Kathrin Grundmann

1.1k total citations
22 papers, 770 citations indexed

About

Kathrin Grundmann is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, Kathrin Grundmann has authored 22 papers receiving a total of 770 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cellular and Molecular Neuroscience, 18 papers in Neurology and 3 papers in Molecular Biology. Recurrent topics in Kathrin Grundmann's work include Neurological disorders and treatments (18 papers), Genetic Neurodegenerative Diseases (16 papers) and Parkinson's Disease Mechanisms and Treatments (7 papers). Kathrin Grundmann is often cited by papers focused on Neurological disorders and treatments (18 papers), Genetic Neurodegenerative Diseases (16 papers) and Parkinson's Disease Mechanisms and Treatments (7 papers). Kathrin Grundmann collaborates with scholars based in Germany, United States and Italy. Kathrin Grundmann's co-authors include Helge Topka, K. Jaschonek, J. Dichgans, B.U. Kleine, Antonio Pisani, Olaf Rieß, Hartwig Wolburg, Rose E. Goodchild, Huu Phuc Nguyen and Ulrich Müller and has published in prestigious journals such as Neurology, Trends in Neurosciences and Journal of Neurology Neurosurgery & Psychiatry.

In The Last Decade

Kathrin Grundmann

22 papers receiving 753 citations

Peers

Kathrin Grundmann
Gretchen M. Thomsen United States
Stefan Braeuninger Germany
Mark P. Bowes United States
Ikuko Takeda Japan
Els K. Vanhoutte Netherlands
Joanna Iłżecka Poland
Roberto Fernández‐Torrón Spain
F. Reisecker Austria
Yoshiteru Soejima Japan
Lars Wallstedt Sweden
Gretchen M. Thomsen United States
Kathrin Grundmann
Citations per year, relative to Kathrin Grundmann Kathrin Grundmann (= 1×) peers Gretchen M. Thomsen

Countries citing papers authored by Kathrin Grundmann

Since Specialization
Citations

This map shows the geographic impact of Kathrin Grundmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kathrin Grundmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kathrin Grundmann more than expected).

Fields of papers citing papers by Kathrin Grundmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kathrin Grundmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kathrin Grundmann. The network helps show where Kathrin Grundmann may publish in the future.

Co-authorship network of co-authors of Kathrin Grundmann

This figure shows the co-authorship network connecting the top 25 collaborators of Kathrin Grundmann. A scholar is included among the top collaborators of Kathrin Grundmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kathrin Grundmann. Kathrin Grundmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lohmann, Ebba, Marc Sturm, Florian Lenz, et al.. (2019). Single Molecule Molecular Inversion Probes for High Throughput Germline Screenings in Dystonia. Frontiers in Neurology. 10. 1332–1332. 2 indexed citations
2.
Lohmann, Ebba, Thomas Gasser, & Kathrin Grundmann. (2017). Needs and Requirements of Modern Biobanks on the Example of Dystonia Syndromes. Frontiers in Neurology. 8. 9–9. 4 indexed citations
3.
Faust, Ulrike, Marc Sturm, Karl Hackmann, et al.. (2015). HBOC multi-gene panel testing: comparison of two sequencing centers. Breast Cancer Research and Treatment. 152(1). 129–136. 35 indexed citations
4.
Ryan, Scott D., Osmar Norberto de Souza, Luís Fernando Saraiva Macedo Timmers, et al.. (2014). Biochemical and Cellular Analysis of Human Variants of the DYT1 Dystonia Protein, TorsinA/TOR1A. Human Mutation. 35(9). 1101–1113. 17 indexed citations
5.
Cheng, Fubo, Jiachun Feng, Jing Miao, et al.. (2014). Combined occurrence of a novel TOR1A and a THAP1 mutation in primary dystonia. Movement Disorders. 29(8). 1079–1083. 16 indexed citations
6.
Dufke, Claudia, Marc Sturm, Christopher Schroeder, et al.. (2014). Screening of mutations in GNAL in sporadic dystonia patients. Movement Disorders. 29(9). 1193–1196. 12 indexed citations
7.
Goodchild, Rose E., Kathrin Grundmann, & Antonio Pisani. (2013). New genetic insights highlight ‘old’ ideas on motor dysfunction in dystonia. Trends in Neurosciences. 36(12). 717–725. 42 indexed citations
8.
Grundmann, Kathrin, Nicola Glöckle, Giuseppina Martella, et al.. (2012). Generation of a novel rodent model for DYT1 dystonia. Neurobiology of Disease. 47(1). 61–74. 64 indexed citations
9.
Glöckle, Nicola, Günther Deuschl, Ute Felbor, et al.. (2010). Prevalence of THAP1 sequence variants in German patients with primary dystonia. Movement Disorders. 25(12). 1982–1986. 24 indexed citations
10.
Walter, Martin A., Marina de Nadai Bonin Gomes, Enza Maria Valente, et al.. (2010). Expression profiling in peripheral blood reveals signature for penetrance in DYT1 dystonia. Neurobiology of Disease. 38(2). 192–200. 12 indexed citations
11.
Regensburger, Martin, Zacharias Kohl, Kathrin Grundmann, et al.. (2009). Adult neural precursor cells unaffected in animal models of DYT1 dystonia. Neuroreport. 20(17). 1529–1533. 3 indexed citations
12.
Zirn, Birgit, et al.. (2008). Novel TOR1A mutation p.Arg288Gln in early-onset dystonia (DYT1). Journal of Neurology Neurosurgery & Psychiatry. 79(12). 1327–1330. 55 indexed citations
13.
Grundmann, Kathrin, Jeannette Hübener‐Schmid, Karina Häbig, et al.. (2008). Gene expression changes in a transgenic mouse model overexpressing human wildtype and mutant torsinA. PROTEOMICS - CLINICAL APPLICATIONS. 2(5). 720–736. 4 indexed citations
14.
Grundmann, Kathrin, Greetje Vanhoutte, Jeannette Hübener‐Schmid, et al.. (2007). Overexpression of human wildtype torsinA and human ΔGAG torsinA in a transgenic mouse model causes phenotypic abnormalities. Neurobiology of Disease. 27(2). 190–206. 103 indexed citations
15.
Grundmann, Kathrin. (2005). Primary Torsion Dystonia. Archives of Neurology. 62(4). 682–682. 9 indexed citations
16.
Grundmann, Kathrin, Ulrike Laubis‐Herrmann, Dirk Dressler, et al.. (2004). Lack of mutations in the epsilon‐sarcoglycan gene in patients with different subtypes of primary dystonias. Movement Disorders. 19(11). 1294–1297. 5 indexed citations
17.
Grundmann, Kathrin, Ulrike Laubis‐Herrmann, Dirk Dressler, et al.. (2004). Mutation at the SCA17 locus is not a common cause of primary dystonia. Journal of Neurology. 251(10). 1232–1234. 4 indexed citations
18.
Grundmann, Kathrin, Ulrike Laubis‐Herrmann, Ingrid Bauer, et al.. (2003). Frequency and Phenotypic Variability of the GAG Deletion of the DYT1 Gene in an Unselected Group of Patients With Dystonia. Archives of Neurology. 60(9). 1266–70. 70 indexed citations
19.
Grundmann, Kathrin, K. Jaschonek, B.U. Kleine, J. Dichgans, & Helge Topka. (2003). Aspirin non-responder status in patients with recurrent cerebral ischemic attacks. Journal of Neurology. 250(1). 63–66. 192 indexed citations
20.
Janecke, Andreas, Moritz Meins, Kathrin Grundmann, et al.. (1999). Twelve novel myosin VIIA mutations in 34 patients with Usher syndrome type I: Confirmation of genetic heterogeneity. Human Mutation. 13(2). 133–140. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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