Kathryn M. Thrailkill

4.4k total citations
75 papers, 3.5k citations indexed

About

Kathryn M. Thrailkill is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Genetics. According to data from OpenAlex, Kathryn M. Thrailkill has authored 75 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Endocrinology, Diabetes and Metabolism, 38 papers in Molecular Biology and 20 papers in Genetics. Recurrent topics in Kathryn M. Thrailkill's work include Growth Hormone and Insulin-like Growth Factors (28 papers), Metabolism, Diabetes, and Cancer (18 papers) and Bone Metabolism and Diseases (12 papers). Kathryn M. Thrailkill is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (28 papers), Metabolism, Diabetes, and Cancer (18 papers) and Bone Metabolism and Diseases (12 papers). Kathryn M. Thrailkill collaborates with scholars based in United States, United Kingdom and Germany. Kathryn M. Thrailkill's co-authors include John L. Fowlkes, R. Clay Bunn, Charles K. Lumpkin, Gael Cockrell, Stephen F. Kemp, Hideaki Nagase, Elizabeth C. Wahl, Delila Serra, Cynthia Moreau and Evangelia Kalaitzoglou and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Clinical Endocrinology & Metabolism and Diabetes Care.

In The Last Decade

Kathryn M. Thrailkill

75 papers receiving 3.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Kathryn M. Thrailkill United States 36 1.5k 1.2k 643 582 539 75 3.5k
John L. Fowlkes United States 38 1.8k 1.2× 1.5k 1.2× 651 1.0× 549 0.9× 724 1.3× 89 4.1k
Yosuke Okada Japan 38 2.0k 1.4× 1.1k 0.9× 440 0.7× 638 1.1× 1.1k 2.0× 235 5.0k
Lars Sävendahl Sweden 40 1.7k 1.2× 1.5k 1.3× 412 0.6× 1.7k 2.9× 506 0.9× 154 4.9k
Cyrille B. Confavreux France 25 1.5k 1.0× 732 0.6× 1.4k 2.1× 403 0.7× 1.1k 2.0× 86 4.4k
Vicky E. MacRae United Kingdom 36 1.3k 0.9× 609 0.5× 286 0.4× 783 1.3× 357 0.7× 103 3.6k
Leon A. Bach Australia 43 2.6k 1.8× 2.8k 2.4× 462 0.7× 700 1.2× 455 0.8× 154 6.3k
R. Ziegler Germany 40 1.7k 1.2× 999 0.9× 1.4k 2.1× 465 0.8× 1.2k 2.2× 214 5.8k
Johannes Pfeilschifter Germany 34 2.3k 1.6× 942 0.8× 1.5k 2.3× 630 1.1× 1.7k 3.2× 102 5.4k
Luca Dalle Carbonare Italy 35 1.2k 0.8× 321 0.3× 937 1.5× 270 0.5× 851 1.6× 151 3.7k
Yasato Komatsu Japan 31 1.6k 1.1× 732 0.6× 233 0.4× 537 0.9× 586 1.1× 74 5.0k

Countries citing papers authored by Kathryn M. Thrailkill

Since Specialization
Citations

This map shows the geographic impact of Kathryn M. Thrailkill's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kathryn M. Thrailkill with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kathryn M. Thrailkill more than expected).

Fields of papers citing papers by Kathryn M. Thrailkill

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kathryn M. Thrailkill. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kathryn M. Thrailkill. The network helps show where Kathryn M. Thrailkill may publish in the future.

Co-authorship network of co-authors of Kathryn M. Thrailkill

This figure shows the co-authorship network connecting the top 25 collaborators of Kathryn M. Thrailkill. A scholar is included among the top collaborators of Kathryn M. Thrailkill based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kathryn M. Thrailkill. Kathryn M. Thrailkill is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
McDonald, Sarah D., et al.. (2022). Heterogeneity and altered β-cell identity in the TallyHo model of early-onset type 2 diabetes. Acta Histochemica. 124(7). 151940–151940. 1 indexed citations
2.
Thrailkill, Kathryn M., R. Clay Bunn, Sasidhar Uppuganti, et al.. (2020). Canagliflozin, an SGLT2 inhibitor, corrects glycemic dysregulation in TallyHO model of T2D but only partially prevents bone deficits. Bone. 141. 115625–115625. 14 indexed citations
3.
Fowlkes, John L., R. Clay Bunn, Evangelia Kalaitzoglou, et al.. (2019). Constitutive activation of MEK1 in osteoprogenitors increases strength of bone despite impairing mineralization. Bone. 130. 115106–115106. 8 indexed citations
4.
Nyman, Jeffry S., Evangelia Kalaitzoglou, R. Clay Bunn, et al.. (2017). Preserving and restoring bone with continuous insulin infusion therapy in a mouse model of type 1 diabetes. Bone Reports. 7. 1–8. 28 indexed citations
5.
Thrailkill, Kathryn M., Jeffry S. Nyman, R. Clay Bunn, et al.. (2016). The impact of SGLT2 inhibitors, compared with insulin, on diabetic bone disease in a mouse model of type 1 diabetes. Bone. 94. 141–151. 56 indexed citations
6.
Kalaitzoglou, Evangelia, Iuliana Popescu, R. Clay Bunn, John L. Fowlkes, & Kathryn M. Thrailkill. (2016). Effects of Type 1 Diabetes on Osteoblasts, Osteocytes, and Osteoclasts. Current Osteoporosis Reports. 14(6). 310–319. 87 indexed citations
7.
Thrailkill, Kathryn M., R. Clay Bunn, Jeffry S. Nyman, et al.. (2015). SGLT2 inhibitor therapy improves blood glucose but does not prevent diabetic bone disease in diabetic DBA/2J male mice. Bone. 82. 101–107. 63 indexed citations
8.
Haafiz, Allah, et al.. (2014). Cushing’s disease presenting as cholestatic hepatitis. Journal of Pediatric Endocrinology and Metabolism. 0(0). 1037–41. 4 indexed citations
9.
Thrailkill, Kathryn M., Cynthia Moreau, Christopher J. Swearingen, et al.. (2011). Insulin Pump Therapy Started at the Time of Diagnosis: Effects on Glycemic Control and Pancreatic β-Cell Function in Type 1 Diabetes. Diabetes Technology & Therapeutics. 13(10). 1023–1030. 21 indexed citations
10.
Nyman, Jeffry S., Jesse L. Even, Chan‐Hee Jo, et al.. (2010). Increasing duration of type 1 diabetes perturbs the strength–structure relationship and increases brittleness of bone. Bone. 48(4). 733–740. 89 indexed citations
11.
12.
Frindik, J. Paul, et al.. (2009). Stimulant Medication Use and Response to Growth Hormone Therapy: An NCGS Database Analysis. Hormone Research in Paediatrics. 72(3). 160–166. 11 indexed citations
13.
14.
Liu, Zhendong, James Aronson, Elizabeth C. Wahl, et al.. (2007). A novel rat model for the study of deficits in bone formation in type-2 diabetes. Acta Orthopaedica. 78(1). 46–55. 40 indexed citations
15.
Fowlkes, John L., R. Clay Bunn, Lichu Liu, et al.. (2007). Runt-Related Transcription Factor 2 (RUNX2) and RUNX2-Related Osteogenic Genes Are Down-Regulated throughout Osteogenesis in Type 1 Diabetes Mellitus. Endocrinology. 149(4). 1697–1704. 93 indexed citations
16.
Fowlkes, John L., Delila Serra, R. Clay Bunn, et al.. (2003). Regulation of Insulin-Like Growth Factor (IGF)-I Action by Matrix Metalloproteinase-3 Involves Selective Disruption of IGF-I/IGF-Binding Protein-3 Complexes. Endocrinology. 145(2). 620–626. 56 indexed citations
17.
Thrailkill, Kathryn M., Teresa Quattrin, Lester Baker, et al.. (1997). Dual Hormonal Replacement Therapy with Insulin and Recombinant Human Insulin-Like Growth Factor (IGF)-I in Insulin-Dependent Diabetes Mellitus: Effects on the Growth Hormone/IGF/IGF-Binding Protein System*. The Journal of Clinical Endocrinology & Metabolism. 82(4). 1181–1187. 33 indexed citations
18.
Thrailkill, Kathryn M., Suresh Siddhanti, John L. Fowlkes, & L. Darryl Quarles. (1995). Differentiation of MC3T3-E1 Osteoblasts is associated with temporal changes in the expression of IGF-I and IGFBPs. Bone. 17(3). 307–313. 42 indexed citations
19.
Poisner, Alan, Kathryn M. Thrailkill, Stuart Handwerger, & R. Poisner. (1991). Cyclic AMP as a second messenger for prorenin release from human decidual cells. Placenta. 12(3). 263–267. 18 indexed citations
20.
Clemmons, David R., et al.. (1990). Three Distinct Forms of Insulin-Like Growth Factor Binding Proteins are Released by Decidual Cells in Culture*. Endocrinology. 127(2). 643–650. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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