Katelyn E. Sadler

954 total citations
28 papers, 635 citations indexed

About

Katelyn E. Sadler is a scholar working on Physiology, Genetics and Molecular Biology. According to data from OpenAlex, Katelyn E. Sadler has authored 28 papers receiving a total of 635 indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Physiology, 7 papers in Genetics and 4 papers in Molecular Biology. Recurrent topics in Katelyn E. Sadler's work include Pain Mechanisms and Treatments (15 papers), Hemoglobinopathies and Related Disorders (7 papers) and Ion Channels and Receptors (4 papers). Katelyn E. Sadler is often cited by papers focused on Pain Mechanisms and Treatments (15 papers), Hemoglobinopathies and Related Disorders (7 papers) and Ion Channels and Receptors (4 papers). Katelyn E. Sadler collaborates with scholars based in United States, Israel and Canada. Katelyn E. Sadler's co-authors include Cheryl L. Stucky, Benedict J. Kolber, Jeffrey S. Mogil, Francie Moehring, Sherri K. Vogt, Robert W. Gereau, Michael R. Bruchas, Clinton D. Morgan, Lara W. Crock and Elena Isaeva and has published in prestigious journals such as Journal of Neuroscience, SHILAP Revista de lepidopterología and Blood.

In The Last Decade

Katelyn E. Sadler

28 papers receiving 628 citations

Peers

Katelyn E. Sadler

PHYSI

CMN

Rachel Wodarski United Kingdom

C. Burgos-Vega United States

Lois J. Kehl United States

Marc‐André Dansereau Canada

Yinghong Tian China

Tony K.Y. Lim Canada

Ricardo Kusuda Brazil

Curtis A. Benson United States

J. F. Bernard France

Anthony J. Kirkup United Kingdom

Rachel Wodarski United Kingdom

Katelyn E. Sadler

460 ×1.3

PHYSI

201 ×1.1

CMN

96 ×0.7

65 ×0.7

27 ×0.4

Citations per year, relative to Katelyn E. Sadler Katelyn E. Sadler (= 1×) peers Rachel Wodarski

Countries citing papers authored by Katelyn E. Sadler

Since Specialization

Citations

This map shows the geographic impact of Katelyn E. Sadler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katelyn E. Sadler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katelyn E. Sadler more than expected).

Fields of papers citing papers by Katelyn E. Sadler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katelyn E. Sadler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katelyn E. Sadler. The network helps show where Katelyn E. Sadler may publish in the future.

Co-authorship network of co-authors of Katelyn E. Sadler

This figure shows the co-authorship network connecting the top 25 collaborators of Katelyn E. Sadler. A scholar is included among the top collaborators of Katelyn E. Sadler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katelyn E. Sadler. Katelyn E. Sadler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dussor, Gregory, et al.. (2024). Probiotic Treatment Alleviates Chronic Pain in Sickle Cell Disease Mice. Journal of Pain. 25(4). 20–20. 1 indexed citations

Sadler, Katelyn E., et al.. (2024). Microbiome contributions to pain: a review of the preclinical literature. Pain. 166(2). 262–281. 2 indexed citations

Waltz, Tyler B., et al.. (2024). High-speed imaging of evoked rodent mechanical behaviors yields variable results that are not predictive of inflammatory injury. Pain. 165(7). 1569–1582. 3 indexed citations

Isaeva, Elena, Marie L. Schulte, Seung Min Shin, et al.. (2024). Increased keratinocyte activity and PIEZO1 signaling contribute to paclitaxel-induced mechanical hypersensitivity. Science Translational Medicine. 16(777). eadn5629–eadn5629. 6 indexed citations

Sadler, Katelyn E., et al.. (2023). Peripheral transient receptor potential vanilloid type 4 hypersensitivity contributes to chronic sickle cell disease pain. Pain. 164(8). 1874–1886. 4 indexed citations

Isaeva, Elena, et al.. (2022). Keratinocyte PIEZO1 modulates cutaneous mechanosensation. eLife. 11. 40 indexed citations

Sadler, Katelyn E., Jeffrey S. Mogil, & Cheryl L. Stucky. (2021). Innovations and advances in modelling and measuring pain in animals. Nature reviews. Neuroscience. 23(2). 70–85. 113 indexed citations

Waltz, Tyler B., Anthony J. Burand, Katelyn E. Sadler, & Cheryl L. Stucky. (2021). Sensory-specific peripheral nerve pathology in a rat model of Fabry disease. SHILAP Revista de lepidopterología. 10. 100074–100074. 9 indexed citations

Sadler, Katelyn E., Francie Moehring, Stephanie Shiers, et al.. (2021). Transient receptor potential canonical 5 mediates inflammatory mechanical and spontaneous pain in mice. Science Translational Medicine. 13(595). 56 indexed citations

10.

Sadler, Katelyn E., Francie Moehring, & Cheryl L. Stucky. (2020). Keratinocytes contribute to normal cold and heat sensation. eLife. 9. 61 indexed citations

11.

Sadler, Katelyn E., et al.. (2019). Gabapentin alleviates chronic spontaneous pain and acute hypoxia‐related pain in a mouse model of sickle cell disease. British Journal of Haematology. 187(2). 246–260. 12 indexed citations

12.

Sadler, Katelyn E., et al.. (2017). Central amygdala activation of extracellular signal-regulated kinase 1 and age-dependent changes in inflammatory pain sensitivity in mice. Neurobiology of Aging. 56. 100–107. 13 indexed citations

13.

Sadler, Katelyn E., et al.. (2016). Divergent functions of the left and right central amygdala in visceral nociception. Pain. 158(4). 747–759. 72 indexed citations

14.

Sadler, Katelyn E., et al.. (2016). Hormonal and molecular effects of restraint stress on formalin-induced pain-like behavior in male and female mice. Physiology & Behavior. 165. 278–285. 28 indexed citations

15.

Sadler, Katelyn E. & Benedict J. Kolber. (2016). Urine Trouble: Alterations in Brain Function Associated with Bladder Pain. The Journal of Urology. 196(1). 24–32. 11 indexed citations

16.

Sadler, Katelyn E., et al.. (2014). Urinary Bladder Distention Evoked Visceromotor Responses as a Model for Bladder Pain in Mice. Journal of Visualized Experiments. 15 indexed citations

17.

Sadler, Katelyn E., et al.. (2014). Urinary Bladder Distention Evoked Visceromotor Responses as a Model for Bladder Pain in Mice. Journal of Visualized Experiments. 5 indexed citations

18.

Sadler, Katelyn E., et al.. (2013). Optimization of a Pain Model: Effects of Body Temperature and Anesthesia on Bladder Nociception in Mice. PLoS ONE. 8(11). e79617–e79617. 13 indexed citations

19.

Crock, Lara W., Benedict J. Kolber, Clinton D. Morgan, et al.. (2012). Central Amygdala Metabotropic Glutamate Receptor 5 in the Modulation of Visceral Pain. Journal of Neuroscience. 32(41). 14217–14226. 89 indexed citations

20.

Lam, TH, Wen‐Chi Foo, Joo Ha Hwang, et al.. (2004). Report of Cancer Expert Working Group on Cancer Prevention and Screening. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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