Kate L. Graham

1.5k total citations
35 papers, 1.2k citations indexed

About

Kate L. Graham is a scholar working on Genetics, Immunology and Infectious Diseases. According to data from OpenAlex, Kate L. Graham has authored 35 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Genetics, 16 papers in Immunology and 11 papers in Infectious Diseases. Recurrent topics in Kate L. Graham's work include Diabetes and associated disorders (19 papers), Immune Cell Function and Interaction (11 papers) and Pancreatic function and diabetes (11 papers). Kate L. Graham is often cited by papers focused on Diabetes and associated disorders (19 papers), Immune Cell Function and Interaction (11 papers) and Pancreatic function and diabetes (11 papers). Kate L. Graham collaborates with scholars based in Australia, Canada and United States. Kate L. Graham's co-authors include Thomas W. H. Kay, Barbara S. Coulson, Helen E. Thomas, Balasubramanian Krishnamurthy, Yoshikazu Takada, Jonathan Chee, Stacey Fynch, Yan Tan, Prerak Trivedi and Péter Halász and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and PLoS ONE.

In The Last Decade

Kate L. Graham

34 papers receiving 1.2k citations

Peers

Kate L. Graham
Mark A. Wallet United States
Chad Storer United States
David Jones United States
Truman Grayson United States
Gady Cojocaru United States
Vinit Karmali United States
Kate L. Graham
Citations per year, relative to Kate L. Graham Kate L. Graham (= 1×) peers Heidi Trusheim

Countries citing papers authored by Kate L. Graham

Since Specialization
Citations

This map shows the geographic impact of Kate L. Graham's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kate L. Graham with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kate L. Graham more than expected).

Fields of papers citing papers by Kate L. Graham

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kate L. Graham. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kate L. Graham. The network helps show where Kate L. Graham may publish in the future.

Co-authorship network of co-authors of Kate L. Graham

This figure shows the co-authorship network connecting the top 25 collaborators of Kate L. Graham. A scholar is included among the top collaborators of Kate L. Graham based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kate L. Graham. Kate L. Graham is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Graham, Kate L., et al.. (2025). The role of 12-lipoxygenase in regulating megakaryocyte maturation and platelet-like particles production. Thrombosis Research. 254. 109445–109445.
3.
Akazawa, Satoru, Gaurang Jhala, Stacey Fynch, et al.. (2021). Deficiency of the innate immune adaptor STING promotes autoreactive T cell expansion in NOD mice. Diabetologia. 64(4). 878–889. 10 indexed citations
4.
Sutherland, Andrew P. R., Kate L. Graham, Gaurang Jhala, et al.. (2019). IL-21 regulates SOCS1 expression in autoreactive CD8+ T cells but is not required for acquisition of CTL activity in the islets of non-obese diabetic mice. Scientific Reports. 9(1). 15302–15302. 4 indexed citations
5.
Neil, Jessica A., Fiona E. Fleming, Kate L. Graham, et al.. (2016). Rotavirus acceleration of type 1 diabetes in non-obese diabetic mice depends on type I interferon signalling. Scientific Reports. 6(1). 29697–29697. 20 indexed citations
6.
Jhala, Gaurang, Jonathan Chee, Prerak Trivedi, et al.. (2016). Perinatal tolerance to proinsulin is sufficient to prevent autoimmune diabetes. JCI Insight. 1(10). e86065–e86065. 167 indexed citations
7.
Scott, John W., Sandra Galić, Kate L. Graham, et al.. (2015). Inhibition of AMP-Activated Protein Kinase at the Allosteric Drug-Binding Site Promotes Islet Insulin Release. Chemistry & Biology. 22(6). 705–711. 50 indexed citations
8.
Krishnamurthy, Balasubramanian, Jonathan Chee, Gaurang Jhala, et al.. (2015). BIM Deficiency Protects NOD Mice From Diabetes by Diverting Thymocytes to Regulatory T Cells. Diabetes. 64(9). 3229–3238. 11 indexed citations
9.
Graham, Kate L., Robyn M. Sutherland, Stuart I. Mannering, et al.. (2012). Pathogenic Mechanisms in Type 1 Diabetes: The Islet is Both Target and Driver of Disease. The Review of Diabetic Studies. 9(4). 148–168. 54 indexed citations
10.
Thomas, Helen E., Kate L. Graham, Jonathan Chee, et al.. (2012). Proinflammatory cytokines contribute to development and function of regulatory T cells in type 1 diabetes. Annals of the New York Academy of Sciences. 1283(1). 81–86. 25 indexed citations
11.
Neil, Jessica A., Nicole L. Webster, Kate L. Graham, et al.. (2012). Rotavirus acceleration of murine type 1 diabetes is associated with a T helper 1-dependent specific serum antibody response and virus effects in regional lymph nodes. Diabetologia. 56(3). 573–582. 19 indexed citations
12.
Mollah, Zia U.A., Kate L. Graham, Balasubramanian Krishnamurthy, et al.. (2012). Granzyme B Is Dispensable in the Development of Diabetes in Non-Obese Diabetic Mice. PLoS ONE. 7(7). e40357–e40357. 12 indexed citations
13.
Mollah, Zia U.A., Jibran A. Wali, Mark McKenzie, et al.. (2011). The pro-apoptotic BH3-only protein Bid is dispensable for development of insulitis and diabetes in the non-obese diabetic mouse. APOPTOSIS. 16(8). 822–830. 6 indexed citations
14.
Graham, Kate L., Balasubramanian Krishnamurthy, Stacey Fynch, et al.. (2011). Autoreactive Cytotoxic T Lymphocytes Acquire Higher Expression of Cytotoxic Effector Markers in the Islets of NOD Mice after Priming in Pancreatic Lymph Nodes. American Journal Of Pathology. 178(6). 2716–2725. 42 indexed citations
15.
Graham, Kate L., Yuxing Zhao, Pere Santamaría, et al.. (2011). An indirect role for NK cells in a CD4+ T‐cell‐dependent mouse model of type I diabetes. Immunology and Cell Biology. 90(2). 243–247. 10 indexed citations
16.
Sachithanandan, Nirupa, Kate L. Graham, Sandra Galić, et al.. (2011). Macrophage Deletion of SOCS1 Increases Sensitivity to LPS and Palmitic Acid and Results in Systemic Inflammation and Hepatic Insulin Resistance. Diabetes. 60(8). 2023–2031. 70 indexed citations
17.
Chee, Jonathan, Lina Mariana, Kate L. Graham, et al.. (2011). TNF Receptor 1 Deficiency Increases Regulatory T Cell Function in Nonobese Diabetic Mice. The Journal of Immunology. 187(4). 1702–1712. 34 indexed citations
18.
Fleming, Fiona E., Kate L. Graham, Yoshikazu Takada, & Barbara S. Coulson. (2010). Determinants of the Specificity of Rotavirus Interactions with the α2β1 Integrin. Journal of Biological Chemistry. 286(8). 6165–6174. 25 indexed citations
19.
Zhan, Yifan, Gayle M. Davey, Kate L. Graham, et al.. (2009). SOCS1 negatively regulates the production of Foxp3+ CD4+ T cells in the thymus. Immunology and Cell Biology. 87(6). 473–480. 23 indexed citations
20.
Fleming, Fiona E., Kate L. Graham, K. Taniguchi, Yoshikazu Takada, & Barbara S. Coulson. (2007). Rotavirus-neutralizing antibodies inhibit virus binding to integrins α2β1 and α4β1. Archives of Virology. 152(6). 1087–1101. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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