Katarzyna Popiołek-Barczyk

2.0k total citations · 1 hit paper
28 papers, 1.6k citations indexed

About

Katarzyna Popiołek-Barczyk is a scholar working on Physiology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Katarzyna Popiołek-Barczyk has authored 28 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Physiology, 10 papers in Cellular and Molecular Neuroscience and 9 papers in Neurology. Recurrent topics in Katarzyna Popiołek-Barczyk's work include Pain Mechanisms and Treatments (18 papers), Neuropeptides and Animal Physiology (9 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). Katarzyna Popiołek-Barczyk is often cited by papers focused on Pain Mechanisms and Treatments (18 papers), Neuropeptides and Animal Physiology (9 papers) and Neuroinflammation and Neurodegeneration Mechanisms (9 papers). Katarzyna Popiołek-Barczyk collaborates with scholars based in Poland, Italy and Germany. Katarzyna Popiołek-Barczyk's co-authors include Joanna Mika, Ewelina Rojewska, Barbara Przewłocka, Magdalena Żychowska, Wioletta Makuch, Anna Piotrowska, Katarzyna Starowicz, Agnieszka M. Jurga, Dominika Piłat and Katarzyna Ciapała and has published in prestigious journals such as PLoS ONE, Scientific Reports and International Journal of Molecular Sciences.

In The Last Decade

Katarzyna Popiołek-Barczyk

28 papers receiving 1.6k citations

Hit Papers

Importance of glial activation in neuropathic pain 2013 2026 2017 2021 2013 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Katarzyna Popiołek-Barczyk Poland 20 929 529 386 329 270 28 1.6k
Xu‐Hong Wei China 24 1.2k 1.2× 657 1.2× 252 0.7× 342 1.0× 215 0.8× 42 1.7k
Nancy Nutile‐McMenemy United States 15 973 1.0× 631 1.2× 480 1.2× 395 1.2× 133 0.5× 18 1.7k
Ewelina Rojewska Poland 34 2.0k 2.2× 1.1k 2.1× 374 1.0× 481 1.5× 585 2.2× 57 2.8k
Yuka Kobayashi Japan 23 1.1k 1.2× 710 1.3× 213 0.6× 533 1.6× 284 1.1× 56 2.1k
Norikazu Kiguchi Japan 31 1.6k 1.7× 1.3k 2.4× 305 0.8× 880 2.7× 230 0.9× 78 2.7k
Kazue Hisaoka‐Nakashima Japan 25 505 0.5× 482 0.9× 307 0.8× 495 1.5× 94 0.3× 69 1.4k
Wangyuan Zou China 26 640 0.7× 400 0.8× 264 0.7× 603 1.8× 115 0.4× 87 1.7k
Rui‐Ping Pang China 21 806 0.9× 433 0.8× 155 0.4× 584 1.8× 162 0.6× 32 1.7k
Jane P. Hughes United Kingdom 13 643 0.7× 559 1.1× 302 0.8× 586 1.8× 91 0.3× 20 2.1k
Yu Cui China 16 762 0.8× 544 1.0× 225 0.6× 276 0.8× 126 0.5× 24 1.2k

Countries citing papers authored by Katarzyna Popiołek-Barczyk

Since Specialization
Citations

This map shows the geographic impact of Katarzyna Popiołek-Barczyk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katarzyna Popiołek-Barczyk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katarzyna Popiołek-Barczyk more than expected).

Fields of papers citing papers by Katarzyna Popiołek-Barczyk

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Katarzyna Popiołek-Barczyk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katarzyna Popiołek-Barczyk. The network helps show where Katarzyna Popiołek-Barczyk may publish in the future.

Co-authorship network of co-authors of Katarzyna Popiołek-Barczyk

This figure shows the co-authorship network connecting the top 25 collaborators of Katarzyna Popiołek-Barczyk. A scholar is included among the top collaborators of Katarzyna Popiołek-Barczyk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Katarzyna Popiołek-Barczyk. Katarzyna Popiołek-Barczyk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Łażewska, Dorota, Gniewomir Latacz, Tadeusz Karcz, et al.. (2025). Histamine H3 receptor blockade alleviates neuropathic pain through the regulation of glial cells activation. Biomedicine & Pharmacotherapy. 183. 117850–117850. 2 indexed citations
3.
Stasiak, Anna, Ewelina Honkisz-Orzechowska, W. C. Wagner, et al.. (2024). AR71, Histamine H3 Receptor Ligand—In Vitro and In Vivo Evaluation (Anti-Inflammatory Activity, Metabolic Stability, Toxicity, and Analgesic Action). International Journal of Molecular Sciences. 25(15). 8035–8035. 2 indexed citations
4.
Fumagalli, Stefano, Katarzyna Popiołek-Barczyk, Marina Sironi, et al.. (2021). Long pentraxin PTX3 is upregulated systemically and centrally after experimental neurotrauma, but its depletion leaves unaltered sensorimotor deficits or histopathology. Scientific Reports. 11(1). 9616–9616. 11 indexed citations
5.
Ciechanowska, A, Katarzyna Popiołek-Barczyk, Katarzyna Pawlik, et al.. (2020). Changes in macrophage inflammatory protein-1 (MIP-1) family members expression induced by traumatic brain injury in mice. Immunobiology. 225(3). 151911–151911. 30 indexed citations
6.
Popiołek-Barczyk, Katarzyna, Katarzyna Pawlik, A Ciechanowska, et al.. (2020). CCR4 antagonist (C021) influences the level of nociceptive factors and enhances the analgesic potency of morphine in a rat model of neuropathic pain. European Journal of Pharmacology. 880. 173166–173166. 17 indexed citations
7.
Popiołek-Barczyk, Katarzyna, A Ciechanowska, Katarzyna Ciapała, et al.. (2020). The CCL2/CCL7/CCL12/CCR2 pathway is substantially and persistently upregulated in mice after traumatic brain injury, and CCL2 modulates the complement system in microglia. Molecular and Cellular Probes. 54. 101671–101671. 40 indexed citations
8.
Pawlik, Katarzyna, Anna Piotrowska, Klaudia Kwiatkowski, et al.. (2020). The blockade of CC chemokine receptor type 1 influences the level of nociceptive factors and enhances opioid analgesic potency in a rat model of neuropathic pain. Immunology. 159(4). 413–428. 31 indexed citations
9.
Kwiatkowski, Klaudia, Katarzyna Popiołek-Barczyk, Anna Piotrowska, et al.. (2019). Chemokines CCL2 and CCL7, but not CCL12, play a significant role in the development of pain-related behavior and opioid-induced analgesia. Cytokine. 119. 202–213. 53 indexed citations
10.
Popiołek-Barczyk, Katarzyna, Dorota Łażewska, Gniewomir Latacz, et al.. (2018). Antinociceptive effects of novel histamine H3 and H4 receptor antagonists and their influence on morphine analgesia of neuropathic pain in the mouse. British Journal of Pharmacology. 175(14). 2897–2910. 36 indexed citations
11.
Zajączkowska, Renata, Katarzyna Popiołek-Barczyk, Dominika Piłat, et al.. (2018). Involvement of microglial cells in the antinociceptive effects of metamizol in a mouse model of neuropathic pain. Pharmacology Biochemistry and Behavior. 175. 77–88. 13 indexed citations
12.
Costante, Roberto, Karel Guillemyn, Katarzyna Popiołek-Barczyk, et al.. (2017). Analgesic Properties of Opioid/NK1 Multitarget Ligands with Distinct in Vitro Profiles in Naive and Chronic Constriction Injury Mice. ACS Chemical Neuroscience. 8(10). 2315–2324. 28 indexed citations
13.
Piotrowska, Anna, Katarzyna Popiołek-Barczyk, Flaminia Pavone, & Joanna Mika. (2017). Comparison of the Expression Changes after Botulinum Toxin Type A and Minocycline Administration in Lipopolysaccharide-Stimulated Rat Microglial and Astroglial Cultures. Frontiers in Cellular and Infection Microbiology. 7. 141–141. 53 indexed citations
14.
Popiołek-Barczyk, Katarzyna, Natalia Kołosowska, Anna Piotrowska, et al.. (2015). Parthenolide Relieves Pain and Promotes M2 Microglia/Macrophage Polarization in Rat Model of Neuropathy. Neural Plasticity. 2015. 1–15. 87 indexed citations
15.
Ślusarczyk, Joanna, Ewa Trojan, Katarzyna Głombik, et al.. (2015). Prenatal stress is a vulnerability factor for altered morphology and biological activity of microglia cells. Frontiers in Cellular Neuroscience. 9. 82–82. 111 indexed citations
16.
Małek, Natalia, Katarzyna Popiołek-Barczyk, Joanna Mika, Barbara Przewłocka, & Katarzyna Starowicz. (2015). Anandamide, Acting viaCB2Receptors, Alleviates LPS-Induced Neuroinflammation in Rat Primary Microglial Cultures. Neural Plasticity. 2015. 1–10. 95 indexed citations
17.
Mika, Joanna, Katarzyna Popiołek-Barczyk, Ewelina Rojewska, et al.. (2014). Delta-Opioid Receptor Analgesia Is Independent of Microglial Activation in a Rat Model of Neuropathic Pain. PLoS ONE. 9(8). e104420–e104420. 70 indexed citations
18.
Popiołek-Barczyk, Katarzyna, Ewelina Rojewska, Agnieszka M. Jurga, et al.. (2014). Minocycline Enhances the Effectiveness of Nociceptin/Orphanin FQ during Neuropathic Pain. BioMed Research International. 2014. 1–12. 29 indexed citations
19.
Popiołek-Barczyk, Katarzyna, Wioletta Makuch, Ewelina Rojewska, Dominika Piłat, & Joanna Mika. (2014). Inhibition of intracellular signaling pathways NF-κB and MEK1/2 attenuates neuropathic pain development and enhances morphine analgesia. Pharmacological Reports. 66(5). 845–851. 52 indexed citations
20.
Mika, Joanna, Magdalena Żychowska, Katarzyna Popiołek-Barczyk, Ewelina Rojewska, & Barbara Przewłocka. (2013). Importance of glial activation in neuropathic pain. European Journal of Pharmacology. 716(1-3). 106–119. 382 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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