
2.
Todoki, Kazuo, Toshizo Toyama, Masato Matsuo, et al.. (2015). Contribution of nitrergic nerve in canine gingival reactive hyperemia. Journal of Clinical Biochemistry and Nutrition. 56(2). 98–104. 11 indexed citations
3.
Toyama, Toshizo, Satoko Wada-Takahashi, Kiyoko Watanabe, et al.. (2014). Reactive Oxygen Species Scavenging Activity of Jixueteng Evaluated by Electron Spin Resonance (ESR) and Photon Emission. Natural Product Communications. 9(12). 1755–9. 12 indexed citations
4.
Toyama, Toshizo, Kazuo Todoki, Yusuke Takahashi, et al.. (2012). Inhibitory effects of Jixueteng on P. gingivalis-induced bone loss and osteoclast differentiation. Archives of Oral Biology. 57(11). 1529–1536. 12 indexed citations
5.
Takahashi, Shunsuke, Ayaka Yoshida, Kyo Kobayashi, et al.. (2012). Gingival vascular functions are altered in type 2 diabetes mellitus model and/or periodontitis model. Journal of Clinical Biochemistry and Nutrition. 51(2). 108–113. 48 indexed citations
6.
Takahashi, Shunsuke, Fumihiko Yoshino, Kazuo Todoki, et al.. (2008). Nitric oxide levels in rat hypothalamus are increased by restraint stress and decreased by biting. Redox Report. 13(1). 31–39. 15 indexed citations
7.
Kinoshita, Yukihiko, Masato Matsuo, Kazuo Todoki, et al.. (2008). Alveolar bone regeneration using absorbable poly(L-lactide-co-ɛ-caprolactone)/β-tricalcium phosphate membrane and gelatin sponge incorporating basic fibroblast growth factor. International Journal of Oral and Maxillofacial Surgery. 37(3). 275–281. 28 indexed citations
8.
9.
Takahashi, Shunsuke, Hiroyuki Miyazaki, Fumihiko Yoshino, et al.. (2003). Real-time monitoring of nitric oxide in ischemic myocardium using an NO-selective electrode calibrated by electron spin resonance. Life Sciences. 74(1). 75–85. 8 indexed citations
10.
Takahashi, Shunsuke, et al.. (2002). Role of nitric oxide in post-ischemic gingival hyperemia in anesthetized dogs. Redox Report. 7(5). 300–303. 16 indexed citations
11.
Nakamura, Yuko, Hitoshi Matsumoto, & Kazuo Todoki. (2002). Endothelium-Dependent Vasorelaxation Induced by Black Currant Concentrate in Rat Thoracic Aorta. The Japanese Journal of Pharmacology. 89(1). 29–35. 65 indexed citations
12.
Okabe, Eiichiro & Kazuo Todoki. (1999). Microcirculatory hemodynamics in oral tissues with reference to neurogenic response and reactive oxygen species interaction.. Folia Pharmacologica Japonica. 113(4). 219–225. 2 indexed citations
13.
Todoki, Kazuo, et al.. (1997). Inhibition by Hydroxyl Radicals of Calcitonin Gene-Related Peptide-Mediated Neurogenic Vasorelaxation in Isolated Canine Lingual Artery. Journal of Pharmacology and Experimental Therapeutics. 280(1). 492–500. 5 indexed citations
14.
Todoki, Kazuo, et al.. (1995). Calcitonin Gene-Related Peptide Mediated Neurogenic Vasorelaxation in the Isolated Canine Lingual Artery. The Japanese Journal of Pharmacology. 67(4). 329–340. 7 indexed citations
15.
Todoki, Kazuo, et al.. (1992). Oxygen free radical-mediated selective endothelial dysfunction in isolated coronary artery. American Journal of Physiology-Heart and Circulatory Physiology. 262(3). H806–H812. 45 indexed citations
16.
17.
Todoki, Kazuo, Eiichiro Okabe, & Haruo Itô. (1989). Vascular endothelium contributes to inhibitory effect of free radicals on relaxation of canine coronary artery to acetylcholine. The Japanese Journal of Pharmacology. 49. 315–315. 1 indexed citations
18.
Todoki, Kazuo. (1988). Effect of vasoactive substances and electrical stimulation of the inferior alveolar nerve on blood flow in the dental pulp in dogs.. Folia Pharmacologica Japonica. 92(2). 61–67. 5 indexed citations
19.
Todoki, Kazuo, Eiichiro Okabe, & Haruo Itô. (1987). Mechanism of the regulation of oral microcirculation. 6(2). 122–129. 1 indexed citations
20.
Todoki, Kazuo, et al.. (1987). [Pharmacological analysis on the role of endogenous vasoactive substances in regulating mechanism of microcirculatory dynamics in oral region. Effect of vasoactive substances on pulpal blood flow].. PubMed. 22(1). 201–5. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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